MAP4-dependent regulation of microtubule formation affects centrosome, cilia, and Golgi architecture as a central mechanism in growth regulation. - Wikidata - wikimedia - TriplyDB

MAP4-dependent regulation of microtubule formation affects centrosome, cilia, and Golgi architecture as a central mechanism in growth regulation.

MAP4-dependent regulation of microtubule formation affects centrosome, cilia, and Golgi architecture as a central mechanism in growth regulation.

http://www.wikidata.org/entity/Q41929231

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Specialized Cilia in Mammalian Sensory Systems DYNC2LI1 mutations broaden the clinical spectrum of dynein-2 defects Mutations in TRAF3IP1/IFT54 reveal a new role for IFT proteins in microtubule stabilization Genomic insights into growth and its disorders: an update.A genome landscape of SRSF3-regulated splicing events and gene expression in human osteosarcoma U2OS cells.Modular transcriptional repertoire and MicroRNA target analyses characterize genomic dysregulation in the thymus of Down syndrome infants.The Protein Encoded by the CCDC170 Breast Cancer Gene Functions to Organize the Golgi-Microtubule Network Primary cilia proteins: ciliary and extraciliary sites and functions.Clinical relevance of systematic phenotyping and exome sequencing in patients with short stature.

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P2860

MAP4-dependent regulation of microtubule formation affects centrosome, cilia, and Golgi architecture as a central mechanism in growth regulation.

http://www.wikidata.org/entity/Q41929231

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2014 nî lūn-bûn

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MAP4-dependent regulation of m ...... echanism in growth regulation.

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MAP4-dependent regulation of m ...... mechanism in growth regulation

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Arif B Ekici

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Diana Zahnleiter

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Helmuth-Günther Dörr

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Holger Blessing

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Kristin Kessler

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Nadine N Hauer

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Steffen Uebe

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Yuya Sugano

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P2860

Assumption-free estimation of heritability from genome-wide identity-by-descent sharing between full siblings

An integrated map of genetic variation from 1,092 human genomes

NIH Image to ImageJ: 25 years of image analysis

A systematic review and meta-analysis to revise the Fenton growth chart for preterm infants

CEP152 is a genome maintenance protein disrupted in Seckel syndrome

MAP4 and CLASP1 operate as a safety mechanism to maintain a stable spindle position in mitosis

NEK1 mutations cause short-rib polydactyly syndrome type majewski

Autosomal Recessive Primary Microcephaly (MCPH): clinical manifestations, genetic heterogeneity and mutation continuum

A centrosomal mechanism involving CDK5RAP2 and CENPJ controls brain size

Adaptor protein complex 4 deficiency causes severe autosomal-recessive intellectual disability, progressive spastic paraplegia, shy character, and short stature

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87-97

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scholarly article

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10.1002/HUMU.22711

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1

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36

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Heinrich Sticht

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Christian T Thiel

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2014-11-28T00:00:00Z

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25323976

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