Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness. - Wikidata - wikimedia - TriplyDB

Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness.

Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness.

http://www.wikidata.org/entity/Q41923388

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Activation of the glutaredoxin-1 gene by nuclear factor κB enhances signaling Different effects of farrerol on an OVA-induced allergic asthma and LPS-induced acute lung injury Loss of mitochondrial protein Fus1 augments host resistance to Acinetobacter baumannii infection Mucosal candidiasis elicits NF-κB activation, proinflammatory gene expression and localized neutrophilia in zebrafish Inhibition of aldose reductase prevents experimental allergic airway inflammation in mice.Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation.Deguelin Attenuates Allergic Airway Inflammation via Inhibition of NF-κb Pathway in Mice.Mucosal sensitization to German cockroach involves protease-activated receptor-2.The glutaredoxin/S-glutathionylation axis regulates interleukin-17A-induced proinflammatory responses in lung epithelial cells in association with S-glutathionylation of nuclear factor κB family proteins.Animal models of allergic airways disease: where are we and where to next?SPLUNC1 regulation in airway epithelial cells: role of Toll-like receptor 2 signaling Airway epithelial NF-κB activation promotes Mycoplasma pneumoniae clearance in mice.Modulation of the oscillatory mechanics of lung tissue and the oxidative stress response induced by arginase inhibition in a chronic allergic inflammation model.Airway epithelial NF-κB activation promotes allergic sensitization to an innocuous inhaled antigen CaMKII is essential for the proasthmatic effects of oxidation.NF-κB mediates IL-1β- and IL-17A-induced MUC5B expression in airway epithelial cells.Effects of β(2) Agonists, Corticosteroids, and Novel Therapies on Rhinovirus-Induced Cytokine Release and Rhinovirus Replication in Primary Airway Fibroblasts Nuclear factor kappa B induction in airway epithelium increases lung inflammation in allergen-challenged mice.Airway epithelial regulation of allergic sensitization in asthma.Myd88 deficiency influences murine tracheal epithelial metaplasia and submucosal gland abundance.Genetic ablation of glutaredoxin-1 causes enhanced resolution of airways hyperresponsiveness and mucus metaplasia in mice with allergic airways disease Cooperation between classical and alternative NF-κB pathways regulates proinflammatory responses in epithelial cells.Airway Hyperresponsiveness and Inflammation: Causation, Correlation, or No Relation?Glutathione S-transferase pi modulates NF-κB activation and pro-inflammatory responses in lung epithelial cells.High-mobility group box 1 impairs airway epithelial barrier function through the activation of the RAGE/ERK pathway.Mechanisms of Asthma in Obesity. Pleiotropic Aspects of Obesity Produce Distinct Asthma Phenotypes NF-kappaB p50 plays distinct roles in the establishment and control of murine gammaherpesvirus 68 latency.Mucus hypersecretion in asthma: causes and effects A pro-inflammatory role for nuclear factor kappa B in childhood obstructive sleep apnea syndrome Highlights of a workshop to discuss targeting inflammation in cystic fibrosis A TLR2 agonist in German cockroach frass activates MMP-9 release and is protective against allergic inflammation in mice.Epithelial NF-κB orchestrates house dust mite-induced airway inflammation, hyperresponsiveness, and fibrotic remodeling Nuclear factor kappaB, airway epithelium, and asthma: avenues for redox control.Update in asthma 2008.Mitochondrial CaMKII inhibition in airway epithelium protects against allergic asthma.Airway remodeling in asthma: what really matters.Tumor necrosis factor-related apoptosis-inducing ligand translates neonatal respiratory infection into chronic lung disease.Epithelial, dendritic, and CD4(+) T cell regulation of and by reactive oxygen and nitrogen species in allergic sensitization.Effects and mechanisms of actions of Chinese herbal medicines for asthma.Cross-roads in the lung: immune cells and tissue interactions as determinants of allergic asthma.

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Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness.

http://www.wikidata.org/entity/Q41923388

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American Journal of Respiratory and Critical Care Medicine

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Nuclear factor-kappaB activati ...... ation and hyperresponsiveness.

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Amy L Brown

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Charles G Irvin

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Cristen Pantano

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Gilman B Allen

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John F Alcorn

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Laurie A Whittaker

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Mieke Bevelander

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Stacie L Beuschel

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P2860

Neutrophil-derived elastase induces TGF-beta1 secretion in human airway smooth muscle via NF-kappaB pathway

Rapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammation

Eosinophilic Inflammation in Asthma

Signaling to NF-kappaB

Hyperplasia of smooth muscle in mild to moderate asthma without changes in cell size or gene expression.

NF-kappa B activation in airways modulates allergic inflammation but not hyperresponsiveness

Asthma. From bronchoconstriction to airways inflammation and remodeling.

Allergen-induced peribronchial fibrosis and mucus production mediated by IkappaB kinase beta-dependent genes in airway epithelium

Blocking airway mucous cell metaplasia by inhibiting EGFR antiapoptosis and IL-13 transdifferentiation signals.

The bronchial epithelium in chronic and severe asthma.

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Matthew E Poynter

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18263801

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2361423

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