Potential of N-glycan in cell adhesion and migration as either a positive or negative regulator.
about
Bisecting GlcNAc residues on laminin-332 down-regulate galectin-3-dependent keratinocyte motility.Recognition of tumor cells by Dectin-1 orchestrates innate immune cells for anti-tumor responses.Role of galectins in re-epithelialization of wounds.Hypoxia-induced changes to integrin α 3 glycosylation facilitate invasion in epidermoid carcinoma cell line A431All-trans-retinoic acid modulates ICAM-1 N-glycan composition by influencing GnT-III levels and inhibits cell adhesion and trans-endothelial migrationThe effect of malnutrition on protein glycosylation in childrenRoles of N-acetylglucosaminyltransferase III in epithelial-to-mesenchymal transition induced by transforming growth factor β1 (TGF-β1) in epithelial cell lines.Two opposing roles of O-glycans in tumor metastasis.The Na-K-ATPase α₁β₁ heterodimer as a cell adhesion molecule in epithelia.Effect of alpha 2,6 sialylation on integrin-mediated adhesion of breast cancer cells to fibronectin and collagen IV.Aberrant glycosylation of plasma proteins in severe preeclampsia promotes monocyte adhesion.Hypoxic regulation of glycosylation via the N-acetylglucosamine cycleThe bisecting GlcNAc in cell growth control and tumor progression.Potential roles of N-glycosylation in cell adhesion.Extracellular galectin-3 in tumor progression and metastasis.β1,6-branched complex-type N-glycans affect FAK signaling in metastatic melanoma cells.Wnt/beta-catenin signaling down-regulates N-acetylglucosaminyltransferase III expression: the implications of two mutually exclusive pathways for regulation.Interactions between the discoidin domain receptor 1 and β1 integrin regulate attachment to collagenMutational deglycosylation of the Fc portion of immunoglobulin G causes O-sulfation of tyrosine adjacently preceding the originally glycosylated site.The expression of annexin II and Lewis y antigen in ovarian epithelial tumors and the correlation between them.Inhibition of the Hexosamine Biosynthetic Pathway by targeting PGM3 causes breast cancer growth arrest and apoptosis.Immunosuppressive drugs affect high-mannose/hybrid N-glycans on human allostimulated leukocytes.Bisecting N-Acetylglucosamine Structures Inhibit Hypoxia-Induced Epithelial-Mesenchymal Transition in Breast Cancer Cells.
P2860
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P2860
Potential of N-glycan in cell adhesion and migration as either a positive or negative regulator.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Potential of N-glycan in cell ...... ositive or negative regulator.
@en
Potential of N-glycan in cell ...... ositive or negative regulator.
@nl
type
label
Potential of N-glycan in cell ...... ositive or negative regulator.
@en
Potential of N-glycan in cell ...... ositive or negative regulator.
@nl
prefLabel
Potential of N-glycan in cell ...... ositive or negative regulator.
@en
Potential of N-glycan in cell ...... ositive or negative regulator.
@nl
P2860
P356
P1476
Potential of N-glycan in cell ...... ositive or negative regulator.
@en
P2093
Jianguo Gu
Naoyuki Taniguchi
P2860
P304
P356
10.4161/CAM.2.4.6748
P577
2008-10-05T00:00:00Z