Testing mutual exclusivity of ETS rearranged prostate cancer.
about
Exome sequencing identifies recurrent SPOP, FOXA1 and MED12 mutations in prostate cancerThe mutational landscape of prostate cancerGenetic profiling to determine risk of relapse-free survival in high-risk localized prostate cancer.Novel RNA hybridization method for the in situ detection of ETV1, ETV4, and ETV5 gene fusions in prostate cancer.Discovery of non-ETS gene fusions in human prostate cancer using next-generation RNA sequencing.The ETS gene ETV4 is required for anchorage-independent growth and a cell proliferation gene expression program in PC3 prostate cells.Gene signatures ESC, MYC and ERG-fusion are early markers of a potentially dangerous subtype of prostate cancer.Analysis of the genetic phylogeny of multifocal prostate cancer identifies multiple independent clonal expansions in neoplastic and morphologically normal prostate tissue.Interaction of the Androgen Receptor, ETV1, and PTEN Pathways in Mouse Prostate Varies with Pathological Stage and Predicts Cancer Progression.Oncogenic activation of ERG: A predominant mechanism in prostate cancer.Specific and redundant activities of ETV1 and ETV4 in prostate cancer aggressiveness revealed by co-overexpression cellular contextsCharacterization of 1577 primary prostate cancers reveals novel biological and clinicopathologic insights into molecular subtypes.Heterogeneity of ERG expression in prostate cancer: a large section mapping study of entire prostatectomy specimens from 125 patients.Distinct ERG rearrangement prevalence in prostate cancer: higher frequency in young age and in low PSA prostate cancer.The TMPRSS2:ERG rearrangement, ERG expression, and prostate cancer outcomes: a cohort study and meta-analysisEarly detection of clinically significant prostate cancer at diagnosis: a prospective study using a novel panel of TMPRSS2:ETS fusion gene markersSPOP mutations in prostate cancer across demographically diverse patient cohorts.Report of a consensus meeting on focal low dose rate brachytherapy for prostate cancer.The complexity of prostate cancer: genomic alterations and heterogeneity.Recurrent gene fusions in prostate cancer: their clinical implications and uses.FLI1 is a novel ETS transcription factor involved in gene fusions in prostate cancer.Intrafocal heterogeneity of ERG protein expression and gene fusion pattern in prostate cancer.The genomic evolution of human prostate cancer.Prostate cancer ETS rearrangements switch a cell migration gene expression program from RAS/ERK to PI3K/AKT regulation.ETV1 positively regulates transcription of tumor suppressor ARF.Rationale for the development of alternative forms of androgen deprivation therapy.High-throughput detection of fusion genes in cancer using the Sequenom MassARRAY platform.Intratumoral and Intertumoral Genomic Heterogeneity of Multifocal Localized Prostate Cancer Impacts Molecular Classifications and Genomic Prognosticators.ERG overexpression and PTEN status predict capsular penetration in prostate carcinoma.Antibody-based detection of ERG rearrangements in prostate core biopsies, including diagnostically challenging cases: ERG staining in prostate core biopsies.ERG rearrangement and protein expression in the progression to castration-resistant prostate cancer.Molecular heterogeneity of localized prostate cancer: more different than alike.An Interaction with Ewing's Sarcoma Breakpoint Protein EWS Defines a Specific Oncogenic Mechanism of ETS Factors Rearranged in Prostate Cancer.ERG is specifically associated with ETS-2 and ETV-4, but not with ETS-1, in prostate cancer.ERG overexpression plus SLC45A3 (prostein) and PTEN expression loss: Strong association of the triple hit phenotype with an aggressive pathway of prostate cancer progression.IDH1R132H Promotes Malignant Transformation of Benign Prostatic Epithelium by Dysregulating MicroRNAs: Involvement of IGF1R-AKT/STAT3 Signaling Pathway.Comprehensive Determination of Prostate Tumor ETS Gene Status in Clinical Samples Using the CLIA Decipher Assay.High concordance of TMPRSS-ERG fusion between primary prostate cancer and its lymph node metastases
P2860
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P2860
Testing mutual exclusivity of ETS rearranged prostate cancer.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Testing mutual exclusivity of ETS rearranged prostate cancer.
@en
Testing mutual exclusivity of ETS rearranged prostate cancer.
@nl
type
label
Testing mutual exclusivity of ETS rearranged prostate cancer.
@en
Testing mutual exclusivity of ETS rearranged prostate cancer.
@nl
prefLabel
Testing mutual exclusivity of ETS rearranged prostate cancer.
@en
Testing mutual exclusivity of ETS rearranged prostate cancer.
@nl
P2093
P2860
P1476
Testing mutual exclusivity of ETS rearranged prostate cancer.
@en
P2093
Ashley M Santa-Cruz
Ashutosh K Tewari
Chol S Yun
Christopher J LaFargue
Dorothee Pflueger
Gary A Pestano
Janice P Riley
Jerry W Kosmeder
Karl E Garsha
P2860
P2888
P304
P356
10.1038/LABINVEST.2010.179
P577
2010-10-25T00:00:00Z
P5875
P6179
1021064973