The reversible Ca2+-induced permeabilization of rat liver mitochondria.
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Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition poreThe roles of phosphate and the phosphate carrier in the mitochondrial permeability transition poreQuantitative evaluation of mitochondrial calcium content in rat cortical neurones following a glutamate stimulusCyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosisThe mitochondrial permeability transition pore and its role in cell deathGenetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathyFluctuations in mitochondrial membrane potential caused by repetitive gating of the permeability transition poreRegulation of the mitochondrial permeability transition in kidney proximal tubules and its alteration during hypoxia-reoxygenationMitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Mitochondrial intermembrane junctional complexes and their role in cell death.Mitofusins and the mitochondrial permeability transition: the potential downside of mitochondrial fusionCiclopirox protects mitochondria from hydrogen peroxide toxicity.A Novel Nicotinamide Adenine Dinucleotide Correction Method for Mitochondrial Ca(2+) Measurement with FURA-2-FF in Single Permeabilized Ventricular Myocytes of Rat.Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Dysregulation of mitochondrial calcium signaling and superoxide flashes cause mitochondrial genomic DNA damage in Huntington disease.Synergistic triggering of superoxide flashes by mitochondrial Ca2+ uniport and basal reactive oxygen species elevation.Crosstalk signaling between mitochondrial Ca2+ and ROS.Therapeutic strategies to protect the immature newborn myocardium during resuscitation following asphyxia.The mitochondrial permeability transition pore: a mystery solved?The entrapment of the Ca2+ indicator arsenazo III in the matrix space of rat liver mitochondria by permeabilization and resealing. Na+-dependent and -independent effluxes of Ca2+ in arsenazo III-loaded mitochondria.Mitochondrial flashes: new insights into mitochondrial ROS signalling and beyond.'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria.Cyclosporin A blocks 6-hydroxydopamine-induced efflux of Ca2+ from mitochondria without inactivating the mitochondrial inner-membrane poreOxidative damage to mitochondria is mediated by the Ca(2+)-dependent inner-membrane permeability transition.Ion modulation of membrane permeability: effect of cations on intact cells and on cells and phospholipid bilayers treated with pore-forming agents.Mitochondrial dysfunction induced by sertraline, an antidepressant agent.Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress.On the interactions of Ca2+ and cyclosporin A with a mitochondrial inner membrane pore: a study using cobaltammine complex inhibitors of the Ca2+ uniporter.Inhibition by spermine of the inner membrane permeability transition of isolated rat heart mitochondria.Cholestasis induced by chronic treatment with alpha-naphthyl-isothiocyanate (ANIT) affects rat renal mitochondrial bioenergetics.Effects of the membrane potential upon the Ca(2+)- and cumene hydroperoxide-induced permeabilization of the inner mitochondrial membrane.Effects of the general anaesthetic Propofol on the Ca2(+)-induced permeabilization of rat liver mitochondria.Excitotoxic injury to mitochondria isolated from cultured neurons.Regulation of superoxide flashes by transient and steady mitochondrial calcium elevations.Prooxidant-induced Ca2+ release from liver mitochondria. Specific versus nonspecific pathways.Control of the pro-oxidant-dependent calcium release from intact liver mitochondria.Different approaches to modeling analysis of mitochondrial swelling.Administration of the Mitochondrial Permeability Transition Pore Inhibitor, TRO40303, prior to Primary Percutaneous Coronary Intervention, Does Not Affect the Levels of Pro-Inflammatory Cytokines or Acute-Phase Proteins.Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition.
P2860
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P2860
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@en
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@nl
type
label
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@en
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@nl
prefLabel
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@en
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@nl
P2860
P356
P1433
P1476
The reversible Ca2+-induced permeabilization of rat liver mitochondria.
@en
P2093
Al-Nasser I
Crompton M
P2860
P356
10.1042/BJ2390019
P407
P577
1986-10-01T00:00:00Z