ChREBP, but not LXRs, is required for the induction of glucose-regulated genes in mouse liver. - Wikidata - wikimedia - TriplyDB

ChREBP, but not LXRs, is required for the induction of glucose-regulated genes in mouse liver.

ChREBP, but not LXRs, is required for the induction of glucose-regulated genes in mouse liver.

http://www.wikidata.org/entity/Q42128898

about

Regulation of glucose metabolism from a liver-centric perspective Transcriptional regulation of hepatic lipogenesis Energy metabolism in the liver CREB and ChREBP oppositely regulate SIRT1 expression in response to energy availability Using transcription modules to identify expression clusters perturbed in Williams-Beuren syndrome Impaired-inactivation of FoxO1 contributes to glucose-mediated increases in serum very low-density lipoprotein CFTR depletion results in changes in fatty acid composition and promotes lipogenesis in intestinal Caco 2/15 cells Liver X receptor β: maintenance of epidermal expression in intrinsic and extrinsic skin aging Fasting-induced FGF21 is repressed by LXR activation via recruitment of an HDAC3 corepressor complex in mice Meeting report: nuclear receptors: transcription factors and drug targets connecting basic research with translational medicine.Minireview: liver X receptor beta: emerging roles in physiology and diseases.High glucose-induced O-GlcNAcylated carbohydrate response element-binding protein (ChREBP) mediates mesangial cell lipogenesis and fibrosis: the possible role in the development of diabetic nephropathy E2F1 inhibits circulating cholesterol clearance by regulating Pcsk9 expression in the liver.A feed-forward loop amplifies nutritional regulation of PNPLA3 Genetic control of de novo lipogenesis: role in diet-induced obesity Minireview: Thioredoxin-interacting protein: regulation and function in the pancreatic β-cell.Thyroid hormone receptor beta (TRbeta) and liver X receptor (LXR) regulate carbohydrate-response element-binding protein (ChREBP) expression in a tissue-selective manner.Foxa1 and Foxa2 maintain the metabolic and secretory features of the mature beta-cell Differing endoplasmic reticulum stress response to excess lipogenesis versus lipid oversupply in relation to hepatic steatosis and insulin resistance.Alpha-lipoic acid attenuates insulin resistance and improves glucose metabolism in high fat diet-fed mice.Liver steatosis and increased ChREBP expression in mice carrying a liver specific SIRT1 null mutation under a normal feeding condition.ChREBP mediates glucose repression of peroxisome proliferator-activated receptor alpha expression in pancreatic beta-cells.Stearoyl-CoA desaturase and its relation to high-carbohydrate diets and obesity.Fatty acid synthase and liver triglyceride metabolism: housekeeper or messenger?O-GlcNAcylation increases ChREBP protein content and transcriptional activity in the liver Preventing β-cell loss and diabetes with calcium channel blockers The lipogenic transcription factor ChREBP dissociates hepatic steatosis from insulin resistance in mice and humans Molecular mechanisms of estrogen receptors' suppression of lipogenesis in pancreatic β-cells.Hepatic insulin signaling is required for obesity-dependent expression of SREBP-1c mRNA but not for feeding-dependent expression LRH-1-dependent glucose sensing determines intermediary metabolism in liver.E2F1 mediates sustained lipogenesis and contributes to hepatic steatosis.Structural characterization of a unique interface between carbohydrate response element-binding protein (ChREBP) and 14-3-3β protein.Cellular steatosis in ethanol oxidizing-HepG2 cells is partially controlled by the transcription factor, early growth response-1.A MED13-dependent skeletal muscle gene program controls systemic glucose homeostasis and hepatic metabolism.Identification of HNF-4α as a key transcription factor to promote ChREBP expression in response to glucose Neuroendocrine Regulation of Metabolism Dietary sucrose is essential to the development of liver injury in the methionine-choline-deficient model of steatohepatitis.Identification of adropin as a secreted factor linking dietary macronutrient intake with energy homeostasis and lipid metabolism.Transcriptional regulation of mitochondrial glycerophosphate acyltransferase is mediated by distal promoter via ChREBP and SREBP-1 Glucose and Insulin Stimulate Lipogenesis in Porcine Adipocytes: Dissimilar and Identical Regulation Pathway for Key Transcription Factors.

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P2860

ChREBP, but not LXRs, is required for the induction of glucose-regulated genes in mouse liver.

http://www.wikidata.org/entity/Q42128898

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

@en

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

@nl

type

label

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

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ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

@nl

prefLabel

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

@en

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

@nl

P1433

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P1433

Journal of Clinical Investigation

P1476

ChREBP, but not LXRs, is requi ...... egulated genes in mouse liver.

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P2093

Jean Girard

http://www.w3.org/2001/XMLSchema#string

Jean-Marc A Lobaccaro

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Lesley Millatt

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Pascale Bossard

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P2860

Role of LXRs in control of lipogenesis

Carbohydrate response element binding protein directly promotes lipogenic enzyme gene transcription

Activation of liver X receptor improves glucose tolerance through coordinate regulation of glucose metabolism in liver and adipose tissue

Direct and indirect mechanisms for regulation of fatty acid synthase gene expression by liver X receptors

The liver X receptor (LXR) and hepatic lipogenesis. The carbohydrate-response element-binding protein is a target gene of LXR

Distinct roles of insulin and liver X receptor in the induction and cleavage of sterol regulatory element-binding protein-1c

A glucose-responsive transcription factor that regulates carbohydrate metabolism in the liver

Sterol regulation of acetyl coenzyme A carboxylase promoter requires two interdependent binding sites for sterol regulatory element binding proteins

Sterol regulatory element binding protein-1c is a major mediator of insulin action on the hepatic expression of glucokinase and lipogenesis-related genes

Glucose-dependent transcriptional regulation by an evolutionarily conserved glucose-sensing module.

P304

956-964

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scholarly article

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10.1172/JCI34314

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P433

3

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P478

118

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Catherine Postic

Pierre-Damien Denechaud

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2008-03-01T00:00:00Z

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18292813

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2248330

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