Induction of nuclear factor-κB responses by the S100A9 protein is Toll-like receptor-4-dependent.
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Resolution of Inflammation: What Controls Its Onset?Lubricin/Proteoglycan 4 binds to and regulates the activity of Toll-Like Receptors In Vitro.The host protein calprotectin modulates the Helicobacter pylori cag type IV secretion system via zinc sequestrationCommon interactions between S100A4 and S100A9 defined by a novel chemical probeCirculating Autoantibodies in Age-Related Macular Degeneration Recognize Human Macular Tissue Antigens Implicated in Autophagy, Immunomodulation, and Protection from Oxidative Stress and ApoptosisS100A8/A9 mRNA induction in an ex vivo model of endotoxin tolerance: roles of IL-10 and IFNγ.The pro-apoptotic and pro-inflammatory effects of calprotectin on human periodontal ligament cells.Human S100A9 protein is stabilized by inflammatory stimuli via the formation of proteolytically-resistant homodimers.S100A8 and S100A9 induce cytokine expression and regulate the NLRP3 inflammasome via ROS-dependent activation of NF-κB(1.).S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.The dietary flavonoid Kaempferol mediates anti-inflammatory responses via the Src, Syk, IRAK1, and IRAK4 molecular targets.CD14 Is a Co-Receptor for TLR4 in the S100A9-Induced Pro-Inflammatory Response in MonocytesThe anti-tumor effect of the quinoline-3-carboxamide tasquinimod: blockade of recruitment of CD11b(+) Ly6C(hi) cells to tumor tissue reduces tumor growthExpression of functional toll like receptor 4 in estrogen receptor/progesterone receptor-negative breast cancer.S100A9 expressed in ER(-)PgR(-) breast cancers induces inflammatory cytokines and is associated with an impaired overall survival.Tasquinimod triggers an early change in the polarization of tumor associated macrophages in the tumor microenvironment.Interleukin-6/signal transducer and activator of transcription 3 (STAT3) pathway is essential for macrophage infiltration and myoblast proliferation during muscle regeneration.S100A9 and ORM1 serve as predictors of therapeutic response and prognostic factors in advanced extranodal NK/T cell lymphoma patients treated with pegaspargase/gemcitabineProtein tyrosine phosphatase 1B negatively regulates S100A9-mediated lung damage during respiratory syncytial virus exacerbations.S100A8/A9, a potent serum and molecular imaging biomarker for synovial inflammation and joint destruction in seronegative experimental arthritis.S100A8 and S100A9: DAMPs at the crossroads between innate immunity, traditional risk factors, and cardiovascular disease.Eritoran inhibits S100A8-mediated TLR4/MD-2 activation and tumor growth by changing the immune microenvironment.Review of Rosai-Dorfman Disease: New Insights into the Pathogenesis of This Rare Disorder.Inhibition of macrophage functions by the C-terminus of murine S100A9 is dependent on B-1 cells.S100A8, S100A9 and S100A12 activate airway epithelial cells to produce MUC5AC via extracellular signal-regulated kinase and nuclear factor-κB pathways.Differential profiles of salivary proteins with affinity to Streptococcus mutans lipoteichoic acid in caries-free and caries-positive human subjects.Comparative quantitative proteomic analysis of disease stratified laser captured microdissected human islets identifies proteins and pathways potentially related to type 1 diabetesS100A9 and tumor growthProinflammatory effects and mechanisms of calprotectin on human gingival fibroblasts.Pro-inflammatory proteins S100A9 and tumor necrosis factor-α suppress erythropoietin elaboration in myelodysplastic syndromes.Coevolution of the Toll-Like Receptor 4 Complex with Calgranulins and Lipopolysaccharide.Secretion of the Phosphorylated Form of S100A9 from Neutrophils Is Essential for the Proinflammatory Functions of Extracellular S100A8/A9.Human S100A9 potentiates IL-8 production in response to GM-CSF or fMLP via activation of a different set of transcription factors in neutrophils.Paquinimod prevents development of diabetes in the non-obese diabetic (NOD) mouse.Exacerbation of hepatic injury during rodent malaria by myeloid-related protein 14.
P2860
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P2860
Induction of nuclear factor-κB responses by the S100A9 protein is Toll-like receptor-4-dependent.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Induction of nuclear factor-κB ...... oll-like receptor-4-dependent.
@en
type
label
Induction of nuclear factor-κB ...... oll-like receptor-4-dependent.
@en
prefLabel
Induction of nuclear factor-κB ...... oll-like receptor-4-dependent.
@en
P2093
P2860
P1433
P1476
Induction of nuclear factor-κB ...... oll-like receptor-4-dependent.
@en
P2093
Dora Hancz
Eva Källberg
Fredrik Ivars
Johannes Roth
Matteo Riva
Thomas Vogl
Tomas Leanderson
P2860
P304
P356
10.1111/J.1365-2567.2012.03619.X
P577
2012-10-01T00:00:00Z