Exercise ameliorates chronic kidney disease-induced defects in muscle protein metabolism and progenitor cell function. - Wikidata - wikimedia - TriplyDB

Exercise ameliorates chronic kidney disease-induced defects in muscle protein metabolism and progenitor cell function.

Exercise ameliorates chronic kidney disease-induced defects in muscle protein metabolism and progenitor cell function.

http://www.wikidata.org/entity/Q42206915

about

Satellite cell dysfunction and impaired IGF-1 signaling cause CKD-induced muscle atrophy PTEN inhibition improves muscle regeneration in mice fed a high-fat diet.Caspase-3 cleaves specific 19 S proteasome subunits in skeletal muscle stimulating proteasome activity.Impact of chronic kidney disease and anemia on physical function in patients with chronic heart failure.Akt1-mediated fast/glycolytic skeletal muscle growth attenuates renal damage in experimental kidney disease.Skeletal muscle wasting in cachexia and sarcopenia: molecular pathophysiology and impact of exercise training.Mechanisms of muscle wasting in chronic kidney disease Low-frequency electrical stimulation attenuates muscle atrophy in CKD--a potential treatment strategy Decreased miR-29 suppresses myogenesis in CKD.Acupuncture plus Low-Frequency Electrical Stimulation (Acu-LFES) Attenuates Diabetic Myopathy by Enhancing Muscle Regeneration.Protein abundance of urea transporters and aquaporin 2 change differently in nephrotic pair-fed vs. non-pair-fed rats.Transcription factor FoxO1, the dominant mediator of muscle wasting in chronic kidney disease, is inhibited by microRNA-486.IL-6 regulation on skeletal muscle mitochondrial remodeling during cancer cachexia in the ApcMin/+ mouse.Muscle mTORC1 suppression by IL-6 during cancer cachexia: a role for AMPK.Systemic Regulators of Skeletal Muscle Regeneration in Obesity.MicroRNA in myogenesis and muscle atrophy Protein Nutrition and Malnutrition in CKD and ESRD.Muscle wasting from kidney failure-a model for catabolic conditions.MicroRNA in skeletal muscle development, growth, atrophy, and disease.Acupuncture plus low-frequency electrical stimulation (Acu-LFES) attenuates denervation-induced muscle atrophy.Chronic kidney disease induces autophagy leading to dysfunction of mitochondria in skeletal muscle.The Effect of Resistance Exercise on Inflammatory and Myogenic Markers in Patients with Chronic Kidney Disease.Chronic kidney disease and sports participation by children and adolescents.MicroRNA-23a and MicroRNA-27a Mimic Exercise by Ameliorating CKD-Induced Muscle Atrophy.Can muscle-kidney crosstalk slow progression of CKD?Insulin resistance and muscle metabolism in chronic kidney disease.Combined walking exercise and alkali therapy in patients with CKD4-5 regulates intramuscular free amino acid pools and ubiquitin E3 ligase expression.Leucine-stimulated mTOR signaling is partly attenuated in skeletal muscle of chronically uremic rats.Aerobic exercise training rescues protein quality control disruption on white skeletal muscle induced by chronic kidney disease in rats.Effect of endurance training and branched-chain amino acids on the signaling for muscle protein synthesis in CKD model rats fed a low-protein diet.Supplementation of ketoacids contributes to the up-regulation of the Wnt7a/Akt/p70S6K pathway and the down-regulation of apoptotic and ubiquitin-proteasome systems in the muscle of 5/6 nephrectomised rats.Current Methods for Skeletal Muscle Tissue Repair and Regeneration.WNK1 regulates skeletal muscle cell hypertrophy by modulating the nuclear localization and transcriptional activity of FOXO4.Commentaries on Viewpoint: What is the relationship between acute measure of muscle protein synthesis and changes in muscle mass?Effects of treadmill running in a rat model of chronic kidney disease

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Exercise ameliorates chronic kidney disease-induced defects in muscle protein metabolism and progenitor cell function.

http://www.wikidata.org/entity/Q42206915

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

@zh-tw

2009年论文

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2009年论文

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2009年论文

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name

Exercise ameliorates chronic k ...... and progenitor cell function.

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Exercise ameliorates chronic k ...... and progenitor cell function.

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type

label

Exercise ameliorates chronic k ...... and progenitor cell function.

@en

Exercise ameliorates chronic k ...... and progenitor cell function.

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prefLabel

Exercise ameliorates chronic k ...... and progenitor cell function.

@en

Exercise ameliorates chronic k ...... and progenitor cell function.

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P1433

Kidney International

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Exercise ameliorates chronic k ...... and progenitor cell function.

@en

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James L Bailey

http://www.w3.org/2001/XMLSchema#string

Janet D Klein

http://www.w3.org/2001/XMLSchema#string

Jie Du

http://www.w3.org/2001/XMLSchema#string

William E Mitch

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Xiaonan H Wang

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P2860

Phosphatidic acid-mediated mitogenic activation of mTOR signaling

Protein degradation by the ubiquitin-proteasome pathway in normal and disease states

Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

Changes in muscle mass and phenotype and the expression of autocrine and systemic growth factors by muscle in response to stretch and overload.

Mechanisms for defects in muscle protein metabolism in rats with chronic uremia. Influence of metabolic acidosis

Satellite cell depletion in degenerative skeletal muscle.

Mechanotransduction and the regulation of protein synthesis in skeletal muscle.

IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.

Myogenic signaling of phosphatidylinositol 3-kinase requires the serine-threonine kinase Akt/protein kinase B.

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751-759

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10.1038/KI.2009.260

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7

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19641484

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3835682

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