Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice. - Wikidata - wikimedia - TriplyDB

Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice.

Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice.

http://www.wikidata.org/entity/Q34080154

about

Pathogenesis of renal disease in systemic lupus erythematosus--the role of autoantibodies and lymphocytes subset abnormalities Nuclear Factor Erythroid 2-related Factor 2 Deficiency Exacerbates Lupus Nephritis in B6/lpr mice by Regulating Th17 Cell Function P2X7 blockade attenuates murine lupus nephritis by inhibiting activation of the NLRP3/ASC/caspase 1 pathway.T-helper 17 cell cytokines and interferon type I: partners in crime in systemic lupus erythematosus?IL-23 promotes TCR-mediated negative selection of thymocytes through the upregulation of IL-23 receptor and RORγt.T cells as therapeutic targets in SLE.Prevention of immune nephritis by the small molecular weight immunomodulator iguratimod in MRL/lpr mice.Autocrine IFNγ controls the regulatory function of lymphoproliferative double negative T cells Increased interleukin-23 receptor(+) T cells in peripheral blood mononuclear cells of patients with systemic lupus erythematosus.The Th17-defining transcription factor RORγt promotes glomerulonephritis FcRγ controls the fas-dependent regulatory function of lymphoproliferative double negative T cells The NZM2410-derived lupus susceptibility locus Sle2c1 increases Th17 polarization and induces nephritis in fas-deficient mice Distinct p21 requirements for regulating normal and self-reactive T cells through IFN-γ production.Estrogen increases, whereas IL-27 and IFN-gamma decrease, splenocyte IL-17 production in WT mice IL-17 and IL-23 in lupus nephritis - association to histopathology and response to treatment.Calcium signaling in systemic lupus erythematosus T cells: a treatment target Abnormalities of T cell signaling in systemic lupus erythematosus.Reduced B lymphoid kinase (Blk) expression enhances proinflammatory cytokine production and induces nephrosis in C57BL/6-lpr/lpr mice.IRF4 deficiency abrogates lupus nephritis despite enhancing systemic cytokine production Signal transduction pathways and transcriptional regulation in Th17 cell differentiation The dysregulation of cytokine networks in systemic lupus erythematosus.The role of cytokine in the lupus nephritis.The current concept of T (h) 17 cells and their expanding role in systemic lupus erythematosus.Cytokine regulation of B-cell migratory behavior favors formation of germinal centers in autoimmune disease.Serum interleukin-17 levels are associated with nephritis in childhood-onset systemic lupus erythematosus.Roles of pro- and anti-inflammatory cytokines in the pathogenesis of SLE T cell signaling abnormalities contribute to aberrant immune cell function and autoimmunity.Defect in regulatory B-cell function and development of systemic autoimmunity in T-cell Ig mucin 1 (Tim-1) mucin domain-mutant mice.The Role of IL-17 and Th17 Lymphocytes in Autoimmune Diseases Effector T-cell subsets in systemic lupus erythematosus: update focusing on Th17 cells.Glucose Oxidation Is Critical for CD4+ T Cell Activation in a Mouse Model of Systemic Lupus Erythematosus.Isoflurane attenuates murine lupus nephritis by inhibiting NLRP3 inflammasome activation.Interleukin-17 cytokines are critical in development of fatal lupus glomerulonephritis IL-21 receptor is required for the systemic accumulation of activated B and T lymphocytes in MRL/MpJ-Fas(lpr/lpr)/J mice.Treatment with anti-interleukin 23 antibody ameliorates disease in lupus-prone mice Effects of 1,25-dihydroxyvitamin D3 on IL-17/IL-23 axis, IFN-γ and IL-4 expression in systemic lupus erythematosus induced mice model.Arginase-1-dependent promotion of TH17 differentiation and disease progression by MDSCs in systemic lupus erythematosus.ICER is requisite for Th17 differentiation.Metabolic Factors that Contribute to Lupus Pathogenesis CaMK4-dependent activation of AKT/mTOR and CREM-α underlies autoimmunity-associated Th17 imbalance

