Autophagy is important in islet homeostasis and compensatory increase of beta cell mass in response to high-fat diet. - Wikidata - wikimedia - TriplyDB

Autophagy is important in islet homeostasis and compensatory increase of beta cell mass in response to high-fat diet.

Autophagy is important in islet homeostasis and compensatory increase of beta cell mass in response to high-fat diet.

http://www.wikidata.org/entity/Q42442818

about

Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasis The Cvt pathway as a model for selective autophagy Endoplasmic reticulum stress and the inflammatory basis of metabolic disease Autophagy: a new target for nonalcoholic fatty liver disease therapy The Role of Organelle Stresses in Diabetes Mellitus and Obesity: Implication for Treatment Atg7 in development and disease: panacea or Pandora's Box?The role of autophagy in the pathogenesis of diabetic nephropathy How exercise may amend metabolic disturbances in diabetic cardiomyopathy Mitophagy in hematopoietic stem cells: the case for exploration Autophagy in cardiovascular biology Regulation of osteoclasts by membrane-derived lipid mediators Regulation of lipid stores and metabolism by lipophagy Mechanisms of mitochondria and autophagy crosstalk Involvement of Programmed Cell Death in Neurotoxicity of Metallic Nanoparticles: Recent Advances and Future Perspectives Mitochondrial dynamics and viral infections: A close nexus Mitochondrial regulation of β-cell function: maintaining the momentum for insulin release UCHL1 deficiency exacerbates human islet amyloid polypeptide toxicity in β-cells: evidence of interplay between the ubiquitin/proteasome system and autophagy Inflammation and cellular stress: a mechanistic link between immune-mediated and metabolically driven pathologies Coordinated regulation of protein synthesis and degradation by mTORC1 Selective autophagy mediated by autophagic adapter proteins Association of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunction Comparative gene identification-58 (CGI-58) promotes autophagy as a putative lysophosphatidylglycerol acyltransferase Pancreatic β-cell death in response to pro-inflammatory cytokines is distinct from genuine apoptosis Palmitate activates autophagy in INS-1E β-cells and in isolated rat and human pancreatic islets Stimulation of autophagy improves endoplasmic reticulum stress-induced diabetes Autophagy proteins control goblet cell function by potentiating reactive oxygen species production PLIN2 is a Key Regulator of the Unfolded Protein Response and Endoplasmic Reticulum Stress Resolution in Pancreatic β Cells Autophagy in mammalian development and differentiation Autophagy and metabolism Hepatic autophagy is suppressed in the presence of insulin resistance and hyperinsulinemia: inhibition of FoxO1-dependent expression of key autophagy genes by insulin.The diabetes-susceptible gene SLC30A8/ZnT8 regulates hepatic insulin clearance Autophagy regulates endothelial cell processing, maturation and secretion of von Willebrand factor Frequency Modulated Translocational Oscillations of Nrf2 Mediate the Antioxidant Response Element Cytoprotective Transcriptional Response.Pancreatic β-cell proliferation in obesity.Role and mechanism of pancreatic β-cell death in diabetes: The emerging role of autophagy.2-Methoxyestradiol ameliorates glucose tolerance with the increase in β-cell mass in db/db mice.Augmented reduction of islet β-cell mass in Goto-Kakizaki rats fed high-fat diet and its suppression by pitavastatin treatment.Mitochondrial dysfunction and oxidative stress mediate the physiological impairment induced by the disruption of autophagy Protective role of human insulin against the cytotoxicity associated with human mutant S20G islet amyloid polypeptide.Age-associated changes of islet endocrine cells and the effects of body mass index in Japanese.

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P2860

Autophagy is important in islet homeostasis and compensatory increase of beta cell mass in response to high-fat diet.

http://www.wikidata.org/entity/Q42442818

description

2008 nî lūn-bûn

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2008年の論文

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2008年论文

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2008年论文

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Autophagy is important in isle ...... in response to high-fat diet.

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Autophagy is important in isle ...... in response to high-fat diet.

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type

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Autophagy is important in isle ...... in response to high-fat diet.

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Autophagy is important in isle ...... in response to high-fat diet.

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Autophagy is important in isle ...... in response to high-fat diet.

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Autophagy is important in isle ...... in response to high-fat diet.

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J.CMET.2008.08.009

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Cell Metabolism

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Autophagy is important in isle ...... in response to high-fat diet.

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Chie Ebato

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Eiki Kominami

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Keiji Tanaka

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Koji Komiya

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Kosuke Azuma

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Masayuki Arakawa

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Ryuzo Kawamori

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Takahisa Hirose

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Takashi Ueno

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Toyoyoshi Uchida

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325-332

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scholarly article

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10.1016/J.CMET.2008.08.009

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4

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Hirotaka Watada

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2008-10-01T00:00:00Z

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