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Sensitizing cancer cells: is it really all about U?

Sensitizing cancer cells: is it really all about U?

http://www.wikidata.org/entity/Q41871066

about

Serine, glycine and one-carbon units: cancer metabolism in full circle A cyclin without cyclin-dependent kinases: cyclin F controls genome stability through ubiquitin-mediated proteolysis.Ribonucleotide reductase and cancer: biological mechanisms and targeted therapies.

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Sensitizing cancer cells: is it really all about U?

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2012 nî lūn-bûn

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Sensitizing cancer cells: is it really all about U?

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Sensitizing cancer cells: is it really all about U?

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Sensitizing cancer cells: is it really all about U?

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Patrick J Stover

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Robert S Weiss

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Cyclin F-mediated degradation of ribonucleotide reductase M2 controls genome integrity and DNA repair

5-fluorouracil: mechanisms of action and clinical strategies

Intracellular location of thymidylate synthase and its state of phosphorylation

Regulation and functional contribution of thymidine kinase 1 in repair of DNA damage

Serine hydroxymethyltransferase anchors de novo thymidylate synthesis pathway to nuclear lamina for DNA synthesis.

Folate deficiency causes uracil misincorporation into human DNA and chromosome breakage: implications for cancer and neuronal damage.

Ribonucleotide reduction is a cytosolic process in mammalian cells independently of DNA damage.

Synthetic lethality by lentiviral short hairpin RNA silencing of thymidylate kinase and doxorubicin in colon cancer cells regardless of the p53 status.

Essential role of Tip60-dependent recruitment of ribonucleotide reductase at DNA damage sites in DNA repair during G1 phase.

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10.1016/J.CCR.2012.06.010

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