HCN212-channel biological pacemakers manifesting ventricular tachyarrhythmias are responsive to treatment with I(f) blockade - Wikidata - wikimedia - TriplyDB

HCN212-channel biological pacemakers manifesting ventricular tachyarrhythmias are responsive to treatment with I(f) blockade

HCN212-channel biological pacemakers manifesting ventricular tachyarrhythmias are responsive to treatment with I(f) blockade

http://www.wikidata.org/entity/Q41875995

about

Ivabradine, coronary artery disease, and heart failure: beyond rhythm control Direct conversion of quiescent cardiomyocytes to pacemaker cells by expression of Tbx18.HCN2/SkM1 gene transfer into canine left bundle branch induces stable, autonomically responsive biological pacing at physiological heart rates.Differential effects of ivabradine and ryanodine on pacemaker activity in canine sinus node and purkinje fibers.Ca(2+)-stimulated adenylyl cyclase AC1 generates efficient biological pacing as single gene therapy and in combination with HCN2 Molecular Mapping of Sinoatrial Node HCN Channel Expression in the Human Heart Perspectives -- biological pacing, a clinical reality?Engineering a biological pacemaker: in vivo, in vitro and in silico models Rate control with ivabradine: angina pectoris and beyond.Regenerative therapies in electrophysiology and pacing: introducing the next steps.The road to biological pacing.Gene- and cell-based bio-artificial pacemaker: what basic and translational lessons have we learned?Gene therapy for restoring heart rhythm.Stem cell-based biological pacemakers from proof of principle to therapy: a review.Gene therapy to restore electrophysiological function in heart failure.In vitro characterization of HCN channel kinetics and frequency dependence in myocytes predicts biological pacemaker functionality.Dysfunctional Hyperpolarization-Activated Cyclic Nucleotide-gated Ion Channels in Cardiac Diseases.Genetic silencing of pacemaker cells: local intervention with global implications mHCN4 genetically modified canine mesenchymal stem cells provide biological pacemaking function in complete dogs with atrioventricular block.Coupling an HCN2-expressing cell to a myocyte creates a two-cell pacing unit.Diet-dependent modulation of gastro-oesphageal vagal afferent mechanosensitivity by endogenous nitric oxide.Fast and furious: new ways to think about, study and treat cardiac arrhythmias.Putting the pacemaker channel through its paces to build a better biological pacemaker.The therapeutic role of ivabradine in heart failure

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HCN212-channel biological pacemakers manifesting ventricular tachyarrhythmias are responsive to treatment with I(f) blockade

http://www.wikidata.org/entity/Q41875995

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

HCN212-channel biological pace ...... o treatment with I(f) blockade

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type

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HCN212-channel biological pace ...... o treatment with I(f) blockade

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prefLabel

HCN212-channel biological pace ...... o treatment with I(f) blockade

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P356

J.HRTHM.2007.09.028

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HCN212-channel biological pace ...... o treatment with I(f) blockade

@en

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Alexei N Plotnikov

http://www.w3.org/2001/XMLSchema#string

Annalisa Bucchi

http://www.w3.org/2001/XMLSchema#string

Ira S Cohen

http://www.w3.org/2001/XMLSchema#string

Iryna Shlapakova

http://www.w3.org/2001/XMLSchema#string

Michael R Rosen

http://www.w3.org/2001/XMLSchema#string

Peter Danilo

http://www.w3.org/2001/XMLSchema#string

Peter R Brink

http://www.w3.org/2001/XMLSchema#string

Richard B Robinson

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of hyperpolarization-activated HCN channel gating and cAMP modulation due to interactions of COOH terminus and core transmembrane regions

Mode of action of bradycardic agent, S 16257, on ionic currents of rabbit sinoatrial node cells.

Biological pacemaker created by gene transfer.

Biological pacemaker implanted in canine left bundle branch provides ventricular escape rhythms that have physiologically acceptable rates.

Properties of ivabradine-induced block of HCN1 and HCN4 pacemaker channels.

Molecular enhancement of porcine cardiac chronotropy.

Heart rate lowering by specific and selective I(f) current inhibition with ivabradine: a new therapeutic perspective in cardiovascular disease.

Use-dependent inhibition of hHCN4 by ivabradine and relationship with reduction in pacemaker activity.

Pacemaker current and automatic rhythms: toward a molecular understanding.

Current-dependent block of rabbit sino-atrial node I(f) channels by ivabradine

P304

282-288

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scholarly article

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10.1016/J.HRTHM.2007.09.028

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2

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5

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2007-10-03T00:00:00Z

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5608754

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18242555

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2254308

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