Ccr1 deficiency reduces inflammatory remodelling and preserves left ventricular function after myocardial infarction.
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Chemokine contribution in stem cell engraftment into the infarcted myocardiumCCR5 signaling suppresses inflammation and reduces adverse remodeling of the infarcted heart, mediating recruitment of regulatory T cellsCC chemokine receptor 5 deletion impairs macrophage activation and induces adverse remodeling following myocardial infarction.Impact of aging vs. estrogen loss on cardiac gene expression: estrogen replacement and inflammationEarly postmyocardial infarction survival in Murphy Roths Large mice is mediated by attenuated apoptosis and inflammation but depends on genetic backgroundCellular repressor of E1A-stimulated genes attenuates cardiac hypertrophy and fibrosis.The immune system and cardiac repair.Repetitive transplantation of different cell types sequentially improves heart function after infarction.Crocetin protects against cardiac hypertrophy by blocking MEK-ERK1/2 signalling pathway.Exogenous administration of a recombinant variant of TWEAK impairs healing after myocardial infarction by aggravation of inflammation.Promiscuous drugs as therapeutics for chemokine receptors.International Union of Basic and Clinical Pharmacology. [corrected]. LXXXIX. Update on the extended family of chemokine receptors and introducing a new nomenclature for atypical chemokine receptors.Chemokines and cardiac fibrosis.Role of CCL7 in Type I Hypersensitivity Reactions in Murine Experimental Allergic Conjunctivitis.Therapeutic potential of CCR1 antagonists for multiple myeloma.Targeting CCL5 in inflammation.Minimal invasive surgical procedure of inducing myocardial infarction in mice.Controlled intramyocardial release of engineered chemokines by biodegradable hydrogels as a treatment approach of myocardial infarction.Induction of the CXC chemokine interferon-gamma-inducible protein 10 regulates the reparative response following myocardial infarction.Compartmentalized protective and detrimental effects of endogenous macrophage migration-inhibitory factor mediated by CXCR2 in a mouse model of myocardial ischemia/reperfusion.Blocking CCL5-CXCL4 heteromerization preserves heart function after myocardial infarction by attenuating leukocyte recruitment and NETosis.
P2860
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P2860
Ccr1 deficiency reduces inflammatory remodelling and preserves left ventricular function after myocardial infarction.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@en
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@nl
type
label
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@en
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@nl
prefLabel
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@en
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@nl
P2093
P2860
P1476
Ccr1 deficiency reduces inflam ...... n after myocardial infarction.
@en
P2093
A Zernecke
E Shagdarsuren
Y Djalali-Talab
P2860
P304
P356
10.1111/J.1582-4934.2007.00194.X
P577
2007-12-14T00:00:00Z