Role of STAT3 in Transformation and Drug Resistance in CML - Wikidata - wikimedia - TriplyDB

Role of STAT3 in Transformation and Drug Resistance in CML

Role of STAT3 in Transformation and Drug Resistance in CML

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Combined STAT3 and BCR-ABL1 inhibition induces synthetic lethality in therapy-resistant chronic myeloid leukemia Breast cancer cells condition lymphatic endothelial cells within pre-metastatic niches to promote metastasis A derivative of epigallocatechin-3-gallate induces apoptosis via SHP-1-mediated suppression of BCR-ABL and STAT3 signalling in chronic myelogenous leukaemia Overcoming Chemoresistance of Pediatric Ependymoma by Inhibition of STAT3 Signaling.Overexpression of RPS27a contributes to enhanced chemoresistance of CML cells to imatinib by the transactivated STAT3.Ginkgolic Acid C 17:1, Derived from Ginkgo biloba Leaves, Suppresses Constitutive and Inducible STAT3 Activation through Induction of PTEN and SHP-1 Tyrosine Phosphatase.Synthesis and Cytotoxicity against K562 Cells of 3-O-Angeloyl-20-O-acetyl Ingenol, a Derivative of Ingenol Mebutate Ginkgetin Blocks Constitutive STAT3 Activation and Induces Apoptosis through Induction of SHP-1 and PTEN Tyrosine Phosphatases.Myxoma Virus Optimizes Cisplatin for the Treatment of Ovarian Cancer In Vitro and in a Syngeneic Murine Dissemination Model.SZRD1 is a Novel Protein that Functions as a Potential Tumor Suppressor in Cervical Cancer.Combined targeting of STAT3 and STAT5: a novel approach to overcome drug resistance in chronic myeloid leukemia.Toosendanin demonstrates promising antitumor efficacy in osteosarcoma by targeting STAT3.IL-6 secreted by cancer-associated fibroblasts induces tamoxifen resistance in luminal breast cancer.Pro-survival role of protein kinase C epsilon in Philadelphia chromosome positive acute leukemia.The Role of Signal Transducer and Activator of Transcription 3 (STAT3) and Its Targeted Inhibition in Hematological Malignancies

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Role of STAT3 in Transformation and Drug Resistance in CML

http://www.wikidata.org/entity/Q42174918

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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Role of STAT3 in Transformation and Drug Resistance in CML

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Role of STAT3 in Transformation and Drug Resistance in CML

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Role of STAT3 in Transformation and Drug Resistance in CML

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Role of STAT3 in Transformation and Drug Resistance in CML

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Role of STAT3 in Transformation and Drug Resistance in CML

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Role of STAT3 in Transformation and Drug Resistance in CML

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FONC.2012.00030

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Frontiers in Oncology

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Role of STAT3 in Transformation and Drug Resistance in CML

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Joel H Tolentino

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Lori A Hazlehurst

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Rajesh R Nair

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The coiled-coil domain of Stat3 is essential for its SH2 domain-mediated receptor binding and subsequent activation induced by epidermal growth factor and interleukin-6

Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway

Principles of interleukin (IL)-6-type cytokine signalling and its regulation

Interleukin-6-type cytokine signalling through the gp130/Jak/STAT pathway

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

Self-renewal of pluripotent embryonic stem cells is mediated via activation of STAT3

Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality

Polymorphisms in signal transducer and activator of transcription 3 and lung function in asthma

Interferon activation of the transcription factor Stat91 involves dimerization through SH2-phosphotyrosyl peptide interactions

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30

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scholarly article

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10.3389/FONC.2012.00030

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2

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2012-04-10T00:00:00Z

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