Transcriptional suppression of the human T-cell leukemia virus type I long terminal repeat occurs by an unconventional interaction of a CREB factor with the R region.
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HUB1, a novel Krüppel type zinc finger protein, represses the human T cell leukemia virus type I long terminal repeat-mediated expressionAn interferon regulatory factor binding site in the U5 region of the bovine leukemia virus long terminal repeat stimulates Tax-independent gene expressionIn vivo protein binding and functional analysis of cis-acting elements in the U3 region of the bovine leukemia virus long terminal repeatTranscriptional regulation of porcine endogenous retroviruses released from porcine and infected human cells by heterotrimeric protein complex NF-Y and impact of immunosuppressive drugs.Human T cell leukemia virus type I and neurologic disease: events in bone marrow, peripheral blood, and central nervous system during normal immune surveillance and neuroinflammation.Transcription regulatory complexes bind the human T-cell leukemia virus 5' and 3' long terminal repeats to control gene expression.Constitutive activation of different Jak tyrosine kinases in human T cell leukemia virus type 1 (HTLV-1) tax protein or virus-transformed cellsThe cellular stress response enhances human T-cell lymphotropic virus type 1 basal gene expression through the core promoter region of the long terminal repeat.Wild-type but not Alzheimer-mutant amyloid precursor protein confers resistance against p53-mediated apoptosis.Targeting HTLV-1 activation of NFκB in mouse models and ATLL patients.Autoantigen Ku protein is involved in DNA binding proteins which recognize the U5 repressive element of human T-cell leukemia virus type I long terminal repeat.Binding and functional effects of transcriptional factor Sp1 on the murine interleukin-6 promotor.Immortalization of CD4(+) and CD8(+) T lymphocytes by human T-cell leukemia virus type 1 Tax mutants expressed in a functional molecular clone.Sp1 family proteins recognize the U5 repressive element of the long terminal repeat of human T cell leukemia virus type I through binding to the CACCC core motif.Upstream stimulatory factors binding to an E box motif in the R region of the bovine leukemia virus long terminal repeat stimulates viral gene expression.
P2860
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P2860
Transcriptional suppression of the human T-cell leukemia virus type I long terminal repeat occurs by an unconventional interaction of a CREB factor with the R region.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
Transcriptional suppression of ...... CREB factor with the R region.
@en
Transcriptional suppression of ...... CREB factor with the R region.
@nl
type
label
Transcriptional suppression of ...... CREB factor with the R region.
@en
Transcriptional suppression of ...... CREB factor with the R region.
@nl
prefLabel
Transcriptional suppression of ...... CREB factor with the R region.
@en
Transcriptional suppression of ...... CREB factor with the R region.
@nl
P2093
P2860
P356
P1476
Transcriptional suppression of ...... CREB factor with the R region.
@en
P2093
P2860
P304
P356
10.1128/MCB.14.8.5371
P407
P577
1994-08-01T00:00:00Z