TSC22D4 is a molecular output of hepatic wasting metabolism. - Wikidata - wikimedia - TriplyDB

TSC22D4 is a molecular output of hepatic wasting metabolism.

TSC22D4 is a molecular output of hepatic wasting metabolism.

http://www.wikidata.org/entity/Q42045525

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Understanding cachexia as a cancer metabolism syndrome Mechanisms of metabolic dysfunction in cancer-associated cachexia Quercetin supplementation attenuates the progression of cancer cachexia in ApcMin/+ mice.microRNA-379 couples glucocorticoid hormones to dysfunctional lipid homeostasis.Liver inflammation and metabolic signaling in ApcMin/+ mice: the role of cachexia progression.Transcriptional co-factor Transducin beta-like (TBL) 1 acts as a checkpoint in pancreatic cancer malignancy.Ataxin-10 is part of a cachexokine cocktail triggering cardiac metabolic dysfunction in cancer cachexia.STAT3 in the systemic inflammation of cancer cachexia.Overexpression of TSC-22 (transforming growth factor- β-stimulated clone-22) causes marked obesity, splenic abnormality and B cell lymphoma in transgenic mice Control of diabetic hyperglycaemia and insulin resistance through TSC22D4.Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism.Short-term pyrrolidine dithiocarbamate administration attenuates cachexia-induced alterations to muscle and liver in ApcMin/+ mice.Complete remission of cancer in late-stage disease by radiation and transfer of allogeneic MHC-matched immune T cells: lessons from GvL studies in animals.Role of interleukin-6 in cachexia: therapeutic implications.The IGF2 mRNA binding protein p62/IGF2BP2-2 induces fatty acid elongation as a critical feature of steatosis.Oxygen drives hepatocyte differentiation and phenotype stability in liver cell lines.

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TSC22D4 is a molecular output of hepatic wasting metabolism.

http://www.wikidata.org/entity/Q42045525

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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2013年學術文章

@zh-hant

name

TSC22D4 is a molecular output of hepatic wasting metabolism.

@en

TSC22D4 is a molecular output of hepatic wasting metabolism.

@nl

type

label

TSC22D4 is a molecular output of hepatic wasting metabolism.

@en

TSC22D4 is a molecular output of hepatic wasting metabolism.

@nl

prefLabel

TSC22D4 is a molecular output of hepatic wasting metabolism.

@en

TSC22D4 is a molecular output of hepatic wasting metabolism.

@nl

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EMBO Molecular Medicine

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TSC22D4 is a molecular output of hepatic wasting metabolism.

@en

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Allan Jones

http://www.w3.org/2001/XMLSchema#string

Barbara Leuchs

http://www.w3.org/2001/XMLSchema#string

Carolyn Algire

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Carsten Sticht

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Daniela Strzoda

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Geesje M Dallinga-Thie

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Karin Müller-Decker

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Kilian Friedrich

http://www.w3.org/2001/XMLSchema#string

Manfred Kögl

http://www.w3.org/2001/XMLSchema#string

Michaela Schäfer

http://www.w3.org/2001/XMLSchema#string

P2860

Transforming growth factor-beta-stimulated clone-22 is a member of a family of leucine zipper proteins that can homo- and heterodimerize and has transcriptional repressor activity

Cancer cachexia: mediators, signaling, and metabolic pathways

Angptl4 upregulates cholesterol synthesis in liver via inhibition of LPL- and HL-dependent hepatic cholesterol uptake

Cholesterol and bile acid metabolism are impaired in mice lacking the nuclear oxysterol receptor LXR alpha

Specific TSC22 domain transcripts are hypertonically induced and alternatively spliced to protect mouse kidney cells during osmotic stress

FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells

CREB regulates hepatic gluconeogenesis through the coactivator PGC-1

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

ApoB100-LDL acts as a metabolic signal from liver to peripheral fat causing inhibition of lipolysis in adipocytes.

Madm (Mlf1 adapter molecule) cooperates with Bunched A to promote growth in Drosophila.

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294-308

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scholarly article

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10.1002/EMMM.201201869

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2

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5

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Mauricio Berriel Diaz

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2013-01-11T00:00:00Z

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234105559

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3569644

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