α2δ expression sets presynaptic calcium channel abundance and release probability - Wikidata - wikimedia - TriplyDB

α2δ expression sets presynaptic calcium channel abundance and release probability

α2δ expression sets presynaptic calcium channel abundance and release probability

http://www.wikidata.org/entity/Q42202772

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Mechanisms of the gabapentinoids and α 2 δ-1 calcium channel subunit in neuropathic pain Deconstructing brain-derived neurotrophic factor actions in adult brain circuits to bridge an existing informational gap in neuro-cell biology Molecular Machines Regulating the Release Probability of Synaptic Vesicles at the Active Zone The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic Potential Genetic disruption of voltage-gated calcium channels in psychiatric and neurological disorders Structure-function of proteins interacting with the α1 pore-forming subunit of high-voltage-activated calcium channels Current Status of the New Antiepileptic Drugs in Chronic Pain Mechanisms of Action of Antiseizure Drugs and the Ketogenic Diet Fragile X mental retardation protein controls synaptic vesicle exocytosis by modulating N-type calcium channel density Central Mechanisms Mediating Thrombospondin-4-induced Pain States.Hair cells use active zones with different voltage dependence of Ca2+ influx to decompose sounds into complementary neural codes.Emerging evidence for specific neuronal functions of auxiliary calcium channel α₂δ subunits.Differences in human cortical gene expression match the temporal properties of large-scale functional networks.Neurexins.Rab Interacting Molecules 2 and 3 Directly Interact with the Pore-Forming CaV1.3 Ca2+ Channel Subunit and Promote Its Membrane Expression.Functional exofacially tagged N-type calcium channels elucidate the interaction with auxiliary α2δ-1 subunits.Activation of MrgC receptor inhibits N-type calcium channels in small-diameter primary sensory neurons in mice.Differential triggering of spontaneous glutamate release by P/Q-, N- and R-type Ca2+ channels.Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation.Independent regulation of basal neurotransmitter release efficacy by variable Ca²+ influx and bouton size at small central synapses.A novel null homozygous mutation confirms CACNA2D2 as a gene mutated in epileptic encephalopathy.Characterization of Cav1.4 complexes (α11.4, β2, and α2δ4) in HEK293T cells and in the retina The upregulation of α2δ-1 subunit modulates activity-dependent Ca2+ signals in sensory neurons Expression and cellular localization of the voltage-gated calcium channel α2δ3 in the rodent retina.Survey of Red Fluorescence Proteins as Markers for Secretory Granule Exocytosis The inhibition of high-voltage-activated calcium current by activation of MrgC11 involves phospholipase C-dependent mechanisms.The active-zone protein Munc13 controls the use-dependence of presynaptic voltage-gated calcium channels Ubiquitin-specific protease USP2-45 acts as a molecular switch to promote α2δ-1-induced downregulation of Cav1.2 channels.BONLAC: A combinatorial proteomic technique to measure stimulus-induced translational profiles in brain slices Isoflurane inhibits synaptic vesicle exocytosis through reduced Ca2+ influx, not Ca2+-exocytosis coupling.Finding Channels.Astrocyte-derived thrombospondins mediate the development of hippocampal presynaptic plasticity in vitro Intrinsic variability in Pv, RRP size, Ca(2+) channel repertoire, and presynaptic potentiation in individual synaptic boutons.Calcium sensor regulation of the CaV2.1 Ca2+ channel contributes to short-term synaptic plasticity in hippocampal neurons Calcium channels and short-term synaptic plasticity.Effect of knockout of α2δ-1 on action potentials in mouse sensory neurons Synapse-Level Determination of Action Potential Duration by K(+) Channel Clustering in Axons Nanoscale-targeted patch-clamp recordings of functional presynaptic ion channels.α2δ-1 gene deletion affects somatosensory neuron function and delays mechanical hypersensitivity in response to peripheral nerve damage.Position of UNC-13 in the active zone regulates synaptic vesicle release probability and release kinetics.

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P2860

α2δ expression sets presynaptic calcium channel abundance and release probability

http://www.wikidata.org/entity/Q42202772

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

α2δ expression sets presynaptic calcium channel abundance and release probability

@en

α2δ expression sets presynaptic calcium channel abundance and release probability

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type

label

α2δ expression sets presynaptic calcium channel abundance and release probability

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α2δ expression sets presynaptic calcium channel abundance and release probability

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prefLabel

α2δ expression sets presynaptic calcium channel abundance and release probability

@en

α2δ expression sets presynaptic calcium channel abundance and release probability

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α2δ expression sets presynaptic calcium channel abundance and release probability

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Michael B Hoppa

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Wojciech Margas

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P2860

Distribution and evolution of von Willebrand/integrin A domains: widely dispersed domains with roles in cell adhesion and elsewhere

Crystal structure of the von Willebrand factor A domain of human capillary morphogenesis protein 2: an anthrax toxin receptor

A genome-wide Drosophila screen for heat nociception identifies α2δ3 as an evolutionarily conserved pain gene.

Functional properties of a new voltage-dependent calcium channel alpha(2)delta auxiliary subunit gene (CACNA2D2)

Structure and regulation of voltage-gated Ca2+ channels

Visualizing secretion and synaptic transmission with pH-sensitive green fluorescent proteins

Improving the photostability of bright monomeric orange and red fluorescent proteins

Imaging neural activity in worms, flies and mice with improved GCaMP calcium indicators

The alpha2delta subunits of voltage-gated calcium channels form GPI-anchored proteins, a posttranslational modification essential for function

The metal-ion-dependent adhesion site in the Von Willebrand factor-A domain of alpha2delta subunits is key to trafficking voltage-gated Ca2+ channels.

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