Identifying the Role of GluN2A in Cerebral Ischemia.

about

Identifying the Role of GluN2A in Cerebral Ischemia.

Item

http://www.wikidata.org/entity/Q42328481

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Identifying the Role of GluN2A in Cerebral Ischemia.

@en

Identifying the Role of GluN2A in Cerebral Ischemia.

@nl

type

Item

label

Identifying the Role of GluN2A in Cerebral Ischemia.

@en

Identifying the Role of GluN2A in Cerebral Ischemia.

@nl

prefLabel

Identifying the Role of GluN2A in Cerebral Ischemia.

@en

Identifying the Role of GluN2A in Cerebral Ischemia.

@nl

P1476

Identifying the Role of GluN2A in Cerebral Ischemia.

@en

P2093

Long Wang

http://www.w3.org/2001/XMLSchema#string

Yongjun Sun

http://www.w3.org/2001/XMLSchema#string

Zibin Gao

http://www.w3.org/2001/XMLSchema#string

P2860

Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders.

NMDA di-heteromeric receptor populations and associated proteins in rat hippocampus

An increase in the association of GluN2B containing NMDA receptors with membrane scaffolding proteins was related to memory declines during aging

Pharmacology of triheteromeric N-Methyl-D-Aspartate Receptors.

Excitotoxicity and stroke: identifying novel targets for neuroprotection.

Extrasynaptic NMDA Receptor in Excitotoxicity: Function Revisited.

Distinct functional and pharmacological properties of Triheteromeric GluN1/GluN2A/GluN2B NMDA receptors.

The Functional and Molecular Properties, Physiological Functions, and Pathophysiological Roles of GluN2A in the Central Nervous System.

Controlling NMDA receptor subunit composition using ectopic retention signals.

Triheteromeric NR1/NR2A/NR2B receptors constitute the major N-methyl-D-aspartate receptor population in adult hippocampal synapses.

P304

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3389/FNMOL.2017.00012

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-01-24T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28174519

http://www.w3.org/2001/XMLSchema#string

P921

cerebral infarction

P932

5258732

http://www.w3.org/2001/XMLSchema#string