Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain.
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Plasticity in the Neonatal Brain following Hypoxic-Ischaemic InjuryPathophysiology and neuroprotection of global and focal perinatal brain injury: lessons from animal modelsRegional and temporal profiles of calpain and caspase-3 activities in postnatal rat brain following repeated propofol administrationNew insights into mitochondrial structure during cell death.Neonatal Hypoxia Ischaemia: Mechanisms, Models, and Therapeutic Challenges.Necrostatin-1 attenuates mitochondrial dysfunction in neurons and astrocytes following neonatal hypoxia-ischemia.Resuscitation of newborn piglets. short-term influence of FiO2 on matrix metalloproteinases, caspase-3 and BDNF.Hypoxic-ischemic encephalopathy in the term infant.Neuroprotective therapies after perinatal hypoxic-ischemic brain injury.Proteases inhibition assessment on PC12 and NGF treated cells after oxygen and glucose deprivation reveals a distinct role for aspartyl proteases.Perinatal hypoxic-ischemic encephalopathy.Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.A translational murine model of sub-lethal intoxication with Shiga toxin 2 reveals novel ultrastructural findings in the brain striatum.Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.The mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivationDiffusion characteristics associated with neuronal injury and glial activation following hypoxia-ischemia in the immature brain.Rewarming from therapeutic hypothermia induces cortical neuron apoptosis in a swine model of neonatal hypoxic-ischemic encephalopathyThe transcription factor c-Myc suppresses MiR-23b and MiR-27b transcription during fetal distress and increases the sensitivity of neurons to hypoxia-induced apoptosisProgrammed Necrosis: A Prominent Mechanism of Cell Death following Neonatal Brain InjuryPathophysiology of perinatal asphyxia: can we predict and improve individual outcomes?Mitochondrial Optic Atrophy (OPA) 1 Processing Is Altered in Response to Neonatal Hypoxic-Ischemic Brain Injury.Automated core-penumbra quantification in neonatal ischemic brain injuryEndoplasmic reticulum pathology and stress response in neurons precede programmed necrosis after neonatal hypoxia-ischemia.White matter apoptosis is increased by delayed hypothermia and rewarming in a neonatal piglet model of hypoxic ischemic encephalopathy.Treatment advances in neonatal neuroprotection and neurointensive care.Molecular regulation of DNA damage-induced apoptosis in neurons of cerebral cortex.Environmental enrichment decreases asphyxia-induced neurobehavioral developmental delay in neonatal rats.Enhancement of autophagic flux after neonatal cerebral hypoxia-ischemia and its region-specific relationship to apoptotic mechanisms.The mitochondrial permeability transition pore: a molecular target for amyotrophic lateral sclerosis therapy.Effect of Trp53 gene deficiency on brain injury after neonatal hypoxia-ischemiaFeasibility of autologous cord blood cells for infants with hypoxic-ischemic encephalopathy.Neuronal cell death in neonatal hypoxia-ischemia.Mitochondria and perinatal brain injury.Autophagy in hypoxia-ischemia induced brain injury.Neuronal autophagy in cerebral ischemia.Targeting neonatal ischemic brain injury with a pentapeptide-based irreversible caspase inhibitor.Scutellarin as a Potential Therapeutic Agent for Microglia-Mediated Neuroinflammation in Cerebral Ischemia.Angiotensin-(1-7) protects from brain damage induced by shiga toxin 2-producing enterohemorrhagic Escherichia coli.Impaired autophagosome clearance contributes to neuronal death in a piglet model of neonatal hypoxic-ischemic encephalopathy.Cell Death in the Developing Brain after Hypoxia-Ischemia
P2860
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P2860
Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@en
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@nl
type
label
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@en
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@nl
prefLabel
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@en
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@nl
P2093
P2860
P1433
P1476
Failure to complete apoptosis ...... sfunction in rodent forebrain.
@en
P2093
D M Ferriero
D P O'Riordan
F J Northington
L J Martin
M E Zelaya
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2007.06.060
P407
P577
2007-08-02T00:00:00Z