Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
about
Many stimuli pull the necrotic trigger, an overviewFueling the flames: Mammalian programmed necrosis in inflammatory diseasesBiomarkers and molecular probes for cell death imaging and targeted therapeuticsThe mechanism of necroptosis in normal and cancer cellsRole of necroptosis in the pathogenesis of solid organ injuryNew advances in molecular mechanisms and emerging therapeutic targets in alcoholic liver diseasesProgrammed necrosis in the cross talk of cell death and inflammationRole of mitochondria in apoptotic and necroptotic cell death in the developing brain5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Inhibits RIPK1/RIPK3-Dependent Necroptosis in THP-1-Derived Foam Cells.Pathophysiology of glia in perinatal white matter injury.Necrostatin-1 attenuates mitochondrial dysfunction in neurons and astrocytes following neonatal hypoxia-ischemia.Mitochondrial dysfunction induces Sarm1-dependent cell death in sensory neuronsNecroptosisCell death signalling mechanisms in heart failure.Nicotinamide mononucleotide adenylyl transferase 1 protects against acute neurodegeneration in developing CNS by inhibiting excitotoxic-necrotic cell death.Upregulation of the receptor-interacting protein 3 expression and involvement in neural tissue damage after spinal cord injury in mice.NO donor induces Nec-1-inhibitable, but RIP1-independent, necrotic cell death in pancreatic β-cells.Past, present, and future of molecular and cellular oncologyProgrammed Necrosis: A Prominent Mechanism of Cell Death following Neonatal Brain InjuryReceptor Interacting Protein 3-Mediated Necroptosis Promotes Lipopolysaccharide-Induced Inflammation and Acute Respiratory Distress Syndrome in Mice.Dichotomy between RIP1- and RIP3-mediated necroptosis in tumor necrosis factor-α-induced shock.Pathophysiology of perinatal asphyxia: can we predict and improve individual outcomes?Sex differences in cell genesis, hippocampal volume and behavioral outcomes in a rat model of neonatal HIEndoplasmic reticulum pathology and stress response in neurons precede programmed necrosis after neonatal hypoxia-ischemia.Necroptosis, a novel type of programmed cell death, contributes to early neural cells damage after spinal cord injury in adult mice.Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease modelsAbsence of receptor interacting protein kinase 3 prevents ethanol-induced liver injuryPerinatal brain damage: The term infant.Two independent pathways of regulated necrosis mediate ischemia-reperfusion injuryCaspase blockade induces RIP3-mediated programmed necrosis in Toll-like receptor-activated microglia.Increased hepatic receptor interacting protein kinase 3 expression due to impaired proteasomal functions contributes to alcohol-induced steatosis and liver injury.Treatment advances in neonatal neuroprotection and neurointensive care.Structural basis of signal transduction in the TNF receptor superfamilyProtective effects of necrostatin-1 against concanavalin A-induced acute hepatic injury in miceVitamin C induces apoptosis in AGS cells via production of ROS of mitochondria.The rodent endovascular puncture model of subarachnoid hemorrhage: mechanisms of brain damage and therapeutic strategies.True grit: programmed necrosis in antiviral host defense, inflammation, and immunogenicity.Akt and mTOR mediate programmed necrosis in neurons.Sexual dimorphism in BDNF signaling after neonatal hypoxia-ischemia and treatment with necrostatin-1.Necrosis-dependent and independent signaling of the RIP kinases in inflammation.
P2860
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P2860
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@ast
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@en
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@nl
type
label
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@ast
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@en
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@nl
prefLabel
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@ast
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@en
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@nl
P2093
P2860
P356
P1476
Necrostatin decreases oxidative damage, inflammation, and injury after neonatal HI.
@en
P2093
Ernest M Graham
Estelle B Gauda
Frances J Northington
Lee J Martin
P2860
P304
P356
10.1038/JCBFM.2010.72
P577
2010-06-23T00:00:00Z