Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades. - Wikidata - wikimedia - TriplyDB

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

http://www.wikidata.org/entity/Q42098324

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Targeting BMP signalling in cardiovascular disease and anaemia Vascularisation of the central nervous system Context-specific interactions between Notch and ALK1 cannot explain ALK1-associated arteriovenous malformations.Excessive vascular sprouting underlies cerebral hemorrhage in mice lacking αVβ8-TGFβ signaling in the brain.Notch2 regulates BMP signaling and epithelial morphogenesis in the ciliary body of the mouse eye A novel mechanism for sevoflurane preconditioning-induced neuroprotection.PTEN mediates Notch-dependent stalk cell arrest in angiogenesis Bone morphogenetic protein 2 signaling negatively modulates lymphatic development in vertebrate embryos.Hes1, an important gene for activation of hepatic stellate cells, is regulated by Notch1 and TGF-β/BMP signaling.Id proteins regulate capillary repair and perivascular cell proliferation following ischemia-reperfusion injury Endothelial depletion of Acvrl1 in mice leads to arteriovenous malformations associated with reduced endoglin expression.Specific bone cells produce DLL4 to generate thymus-seeding progenitors from bone marrow.Serum induces transcription of Hey1 and Hey2 genes by Alk1 but not Notch signaling in endothelial cells SMAD1 and SMAD5 Expression Is Coordinately Regulated by FLI1 and GATA2 during Endothelial Development.When it is better to regress: dynamics of vascular pruning.Characterization of the BAC Id3-enhanced green fluorescent protein transgenic mouse line for in vivo imaging of astrocytes miR-342-5p Is a Notch Downstream Molecule and Regulates Multiple Angiogenic Pathways Including Notch, Vascular Endothelial Growth Factor and Transforming Growth Factor β Signaling.Alk1 and Alk5 inhibition by Nrp1 controls vascular sprouting downstream of Notch.BMP signalling differentially regulates distinct haematopoietic stem cell types BMP9 and BMP10 are critical for postnatal retinal vascular remodeling.BMP9/ALK1 inhibits neovascularization in mouse models of age-related macular degeneration.BMP-SMAD signalling output is highly regionalized in cardiovascular and lymphatic endothelial networks.Vascular Integrity in the Pathogenesis of Brain Arteriovenous Malformation.Smad1/5/8 are myogenic regulators of murine and human mesoangioblasts The importance of microglia in the development of the vasculature in the central nervous system Matrix Gla protein reinforces angiogenic resolution.Correction: The importance of microglia in the development of the vasculature in the central nervous system Circulating Bmp10 acts through endothelial Alk1 to mediate flow-dependent arterial quiescence SMAD1 deficiency in either endothelial or smooth muscle cells can predispose mice to pulmonary hypertension Notch regulates BMP responsiveness and lateral branching in vessel networks via SMAD6.Alk3/Alk3b and Smad5 mediate BMP signaling during lymphatic development in zebrafish.Endothelial signaling and the molecular basis of arteriovenous malformation.Tumor-infiltrating myeloid cells activate Dll4/Notch/TGF-β signaling to drive malignant progression Neurovascular development and links to disease.Recent molecular discoveries in angiogenesis and antiangiogenic therapies in cancer BMP growth factor signaling in a biomechanical context.The role of BMPs in endothelial cell function and dysfunction.VEGF, Notch and TGFβ/BMPs in regulation of sprouting angiogenesis and vascular patterning.Cell signaling pathways involved in hematopoietic stem cell specification.Notch signal integration in the vasculature during remodeling

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Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

http://www.wikidata.org/entity/Q42098324

description

2012 nî lūn-bûn

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2012年の論文

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2012年论文

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2012年论文

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2012年论文

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name

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@en

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@nl

type

label

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@en

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@nl

prefLabel

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@en

Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

@nl

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J.DEVCEL.2012.01.007

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Developmental Cell

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Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.

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Annick Francis

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Elizabeth J Robertson

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Elke Maas

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Iván M Moya

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Karen Beets

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Lieve Umans

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Ward Sents

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P2860

Id sustains Hes1 expression to inhibit precocious neurogenesis by releasing negative autoregulation of Hes1

The cyclic gene Hes1 contributes to diverse differentiation responses of embryonic stem cells

Delta-like ligand 4 (Dll4) is induced by VEGF as a negative regulator of angiogenic sprouting

Tipping the balance: robustness of tip cell selection, migration and fusion in angiogenesis

Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

Id proteins in cell cycle control and cellular senescence

Oscillations in notch signaling regulate maintenance of neural progenitors

Dll4 signalling through Notch1 regulates formation of tip cells during angiogenesis

VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodia

Endothelial signalling by the Notch ligand Delta-like 4 restricts angiogenesis.

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Christine Mummery

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Paulo N G Pereira

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