Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
about
Targeting BMP signalling in cardiovascular disease and anaemiaVascularisation of the central nervous systemContext-specific interactions between Notch and ALK1 cannot explain ALK1-associated arteriovenous malformations.Excessive vascular sprouting underlies cerebral hemorrhage in mice lacking αVβ8-TGFβ signaling in the brain.Notch2 regulates BMP signaling and epithelial morphogenesis in the ciliary body of the mouse eyeA novel mechanism for sevoflurane preconditioning-induced neuroprotection.PTEN mediates Notch-dependent stalk cell arrest in angiogenesisBone morphogenetic protein 2 signaling negatively modulates lymphatic development in vertebrate embryos.Hes1, an important gene for activation of hepatic stellate cells, is regulated by Notch1 and TGF-β/BMP signaling.Id proteins regulate capillary repair and perivascular cell proliferation following ischemia-reperfusion injuryEndothelial depletion of Acvrl1 in mice leads to arteriovenous malformations associated with reduced endoglin expression.Specific bone cells produce DLL4 to generate thymus-seeding progenitors from bone marrow.Serum induces transcription of Hey1 and Hey2 genes by Alk1 but not Notch signaling in endothelial cellsSMAD1 and SMAD5 Expression Is Coordinately Regulated by FLI1 and GATA2 during Endothelial Development.When it is better to regress: dynamics of vascular pruning.Characterization of the BAC Id3-enhanced green fluorescent protein transgenic mouse line for in vivo imaging of astrocytesmiR-342-5p Is a Notch Downstream Molecule and Regulates Multiple Angiogenic Pathways Including Notch, Vascular Endothelial Growth Factor and Transforming Growth Factor β Signaling.Alk1 and Alk5 inhibition by Nrp1 controls vascular sprouting downstream of Notch.BMP signalling differentially regulates distinct haematopoietic stem cell typesBMP9 and BMP10 are critical for postnatal retinal vascular remodeling.BMP9/ALK1 inhibits neovascularization in mouse models of age-related macular degeneration.BMP-SMAD signalling output is highly regionalized in cardiovascular and lymphatic endothelial networks.Vascular Integrity in the Pathogenesis of Brain Arteriovenous Malformation.Smad1/5/8 are myogenic regulators of murine and human mesoangioblastsThe importance of microglia in the development of the vasculature in the central nervous systemMatrix Gla protein reinforces angiogenic resolution.Correction: The importance of microglia in the development of the vasculature in the central nervous systemCirculating Bmp10 acts through endothelial Alk1 to mediate flow-dependent arterial quiescenceSMAD1 deficiency in either endothelial or smooth muscle cells can predispose mice to pulmonary hypertensionNotch regulates BMP responsiveness and lateral branching in vessel networks via SMAD6.Alk3/Alk3b and Smad5 mediate BMP signaling during lymphatic development in zebrafish.Endothelial signaling and the molecular basis of arteriovenous malformation.Tumor-infiltrating myeloid cells activate Dll4/Notch/TGF-β signaling to drive malignant progressionNeurovascular development and links to disease.Recent molecular discoveries in angiogenesis and antiangiogenic therapies in cancerBMP growth factor signaling in a biomechanical context.The role of BMPs in endothelial cell function and dysfunction.VEGF, Notch and TGFβ/BMPs in regulation of sprouting angiogenesis and vascular patterning.Cell signaling pathways involved in hematopoietic stem cell specification.Notch signal integration in the vasculature during remodeling
P2860
Q26783734-0ABB54B0-C1C6-4072-AFBB-0C8BBED449D2Q26801786-60B81EE6-C77D-4EF9-800C-8EF25A3C5AB9Q27302123-98FC25E8-6C51-4912-AA87-B47E4A5832E5Q27346828-48EE819B-8BF8-4681-83DF-3119837ABB81Q28593168-9F6020E8-D092-4C2A-87C7-CD054D8B637BQ30416485-C78A1C10-FD7C-4ACF-8C5D-948EA92DFF07Q33663049-52F1E8A4-758D-4CB6-8749-F72BCCA524ECQ33719268-58DBF97E-0A2C-425E-A4F7-B5AA4C976482Q34986010-3D0E8134-4F97-4822-88D3-EC03722E7FA5Q35091758-483018EC-B86A-4BAB-A9B4-E87DFB0B5615Q35181476-C02AA74E-182D-49A0-82C8-25C5DA4C0AE0Q35568443-8F635FF9-9518-4F91-B25C-2BA9607A980FQ35584425-DDAECDCF-E367-4B32-9066-29DE28282F99Q35626101-A567D044-B91C-4AF1-B8A3-4FADF3854A58Q35633027-FFCBEC90-5882-43B0-93AB-3B1B0B20CA1CQ35775185-926FD6AA-B5B8-4D58-8350-2226EB50AB07Q35917632-D2068FC1-9284-4462-BC4A-EDC3D7F54175Q36018663-0771659E-DE9F-4BF8-AFE9-B3D10FFE4DA7Q36018760-90478366-CAEB-4336-9813-347AE3D8F68BQ36057921-D51BCE69-31F6-44CA-AA29-17461DDBC54CQ36102528-E8A48536-0C3E-4C79-9BC2-CE4F4E8B8AD1Q36159603-E1103A9B-011F-4C95-828C-8A139D5844D8Q36304260-2356BAD2-4A06-43CD-8732-FF5CF59B9267Q36455338-179E9B51-23A1-43CC-9ADB-53952439F9B5Q36643118-14782544-D9D4-42A1-A47B-0A56F6048AD6Q36774809-DFD75DCF-082C-4304-92AB-BACED454EEBDQ36964429-6875F827-38B4-408D-B947-F77A1A2AA702Q37080802-C0BB4F15-F75B-46F0-9D7B-D3DEBC34DAC0Q37087816-BA7F3789-7190-4974-82E3-5AFE6D22C0FAQ37421707-8690D704-A040-4573-8420-7C2A436A76B0Q37671260-25B9F093-9BC4-40D3-AAF0-423C1507CC33Q37671796-C197ACBF-E629-4784-B531-FB71830007E8Q37700357-79A4FDC6-A3A9-4D1F-83C2-976D239312BDQ38087874-5C880993-2C73-4AE0-8CC1-4A523BC6B461Q38125964-09C1C3D1-336C-4017-B3ED-50117497A7B1Q38152403-4EBDA3C6-E60F-4F87-9669-C261A5F5AFC2Q38218507-34DEE195-BFFF-4095-BF66-5C283A2E67F6Q38268447-D08B2D21-250B-4FA1-BC24-7D880012A2C3Q38275152-3B9A6872-BAC6-40B1-AD41-6D943A298B83Q38281219-51DF0B9A-0424-4589-82B0-1D3B18120F91
P2860
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@en
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@nl
type
label
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@en
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@nl
prefLabel
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@en
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@nl
P2093
P2860
P50
P1433
P1476
Stalk cell phenotype depends on integration of Notch and Smad1/5 signaling cascades.
@en
P2093
Annick Francis
Elizabeth J Robertson
Iván M Moya
Karen Beets
Lieve Umans
Ward Sents
P2860
P304
P356
10.1016/J.DEVCEL.2012.01.007
P407
P577
2012-02-23T00:00:00Z