Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress. - Wikidata - wikimedia - TriplyDB

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

http://www.wikidata.org/entity/Q42486562

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Mitophagy programs: mechanisms and physiological implications of mitochondrial targeting by autophagy A Mechanistic Review of Mitophagy and Its Role in Protection against Alcoholic Liver Disease Molecular mechanisms that differentiate apoptosis from programmed necrosis Mitophagy: mechanisms, pathophysiological roles, and analysis Mitochondria and mitophagy: the yin and yang of cell death control The critical roles of mitophagy in cerebral ischemia The fibrosis-cell death axis in heart failure Proapoptotic Nix activates the JNK pathway by interacting with POSH and mediates death in a Parkinson disease model NIX is required for programmed mitochondrial clearance during reticulocyte maturation Nix Nought Nothing: fairy tale or real deal.BNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway.EndoG links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy.BNIP3 subfamily BH3-only proteins: mitochondrial stress sensors in normal and pathological functions.Mitochondrial pruning by Nix and BNip3: an essential function for cardiac-expressed death factors Signaling pathways in mitochondrial dysfunction and aging.Two close, too close: sarcoplasmic reticulum-mitochondrial crosstalk and cardiomyocyte fate Loss of Nix in Pdx1-deficient mice prevents apoptotic and necrotic β cell death and diabetes Nix protein positively regulates NF-κB activation in gliomas Peroxisome proliferator-activated receptor δ agonist, HPP593, prevents renal necrosis under chronic ischemia.Mechanisms and biology of B-cell leukemia/lymphoma 2/adenovirus E1B interacting protein 3 and Nip-like protein X.The mitochondrial dynamism-mitophagy-cell death interactome: multiple roles performed by members of a mitochondrial molecular ensemble.Oxygen Sensing, Cardiac Ischemia, HIF-1α and Some Emerging Concepts Role of BNIP3 and NIX in cell death, autophagy, and mitophagy Ataxia telangiectasia mutated kinase plays a protective role in β-adrenergic receptor-stimulated cardiac myocyte apoptosis and myocardial remodeling.Receptor-independent protein kinase C alpha (PKCalpha) signaling by calpain-generated free catalytic domains induces HDAC5 nuclear export and regulates cardiac transcription.The stress-related hormone norepinephrine induced upregulation of Nix, contributing to ECM protein expression.Cell death signalling mechanisms in heart failure.Mechanisms of non-apoptotic programmed cell death in diabetes and heart failure.Unrestrained erythroblast development in Nix-/- mice reveals a mechanism for apoptotic modulation of erythropoiesis.Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice.p75 neurotrophin receptor and its novel interaction partner, NIX, are involved in neuronal apoptosis after intracerebral hemorrhage.Bcl-2 family members and apoptosis, taken to heart.Decompensation of cardiac hypertrophy: cellular mechanisms and novel therapeutic targets.A nucleus-targeted alternately spliced Nix/Bnip3L protein isoform modifies nuclear factor κB (NFκB)-mediated cardiac transcription.Nutrient-sensing mTORC1: Integration of metabolic and autophagic signals Endoplasmic reticulum-mitochondria crosstalk in NIX-mediated murine cell death Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling.Interconnections between apoptotic, autophagic and necrotic pathways: implications for cancer therapy development.The role of Bcl-2 family member BNIP3 in cell death and disease: NIPping at the heels of cell death.

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P2860

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

http://www.wikidata.org/entity/Q42486562

description

2005 nî lūn-bûn

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2005年の論文

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2005年论文

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name

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@en

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@nl

type

label

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@en

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@nl

prefLabel

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@en

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

@nl

P1433

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P1433

Journal of Biological Chemistry

P1476

Distinct pathways regulate proapoptotic Nix and BNip3 in cardiac stress.

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P2093

Anita S Gálvez

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Bonnie J Benner

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Eric W Brunskill

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Gerald W Dorn

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Kelly M Regula

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Lorrie A Kirschenbaum

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Yehia Marreez

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P2860

Protein kinase Calpha negatively regulates systolic and diastolic function in pathological hypertrophy

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

Inducible expression of manganese superoxide dismutase by phorbol 12-myristate 13-acetate is mediated by Sp1 in endothelial cells

Nix and Nip3 form a subfamily of pro-apoptotic mitochondrial proteins

Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3.

The proapoptotic factor Nix is coexpressed with Bcl-xL during terminal erythroid differentiation.

The cardiac beta-myosin heavy chain isogene is induced selectively in alpha 1-adrenergic receptor-stimulated hypertrophy of cultured rat heart myocytes

Isolation, mapping, and functional analysis of a novel human cDNA (BNIP3L) encoding a protein homologous to human NIP3.

Understanding the impact of mitochondrial defects in cardiovascular disease: a review.

BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

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1442-1448

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10.1074/JBC.M509056200

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3

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281

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2005-11-16T00:00:00Z

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16291751

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