about
Phosphofructo-1-kinase deficiency leads to a severe cardiac and hematological disorder in addition to skeletal muscle glycogenosisTransgenic mice overexpressing phosphoenolpyruvate carboxykinase develop non-insulin-dependent diabetes mellitus.Enforced expression of protein kinase C in skeletal muscle causes physical inactivity, fatty liver and insulin resistance in the brain.Progressive neurologic and somatic disease in a novel mouse model of human mucopolysaccharidosis type IIIC.Correction of diabetic alterations by glucokinase.Disease correction by AAV-mediated gene therapy in a new mouse model of mucopolysaccharidosis type IIID.Transgenic mice overexpressing insulin-like growth factor-II in beta cells develop type 2 diabetes.Transgenic mice overexpressing human G972R IRS-1 show impaired insulin action and insulin secretion.Adipose tissue overexpression of vascular endothelial growth factor protects against diet-induced obesity and insulin resistance.Vascular endothelial growth factor-mediated islet hypervascularization and inflammation contribute to progressive reduction of β-cell mass.Increased fatty acid re-esterification by PEPCK overexpression in adipose tissue leads to obesity without insulin resistance.Counteraction of type 1 diabetic alterations by engineering skeletal muscle to produce insulin: insights from transgenic mice.HMGA1 overexpression in adipose tissue impairs adipogenesis and prevents diet-induced obesity and insulin resistance.Chronically increased glucose uptake by adipose tissue leads to lactate production and improved insulin sensitivity rather than obesity in the mouse.The suppressor of cytokine signalling 2 (SOCS2) is a key repressor of insulin secretion.Regulated expression of human insulin in the liver of transgenic mice corrects diabetic alterations.Protein kinase Cδ regulates nuclear export of FOXO1 through phosphorylation of the chaperone 14-3-3ζ.Overexpression of ubiquitous 6-phosphofructo-2-kinase in the liver of transgenic mice results in weight gain.Assessment of a dual regulatory role for NO in liver regeneration after partial hepatectomy: protection against apoptosis and retardation of hepatocyte proliferation.Evidence from Transgenic Mice That Interferon-β May Be Involved in the Onset of Diabetes MellitusOverexpression of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase in transgenic mice causes hepatic hyperketogenesisExpression of Glucokinase in Skeletal Muscle: A New Approach to Counteract Diabetic HyperglycemiaTRANSGENIC MICE IN THE ANALYSIS OF METABOLIC REGULATIONExpression of GLUT-2 antisense RNA in beta cells of transgenic mice leads to diabetesEvidence from transgenic mice that myc regulates hepatic glycolysis
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Anna Pujol
@ast
Anna Pujol
@en
Anna Pujol
@es
Anna Pujol
@nl
type
label
Anna Pujol
@ast
Anna Pujol
@en
Anna Pujol
@es
Anna Pujol
@nl
prefLabel
Anna Pujol
@ast
Anna Pujol
@en
Anna Pujol
@es
Anna Pujol
@nl
P106
P21
P31
P496
0000-0002-3484-598X