P2860

Q28080571-E15DB489-D02C-4E69-9680-2530F57DCE06 Q28396263-596B2672-8ECD-4BB7-8FB6-CCCA1D7201AB Q30409205-13D55E51-74D6-4217-ABEF-7B83BFD3ED96 Q33765598-AA7018E5-5157-4F4D-AD02-DD1152E03E74 Q34060009-5F702E3D-FBA6-4A36-858D-DB18E78C228A Q34075552-207BA012-81A6-486C-8AA8-7189C15DF0CA Q34278189-88D236D1-394B-4411-A2D7-A57452804D2C Q34451062-DC481D40-7D49-49D9-B418-25E0077DAA60 Q34614005-3CF0B335-0376-430E-91EF-1EC10347E31F Q34695374-6EF9CEA4-FE9A-4E1F-8062-271E36752755 Q34769749-95CA1F6D-4774-4035-8415-39EB560B8F1B Q34821636-8A2A8D56-32C3-4FAD-9A67-838B8D6D4E6A Q34877991-C99513FF-2D67-4D3C-9BC8-0D48968D05DF Q34989645-94A10BB6-E8D5-4505-8296-D7A52DF94254 Q35078183-BE8F9B60-C4CE-456A-8883-F620FE4DD782 Q35082332-A74DF395-B24D-4666-BE19-B0306C187A91 Q35092855-5F972C27-41B3-4B05-AB2A-024A06FF5AE1 Q35122990-D9B4658A-F21D-4D48-870D-BEE8BAE4E3D2 Q35142708-A63C594D-C6C6-4A99-90F9-6C6A3BABF8EC Q35238480-CCF2FA97-F8DE-4E18-A19F-8A3DDF7A2346 Q35294513-6C6E5ECA-1534-427A-A344-D0600FEA2F65 Q35418333-6DAAFD59-ACA4-4789-98F1-5C50513C9F3C Q35579076-DB122B45-829B-4C6D-BD87-388358A553EE Q35644984-4DDE76A3-DDD6-4491-9ED6-BD64199AA50F Q35666611-8D7BB5AD-B195-4733-872B-37417B2B1871 Q35829864-3F9946AA-3451-45C3-B3A0-1E8D0D6A6764 Q35836112-E5D03BFF-08F6-45BD-9B8E-C530660AEC96 Q36132667-5E136D0B-3D81-4380-BE9E-F428FE13066A Q36249305-5EFB77B4-E7E0-462E-A921-185E4838817E Q36373858-98923C9B-0FD2-4688-92E3-1B9A92B9B067 Q36387092-FA90B10B-1A7D-45EC-AA60-B6327096DA38 Q36411613-EA2C0275-B046-4F0B-81BB-A3E4A49B404A Q36676453-A3778F07-DDB4-4897-84EE-E801146EEE24 Q36745302-C978F568-0BCE-4F66-BF18-8CDB8ED5A50E Q36948466-15DD73AA-F029-4CF7-BFE0-1913FAFC7DB3 Q36953169-462C42D6-6F05-4BC8-A847-5515844BDEC9 Q36974463-8DC98A0C-64D8-4075-B803-86CE8FBE7106 Q37324043-D29317F8-E32F-442B-B50E-282A3EC159FE Q37612885-3BD41BF6-4798-4421-866D-F293E439CCC9 Q37726675-E401167C-6B2D-4398-BA26-5D1206EC9032

P2860

Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice.

http://www.wikidata.org/entity/Q34080154

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մարտին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@ast

