Partial mesenchymal to epithelial reverting transition in breast and prostate cancer metastases. - Wikidata - wikimedia - TriplyDB

Partial mesenchymal to epithelial reverting transition in breast and prostate cancer metastases.

Partial mesenchymal to epithelial reverting transition in breast and prostate cancer metastases.

http://www.wikidata.org/entity/Q42145874

about

Homeostatic Signaling by Cell-Cell Junctions and Its Dysregulation during Cancer Progression Role of EMT in Metastasis and Therapy Resistance Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis CXCR3 in carcinoma progression The Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and Treatment The role of Snail in prostate cancer Cellular Plasticity in Prostate Cancer Bone Metastasis TGF-β-induced epithelial-to-mesenchymal transition proceeds through stepwise activation of multiple feedback loops Coupled reversible and irreversible bistable switches underlying TGFβ-induced epithelial to mesenchymal transition Microenvironmental regulation of tumor progression and metastasis Caught in the act: revealing the metastatic process by live imaging.Hepatic nonparenchymal cells drive metastatic breast cancer outgrowth and partial epithelial to mesenchymal transition The mitogen-activated protein (MAP) kinases p38 and extracellular signal-regulated kinase (ERK) are involved in hepatocyte-mediated phenotypic switching in prostate cancer cells EMT and MET: necessary or permissive for metastasis?Heterogeneous Cadherin Expression and Multicellular Aggregate Dynamics in Ovarian Cancer Dissemination Combinatorial interventions inhibit TGFβ-driven epithelial-to-mesenchymal transition and support hybrid cellular phenotypes.Bioinformatic approaches to augment study of epithelial-to-mesenchymal transition in lung cancer The epigenetics of epithelial-mesenchymal plasticity in cancer.EMT and EGFR in CTCs cytokeratin negative non-metastatic breast cancer Metastasis as a therapeutic target in prostate cancer: a conceptual framework.Spontaneous dormancy of metastatic breast cancer cells in an all human liver microphysiologic system Epithelial-mesenchymal transition in prostate cancer is associated with quantifiable changes in nuclear structure.Statin-induced mevalonate pathway inhibition attenuates the growth of mesenchymal-like cancer cells that lack functional E-cadherin mediated cell cohesion Epithelial-mesenchymal transition markers in lymph node metastases and primary breast tumors - relation to dissemination and proliferation.The role of hepatocyte nuclear factor 4alpha in metastatic tumor formation of hepatocellular carcinoma and its close relationship with the mesenchymal-epithelial transition markers.Lung epithelial cells induce both phenotype alteration and senescence in breast cancer cells.Targeting connexin 43 with α-connexin carboxyl-terminal (ACT1) peptide enhances the activity of the targeted inhibitors, tamoxifen and lapatinib, in breast cancer: clinical implication for ACT1 OVOL guides the epithelial-hybrid-mesenchymal transition.A microphysiological system model of therapy for liver micrometastases The glycosyltransferase LARGE2 is repressed by Snail and ZEB1 in prostate cancer.Cx26 knockout predisposes the mammary gland to primary mammary tumors in a DMBA-induced mouse model of breast cancer Role of connexins in metastatic breast cancer and melanoma brain colonization Synergistic action of master transcription factors controls epithelial-to-mesenchymal transition Late recurrence of breast carcinoma metastasis to the hypopharynx: a case report The paradox of E-cadherin: role in response to hypoxia in the tumor microenvironment and regulation of energy metabolism.Distinct routes to metastasis: plasticity-dependent and plasticity-independent pathways Primary versus castration-resistant prostate cancer: modeling through novel murine prostate cancer cell lines.Profilin-1 downregulation has contrasting effects on early vs late steps of breast cancer metastasis Mesenchymal-epithelial transition (MET) as a mechanism for metastatic colonisation in breast cancer.The rejuvenated scenario of epithelial-mesenchymal transition (EMT) and cancer metastasis.

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P2860

Partial mesenchymal to epithelial reverting transition in breast and prostate cancer metastases.

http://www.wikidata.org/entity/Q42145874

description

2011 nî lūn-bûn

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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type

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Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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P1433

Cancer microenvironment : official journal of the International Cancer Microenvironment Society

P1476

Partial mesenchymal to epithel ...... nd prostate cancer metastases.

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P2093

Marie Acquafondata

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Qian Wu

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Rajiv Dhir

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Yvonne Chao

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P2860

Adhesion-independent mechanism for suppression of tumor cell invasion by E-cadherin.

E-cadherin expression in primary carcinomas of the breast and its distant metastases

Epithelial-mesenchymal transitions in tumour progression

Selectins and selectin ligands in extravasation of cancer cells and organ selectivity of metastasis

Collective cell migration in morphogenesis, regeneration and cancer

Visualizing extravasation dynamics of metastatic tumor cells

Vimentin induces changes in cell shape, motility, and adhesion during the epithelial to mesenchymal transition.

Generation of an immortalized astrocyte cell line from H-2Kb-tsA58 mice to study the role of astrocytes in brain metastasis.

Variable beta-catenin expression in colorectal cancers indicates tumor progression driven by the tumor environment.

Breast carcinoma cells re-express E-cadherin during mesenchymal to epithelial reverting transition.

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