Functional defects in the external and internal thin gates of the γ-aminobutyric acid (GABA) transporter GAT-1 can compensate each other
about
GABA and Glutamate Transporters in BrainA conserved salt bridge between transmembrane segments 1 and 10 constitutes an extracellular gate in the dopamine transporterMutations in the GABA Transporter SLC6A1 Cause Epilepsy with Myoclonic-Atonic SeizuresAn Extra Amino Acid Residue in Transmembrane Domain 10 of the γ-Aminobutyric Acid (GABA) Transporter GAT-1 Is Required for Efficient Ion-coupled Transport.SLC6 Transporter Folding Diseases and Pharmacochaperoning.
P2860
Functional defects in the external and internal thin gates of the γ-aminobutyric acid (GABA) transporter GAT-1 can compensate each other
description
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2013年の論文
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name
Functional defects in the exte ...... AT-1 can compensate each other
@en
type
label
Functional defects in the exte ...... AT-1 can compensate each other
@en
prefLabel
Functional defects in the exte ...... AT-1 can compensate each other
@en
P2860
P356
P1476
Functional defects in the exte ...... AT-1 can compensate each other
@en
P2093
Assaf Ben-Yona
Baruch I Kanner
P2860
P304
P356
10.1074/JBC.M112.430215
P407
P577
2013-01-03T00:00:00Z