RhoA expression is controlled by nitric oxide through cGMP-dependent protein kinase activation. - Wikidata - wikimedia - TriplyDB

RhoA expression is controlled by nitric oxide through cGMP-dependent protein kinase activation.

RhoA expression is controlled by nitric oxide through cGMP-dependent protein kinase activation.

http://www.wikidata.org/entity/Q42435598

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The 'invisible hand': regulation of RHO GTPases by RHOGDIs Cellular mediators of renal vascular dysfunction in hypertension Expression and localization of the multidrug resistance protein 5 (MRP5/ABCC5), a cellular export pump for cyclic nucleotides, in human heart Regulation of intraocular pressure by soluble and membrane guanylate cyclases and their role in glaucoma Nitric oxide promotes epidermal stem cell migration via cGMP-Rho GTPase signalling.PDE5 inhibitor promotes melanin synthesis through the PKG pathway in B16 melanoma cells Alcohol decreases RhoA activity through a nitric oxide (NO)/cyclic GMP(cGMP)/protein kinase G (PKG)-dependent pathway in the airway epithelium Inhibition of RhoA/Rho kinase pathway is involved in the beneficial effect of sildenafil on pulmonary hypertension Proteolytic processing of cGMP-dependent protein kinase I mediates nuclear cGMP signaling in vascular smooth muscle cells Increased hepatic UCP2 expression in rats with nonalcoholic steatohepatitis is associated with upregulation of Sp1 binding to its motif within the proximal promoter region.Fucoxanthin provides neuroprotection in models of traumatic brain injury via the Nrf2-ARE and Nrf2-autophagy pathways.Coronary spasm that caused non-ST elevation myocardial infarction appeared in cath lab due to vasovagal reaction Regulation of retinoschisin secretion in Weri-Rb1 cells by the F-actin and microtubule cytoskeleton.GREBP, a cGMP-response element-binding protein repressing the transcription of natriuretic peptide receptor 1 (NPR1/GCA).PKG-1α leucine zipper domain defect increases pulmonary vascular tone: implications in hypoxic pulmonary hypertension.Current advances in the understanding of coronary vasospasm.Omega-3 fatty acid oxidation products prevent vascular endothelial cell activation by coplanar polychlorinated biphenyls.HIV-1 Tat activates a RhoA signaling pathway to reduce NMDA-evoked calcium responses in hippocampal neurons via an actin-dependent mechanism Reduced NO signaling during pregnancy attenuates outward uterine artery remodeling by altering MMP expression and collagen and elastin deposition S-nitrosylation in the regulation of gene transcription Pleiotropic effects of statins in distal human pulmonary artery smooth muscle cells Increased leukocyte Rho-associated coiled-coil containing protein kinase activity predicts the presence and severity of coronary vasospastic angina.Endothelial nitric oxide synthase keeps erection regulatory function balance in the penis.Comparative Proteomics Analysis Reveals L-Arginine Activates Ethanol Degradation Pathways in HepG2 Cells.Rho kinase as potential therapeutic target for cardiovascular diseases: opportunities and challenges.The proximal STAT6 and NF-kappaB sites are responsible for IL-13- and TNF-alpha-induced RhoA transcriptions in human bronchial smooth muscle cells.Understanding and targeting the Rho kinase pathway in erectile dysfunction.Direct binding and regulation of RhoA protein by cyclic GMP-dependent protein kinase Iα Transcriptional and post-transcriptional regulation of cGMP-dependent protein kinase (PKG-I): pathophysiological significance High blood pressure arising from a defect in vascular function.H2S-donating sildenafil (ACS6) inhibits superoxide formation and gp91phox expression in arterial endothelial cells: role of protein kinases A and G Proprotein convertases play an important role in regulating PKGI endoproteolytic cleavage and nuclear transport.Differential regulation of RhoA-mediated signaling by the TPalpha and TPbeta isoforms of the human thromboxane A2 receptor: independent modulation of TPalpha signaling by prostacyclin and nitric oxide.PKC ε Phosphorylates and Mediates the Cell Membrane Localization of RhoA.Inhibition of TRPC1/TRPC3 by PKG contributes to NO-mediated vasorelaxation.Soluble guanylate cyclase generation of cGMP regulates migration of MGE neurons.Regulation of smooth muscle contraction by small GTPases.NO orchestrates the loss of synaptic boutons from adult "sick" motoneurons: modeling a molecular mechanism.Rho GTPases in the regulation of pulmonary vascular barrier function.Mechanistic approach to the pathophysiology of target organ damage in hypertension from studies in a human model with characteristics opposite to hypertension: Bartter's and Gitelman's syndromes.

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P2860

RhoA expression is controlled by nitric oxide through cGMP-dependent protein kinase activation.

http://www.wikidata.org/entity/Q42435598

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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name

RhoA expression is controlled ...... ent protein kinase activation.

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RhoA expression is controlled ...... ent protein kinase activation.

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type

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RhoA expression is controlled ...... ent protein kinase activation.

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RhoA expression is controlled ...... ent protein kinase activation.

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RhoA expression is controlled ...... ent protein kinase activation.

@en

RhoA expression is controlled ...... ent protein kinase activation.

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P1433

Journal of Biological Chemistry

P1476

RhoA expression is controlled ...... ent protein kinase activation.

@en

P2093

Céline Marionneau

http://www.w3.org/2001/XMLSchema#string

Gervaise Loirand

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Malvyne Rolli-Derkinderen

http://www.w3.org/2001/XMLSchema#string

Pierre Pacaud

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Vincent Sauzeau

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P2860

Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

Ubiquitin-dependent protein degradation

Cyclic GMP-dependent protein kinase signaling pathway inhibits RhoA-induced Ca2+ sensitization of contraction in vascular smooth muscle

p11 expression in human bronchial epithelial cells is increased by nitric oxide in a cGMP-dependent pathway involving protein kinase G activation

NO activation of fos promoter elements requires nuclear translocation of G-kinase I and CREB phosphorylation but is independent of MAP kinase activation

Upregulation of small GTPase RhoA in the basilar artery from diabetic (mellitus) rats

Stress-induced stimulation of early growth response gene-1 by p38/stress-activated protein kinase 2 is mediated by a cAMP-responsive promoter element in a MAPKAP kinase 2-independent manner

Rho GTPases and signaling networks

Signal transduction by G-proteins, rho-kinase and protein phosphatase to smooth muscle and non-muscle myosin II.

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9472-9480

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scholarly article

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10.1074/JBC.M212776200

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11

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278

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2003-01-09T00:00:00Z

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12524425

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