Predisposition for disrepair in the aged lung. - Wikidata - wikimedia - TriplyDB

Predisposition for disrepair in the aged lung.

Predisposition for disrepair in the aged lung.

http://www.wikidata.org/entity/Q42234172

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The effect of environmental chemicals on the tumor microenvironment Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis Nicotine stimulates collagen type I expression in lung via α7 nicotinic acetylcholine receptors.Aging mesenchymal stem cells fail to protect because of impaired migration and antiinflammatory response.Pathogenesis of idiopathic pulmonary fibrosis TGFβ1 mediates alcohol-induced Nrf2 suppression in lung fibroblasts.Type V collagen induced tolerance suppresses collagen deposition, TGF-β and associated transcripts in pulmonary fibrosis Blue journal conference. Aging and susceptibility to lung disease.Plasminogen activator inhibitor 1, fibroblast apoptosis resistance, and aging-related susceptibility to lung fibrosis Decreased Laminin Expression by Human Lung Epithelial Cells and Fibroblasts Cultured in Acellular Lung Scaffolds from Aged Mice.Reversal of persistent fibrosis in aging by targeting Nox4-Nrf2 redox imbalance Conformational coupling of integrin and Thy-1 regulates Fyn priming and fibroblast mechanotransduction Alteration of Aging-Dependent MicroRNAs in Idiopathic Pulmonary Fibrosis.TGF-β1 epigenetically modifies Thy-1 expression in primary lung fibroblasts Senescent Cells Contribute to the Physiological Remodeling of Aged Lungs Mechanisms of Lung Fibrosis Resolution.Detecting splicing variants in idiopathic pulmonary fibrosis from non-differentially expressed genes.Therapeutic benefits of young, but not old, adipose-derived mesenchymal stem cells in a chronic mouse model of bleomycin-induced pulmonary fibrosis.Differential Regulation of the Extracellular Cysteine/Cystine Redox State (EhCySS) by Lung Fibroblasts from Young and Old Mice.Animal models of fibrotic lung disease.Aging promotes pro-fibrotic matrix production and increases fibrocyte recruitment during acute lung injury.Experimental basis and new insights for cell therapy in Chronic Obstructive Pulmonary Disease.Influence of age on wound healing and fibrosis.The potential of cell-based therapy in lung diseases.Translational models of lung disease.Senolytic drugs target alveolar epithelial cell function and attenuate experimental lung fibrosis ex vivo.Aging and Lung Disease. Clinical Impact and Cellular and Molecular Pathways.An Official American Thoracic Society Workshop Report: Use of Animal Models for the Preclinical Assessment of Potential Therapies for Pulmonary Fibrosis.Lung carcinogenesis and fibrosis taken together: just coincidence?Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis.Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis."Scar-cinoma": viewing the fibrotic lung mesenchymal cell in the context of cancer biology.What Should Be Chronic: The Animal, the Model, or Both?Deregulation of selective autophagy during aging and pulmonary fibrosis: the role of TGFβ1.Mechanisms and Consequences of Oxidative Stress in Lung Disease: Therapeutic Implications for an Aging Populace.MiR-21-Mediated Suppression of Smad7 Induces TGFβ1 and Can Be Inhibited by Activation of Nrf2 in Alcohol-Treated Lung Fibroblasts.Age-related lung tissue remodeling due to the local distribution of MMP-2, TIMP-2, TGF-β and Hsp70.Lung Fibroblasts, Aging, and Idiopathic Pulmonary Fibrosis.Club cell protein 16 (Cc16) deficiency increases inflamm-aging in the lungs of mice

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P2860

Predisposition for disrepair in the aged lung.

http://www.wikidata.org/entity/Q42234172

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Predisposition for disrepair in the aged lung.

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Predisposition for disrepair in the aged lung.

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type

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Predisposition for disrepair in the aged lung.

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Predisposition for disrepair in the aged lung.

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Predisposition for disrepair in the aged lung.

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Predisposition for disrepair in the aged lung.

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Predisposition for disrepair in the aged lung.

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David C Neujahr

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David Guidot

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Jeffrey D Ritzenthaler

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Jesse Roman

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Mauricio Rojas

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S Todd Mills

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Susanne Roser-Page

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Viranuj Sueblinvong

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P2860

TGF beta signals through a heteromeric protein kinase receptor complex

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

Matrix elasticity directs stem cell lineage specification

Making sense of latent TGFbeta activation

An official ATS/ERS/JRS/ALAT statement: idiopathic pulmonary fibrosis: evidence-based guidelines for diagnosis and management

Pulmonary fibrosis: pathogenesis, etiology and regulation

[Effect of aging on pulmonary ICAM-1 and MCP-1 expressions in rats with lipopolysaccharide- induced acute lung injury]

Regulated splicing of the fibronectin EDA exon is essential for proper skin wound healing and normal lifespan

Cell-extracellular matrix interactions regulate neural differentiation of human embryonic stem cells.

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