On How Fas Apoptosis-Independent Pathways Drive T Cell Hyperproliferation and Lymphadenopathy in lpr Mice.

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On How Fas Apoptosis-Independent Pathways Drive T Cell Hyperproliferation and Lymphadenopathy in lpr Mice.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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On How Fas Apoptosis-Independe ...... d Lymphadenopathy in lpr Mice.

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On How Fas Apoptosis-Independe ...... d Lymphadenopathy in lpr Mice.

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On How Fas Apoptosis-Independe ...... d Lymphadenopathy in lpr Mice.

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P1476

On How Fas Apoptosis-Independe ...... nd Lymphadenopathy in lpr Mice

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P2093

Carlos Martínez-A

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Cristina Vázquez-Mateo

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Lidia Daszkiewicz

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P2860

Fas Versatile Signaling and Beyond: Pivotal Role of Tyrosine Phosphorylation in Context-Dependent Signaling and Diseases

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

An Evolution-Guided Analysis Reveals a Multi-Signaling Regulation of Fas by Tyrosine Phosphorylation and its Implication in Human Cancers

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

An inherited disorder of lymphocyte apoptosis: the autoimmune lymphoproliferative syndrome.

Fas-mediated apoptosis regulates the composition of peripheral alphabeta T cell repertoire by constitutively purging out double negative T cells

Human TCR-alpha beta+ CD4- CD8- T cells can derive from CD8+ T cells and display an inflammatory effector phenotype

A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice.

New advances in the diagnosis and treatment of autoimmune lymphoproliferative syndrome

P304

237

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scholarly article

P356

10.3389/FIMMU.2017.00237

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P478

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P50

Dimitrios Balomenos

Rahman Shokri

P577

2017-03-10T00:00:00Z

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P698

28344578

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P932

5344898

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