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@en

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@nl

type

label

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@ast

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@en

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@nl

prefLabel

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@ast

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@en

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@nl

P1433

Q34080154-ED302E87-9579-4CA9-A8AC-114A49F76067

P1476

Q34080154-4082EC31-AE0C-4EB0-82C1-A791D98BECC9

P2093

Q34080154-01A92BE2-ACAC-44F0-8E75-DF9E5E10CE0C

Q34080154-73827A40-1107-436E-8CAE-ACC1872E0B7F

Q34080154-7536AF7A-91C5-44CE-89F0-237F9951A028

Q34080154-81AA38D0-BED6-486A-9F50-C3DD26B0F8F3

Q34080154-C26AE682-1E24-4449-90E0-BC0DA90E6079

P2860

Q34080154-0E866FB0-9D47-4B8B-9FB7-3CB634578E76

Q34080154-35B26DC0-28C3-45CD-885C-FEDBBE663CCE

Q34080154-79FD0F01-2206-4271-8561-D229E96D791E

Q34080154-C2985FBF-9FC6-4AE6-8F49-CC4F07B4DAD5

Q34080154-C68EF2CE-94C5-4FCF-B113-5C840797FDB3

Q34080154-C6FEA3F3-4E5F-4176-AEC6-F83026FD303C

Q34080154-CB3A5172-F4CD-46D2-8D35-FCED3D05898B

Q34080154-D0BDBA74-C45E-4BAC-AE95-8CAE9145C56A

Q34080154-E31D0B7C-5452-4AFB-AB4F-B87186DD74CE

P304

Q34080154-405C06C6-6F30-427F-88D0-AF1CFA603626

P31

Q34080154-985F0773-8555-4479-9431-7993D4EBA769

P356

Q34080154-A0799C51-A142-48E1-88E8-A440B879C7EE

P407

Q34080154-CD0D1A90-E066-4A28-A67B-FE58AFFEB8B2

P433

Q34080154-FE5AAAF9-2C0D-42C9-8383-E5E55B3E8980

P478

Q34080154-1F289F8C-659E-4EFE-83F8-4C3D34017C1C

P577

Q34080154-B543751D-CCDA-4616-8B13-836D985715A9

P5875

Q34080154-898CAAA4-D8FC-4FF9-8155-E334CC486577

P698

Q34080154-AA00CA52-8E2F-481A-9472-D7DB658A983D

P932

Q34080154-98EAB9F9-02C2-4E5E-96C0-35465570FC6B

P356

JIMMUNOL.0903595

P1433

Journal of Immunology

P1476

Cutting edge: IL-23 receptor d ...... ritis in C57BL/6-lpr/lpr mice.

@en

P2093

George C Tsokos

http://www.w3.org/2001/XMLSchema#string

Mohamed Oukka

http://www.w3.org/2001/XMLSchema#string

Vasileios C Kyttaris

http://www.w3.org/2001/XMLSchema#string

Vijay K Kuchroo

http://www.w3.org/2001/XMLSchema#string

Zheng Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

IL-17 and Th17 Cells

Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

Human TCR-alpha beta+ CD4- CD8- T cells can derive from CD8+ T cells and display an inflammatory effector phenotype

Expanded double negative T cells in patients with systemic lupus erythematosus produce IL-17 and infiltrate the kidneys

Treatment of murine lupus with cDNA encoding IFN-gammaR/Fc.

Immune cells and cytokines in systemic lupus erythematosus: an update.

ICOS-dependent extrafollicular helper T cells elicit IgG production via IL-21 in systemic autoimmunity.

Cutting edge: IL-23 receptor gfp reporter mice reveal distinct populations of IL-17-producing cells

The role of IL-23/IL-17 axis in lupus nephritis.

P304

4605-4609

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4049/JIMMUNOL.0903595

http://www.w3.org/2001/XMLSchema#string

P407

P433

9

http://www.w3.org/2001/XMLSchema#string

P478

184

http://www.w3.org/2001/XMLSchema#string

P577

2010-03-22T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

42389618

http://www.w3.org/2001/XMLSchema#string

P698

20308633

http://www.w3.org/2001/XMLSchema#string

P932

2926666

http://www.w3.org/2001/XMLSchema#string