Nat1 Deficiency Is Associated with Mitochondrial Dysfunction and Exercise Intolerance in Mice.
about
Validation of optimal reference genes for quantitative real time PCR in muscle and adipose tissue for obesity and diabetes researchMechanism by which arylamine N-acetyltransferase 1 ablation causes insulin resistance in mice.Physiological Mitochondrial Fragmentation Is a Normal Cardiac Adaptation to Increased Energy Demand.Biallelic Mutations in ATP5F1D, which Encodes a Subunit of ATP Synthase, Cause a Metabolic Disorder.Reduced expression of Twist 1 is protective against insulin resistance of adipocytes and involves mitochondrial dysfunction
P2860
Nat1 Deficiency Is Associated with Mitochondrial Dysfunction and Exercise Intolerance in Mice.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@en
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@nl
type
label
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@en
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@nl
prefLabel
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@en
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@nl
P2093
P2860
P1433
P1476
Nat1 Deficiency Is Associated ...... Exercise Intolerance in Mice.
@en
P2093
Alan D Attie
Andrew J Whittle
Daniel Bernstein
Giovanni Fajardo
Indumathi Chennamsetty
Ivan Carcamo-Orive
John Sandin
Joshua W Knowles
Kévin Contrepois
Mark P Keller
P2860
P304
P356
10.1016/J.CELREP.2016.09.005
P577
2016-10-01T00:00:00Z