Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
about
G-protein betagamma-complex is crucial for efficient signal amplification in visionMembrane guanylyl cyclase complexes shape the photoresponses of retinal rods and cones.Dysregulation of Ca(v)1.4 channels disrupts the maturation of photoreceptor synaptic ribbons in congenital stationary night blindness type 2RGS9 knockout causes a short delay in light responses of ON-bipolar cells.Experimental protocols alter phototransduction: the implications for retinal processing at visual thresholdAbsence of synaptic regulation by phosducin in retinal slices.Anti-amyloid therapy protects against retinal pigmented epithelium damage and vision loss in a model of age-related macular degeneration.Regulation of age-related macular degeneration-like pathology by complement factor H.Phosphorylation of phosducin accelerates rod recovery from transducin translocation.A new mouse model for stationary night blindness with mutant Slc24a1 explains the pathophysiology of the associated human disease.How rods respond to single photons: Key adaptations of a G-protein cascade that enable vision at the physical limit of perception.Structural modulation of phosducin by phosphorylation and 14-3-3 protein binding.The relationship between slow photoresponse recovery rate and temporal resolution of vision.Functional comparison of rod and cone Gα(t) on the regulation of light sensitivity.Gene expression changes in the retina following subretinal injection of human neural progenitor cells into a rodent model for retinal degeneration.Transducin translocation contributes to rod survival and enhances synaptic transmission from rods to rod bipolar cellsThe physiological roles of phosducin: from retinal function to stress-dependent hypertension.Rod vision is controlled by dopamine-dependent sensitization of rod bipolar cells by GABA.AAV-Nrf2 Promotes Protection and Recovery in Animal Models of Oxidative Stress.Structural Characterization of Phosducin and Its Complex with the 14-3-3 Protein.Proteomic profiling of a layered tissue reveals unique glycolytic specializations of photoreceptor cells.Expression of human complement factor H prevents age-related macular degeneration-like retina damage and kidney abnormalities in aged Cfh knockout mice.A Presynaptic Group III mGluR Recruits Gβγ/SNARE Interactions to Inhibit Synaptic Transmission by Cone Photoreceptors in the Vertebrate Retina.Structural Basis for the 14-3-3 Protein-Dependent Inhibition of Phosducin Function.Increased proteasomal activity supports photoreceptor survival in inherited retinal degeneration.
P2860
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P2860
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@en
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@nl
type
label
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@en
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@nl
prefLabel
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@en
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse.
@nl
P2093
P2860
P1476
Phosducin regulates transmission at the photoreceptor-to-ON-bipolar cell synapse
@en
P2093
Christopher Kessler
Laura J Frishman
Marie E Burns
Rolf Herrmann
Timothy Hammond
Vadim Y Arshavsky
P2860
P304
P356
10.1523/JNEUROSCI.4775-09.2010
P407
P577
2010-03-01T00:00:00Z