The C. elegans p38 MAPK pathway regulates nuclear localization of the transcription factor SKN-1 in oxidative stress response - Wikidata - wikimedia - TriplyDB

The C. elegans p38 MAPK pathway regulates nuclear localization of the transcription factor SKN-1 in oxidative stress response

The C. elegans p38 MAPK pathway regulates nuclear localization of the transcription factor SKN-1 in oxidative stress response

http://www.wikidata.org/entity/Q41916654

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Lessons from C. elegans: signaling pathways for longevity GATA transcription factors as tissue-specific master regulators for induced responses Biochemical Genetic Pathways that Modulate Aging in Multiple Species DLK-1, SEK-3 and PMK-3 Are Required for the Life Extension Induced by Mitochondrial Bioenergetic Disruption in C. elegans Graded Proteasome Dysfunction in Caenorhabditis elegans Activates an Adaptive Response Involving the Conserved SKN-1 and ELT-2 Transcription Factors and the Autophagy-Lysosome Pathway The Developmental Intestinal Regulator ELT-2 Controls p38-Dependent Immune Responses in Adult C. elegans Gamma-linolenic and stearidonic acids are required for basal immunity in Caenorhabditis elegans through their effects on p38 MAP kinase activity p38 MAPK regulates expression of immune response genes and contributes to longevity in C. elegans Regulation of synaptic nlg-1/neuroligin abundance by the skn-1/Nrf stress response pathway protects against oxidative stress A shift to organismal stress resistance in programmed cell death mutants Integration of the unfolded protein and oxidative stress responses through SKN-1/Nrf The conserved SKN-1/Nrf2 stress response pathway regulates synaptic function in Caenorhabditis elegans Conserved nutrient sensor O-GlcNAc transferase is integral to C. elegans pathogen-specific immunity Regulation of Drosophila metamorphosis by xenobiotic response regulators Stimulation of host immune defenses by a small molecule protects C. elegans from bacterial infection Nuclear hormone receptor regulation of microRNAs controls innate immune responses in C. elegans Relationship between mitochondrial electron transport chain dysfunction, development, and life extension in Caenorhabditis elegans Specific SKN-1/Nrf stress responses to perturbations in translation elongation and proteasome activity Phosphorylation of the conserved transcription factor ATF-7 by PMK-1 p38 MAPK regulates innate immunity in Caenorhabditis elegans Neuronal responses to stress and injury in C. elegans Nrf1 and Nrf2 play distinct roles in activation of antioxidant response element-dependent genes Multiple roles of Nrf2-Keap1 signaling: regulation of development and xenobiotic response using distinct mechanisms Stress-activated cap'n'collar transcription factors in aging and human disease Dopamine signaling promotes the xenobiotic stress response and protein homeostasis A role for SKN-1/Nrf in pathogen resistance and immunosenescence in Caenorhabditis elegans Direct inhibition of the longevity-promoting factor SKN-1 by insulin-like signaling in C. elegans Dietary restriction suppresses proteotoxicity and enhances longevity by an hsf-1-dependent mechanism in Caenorhabditis elegans.Environmental exposure, obesity, and Parkinson's disease: lessons from fat and old worms.ELT-2 is the predominant transcription factor controlling differentiation and function of the C. elegans intestine, from embryo to adult.Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans.The Caenorhabditis elegans germ line regulates distinct signaling pathways to control lifespan and innate immunity.The Caenorhabditis elegans Ste20-related kinase and Rac-type small GTPase regulate the c-Jun N-terminal kinase signaling pathway mediating the stress response WDR23 regulates NRF2 independently of KEAP1.Metformin promotes lifespan through mitohormesis via the peroxiredoxin PRDX-2 Dynamic O-GlcNAc cycling at promoters of Caenorhabditis elegans genes regulating longevity, stress, and immunity.skn-1-Dependent and -independent regulation of aip-1 expression following metabolic stress in Caenorhabditis elegans.Molecular characterization of numr-1 and numr-2: genes that increase both resistance to metal-induced stress and lifespan in Caenorhabditis elegans.Environmentally induced changes in correlated responses to selection reveal variable pleiotropy across a complex genetic network The DM domain transcription factor MAB-3 regulates male hypersensitivity to oxidative stress in Caenorhabditis elegans RNAi screening implicates a SKN-1-dependent transcriptional response in stress resistance and longevity deriving from translation inhibition.

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The C. elegans p38 MAPK pathway regulates nuclear localization of the transcription factor SKN-1 in oxidative stress response

http://www.wikidata.org/entity/Q41916654

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2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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name

The C. elegans p38 MAPK pathwa ...... 1 in oxidative stress response

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type

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The C. elegans p38 MAPK pathwa ...... 1 in oxidative stress response

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The C. elegans p38 MAPK pathwa ...... 1 in oxidative stress response

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Genes & Development

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The C. elegans p38 MAPK pathwa ...... 1 in oxidative stress response

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Eisuke Nishida

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Hideki Inoue

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Jae Hyung An

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Kunihiro Matsumoto

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Naoki Hisamoto

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T Keith Blackwell

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P2860

The kinase TAK1 can activate the NIK-I kappaB as well as the MAP kinase cascade in the IL-1 signalling pathway

A Caenorhabditis elegans JNK signal transduction pathway regulates coordinated movement via type-D GABAergic motor neurons.

The genetics of Caenorhabditis elegans

Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

Efficient gene transfer in C.elegans: extrachromosomal maintenance and integration of transforming sequences

Mammalian MAP kinase signalling cascades

Liver-specific inactivation of the Nrf1 gene in adult mouse leads to nonalcoholic steatohepatitis and hepatic neoplasia

Nrf2-Keap1 defines a physiologically important stress response mechanism

Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation

Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases

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10.1101/GAD.1324805

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19

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19

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Riva de Paula Oliveira

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2005-09-15T00:00:00Z

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7597522

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16166371

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positive regulation of transcription from RNA polymerase II promoter in response to reactive oxygen species

positive regulation of transcription from RNA polymerase II promoter in response to superoxide

Caenorhabditis elegans

P932

1240035

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