Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
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WISP-1/CCN4 regulates osteogenesis by enhancing BMP-2 activityWISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte deathWNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosisWnts talking with the TGF-β superfamily: WISPers about modulation of osteoarthritisTargeting disease through novel pathways of apoptosis and autophagyInterleukin-18/WNT1-inducible signaling pathway protein-1 signaling mediates human saphenous vein smooth muscle cell proliferationAngiotensin-II type 1 receptor and NOX2 mediate TCF/LEF and CREB dependent WISP1 induction and cardiomyocyte hypertrophyA role for Wnt signaling genes in the pathogenesis of impaired lung function in asthmaSerum levels of WNT1-inducible signaling pathway protein-1 (WISP-1): a noninvasive biomarker of renal fibrosis in subjects with chronic kidney disease.WNT1-inducible signaling pathway protein-1 activates diverse cell survival pathways and blocks doxorubicin-induced cardiomyocyte deathThe Nox1/4 Dual Inhibitor GKT137831 or Nox4 Knockdown Inhibits Angiotensin-II-Induced Adult Mouse Cardiac Fibroblast Proliferation and Migration. AT1 Physically Associates With Nox4.Resveratrol inhibits high glucose-induced PI3K/Akt/ERK-dependent interleukin-17 expression in primary mouse cardiac fibroblasts.The small molecule Wnt signaling modulator ICG-001 improves contractile function in chronically infarcted rat myocardium.Matricellular proteins in cardiac adaptation and disease.Wnt1 inducible signaling pathway protein 1 (WISP1) blocks neurodegeneration through phosphoinositide 3 kinase/Akt1 and apoptotic mitochondrial signaling involving Bad, Bax, Bim, and Bcl-xLWISP1 (CCN4) autoregulates its expression and nuclear trafficking of β-catenin during oxidant stress with limited effects upon neuronal autophagy.Aldehyde dehydrogenase-2 protects against myocardial infarction-related cardiac fibrosis through modulation of the Wnt/β-catenin signaling pathway.Extracellular matrix and fibroblast communication following myocardial infarction.Wnt1 inducible signaling pathway protein 1 (WISP1) targets PRAS40 to govern β-amyloid apoptotic injury of microglia.Tuberous sclerosis protein 2 (TSC2) modulates CCN4 cytoprotection during apoptotic amyloid toxicity in microglia.WISP1 neuroprotection requires FoxO3a post-translational modulation with autoregulatory control of SIRT1Myocardial AKT: the omnipresent nexus.Neutralization of interleukin-18 ameliorates ischemia/reperfusion-induced myocardial injury.The myofibroblast matrix: implications for tissue repair and fibrosis.Role of the Wnt-Frizzled system in cardiac pathophysiology: a rapidly developing, poorly understood area with enormous potential.Cellular and molecular actions of CCN2/CTGF and its role under physiological and pathological conditions.EMX2 activates slow myosin heavy chain 2 gene expression in embryonic muscle fibers.Wnt5a participates in hepatic stellate cell activation observed by gene expression profile and functional assays.Transcriptional remodeling of rapidly stimulated HL-1 atrial myocytes exhibits concordance with human atrial fibrillationTranscriptional changes in right ventricular tissues are enriched in the outflow tract compared with the apex during chronic pulmonary embolism in rats.Wnt Signaling in Cardiac Disease.Involvement of multiple CCN family members in platelets that support regeneration of joint tissues.Functional Effects of WNT1-Inducible Signaling Pathway Protein-1 on Bronchial Smooth Muscle Cell Migration and Proliferation in OVA-Induced Airway Remodeling.Targeting TRAF3IP2 by Genetic and Interventional Approaches Inhibits Ischemia/Reperfusion-induced Myocardial Injury and Adverse Remodeling.The food contaminant and nephrotoxin ochratoxin A enhances Wnt1 inducible signaling protein 1 and tumor necrosis factor-α expression in human primary proximal tubule cells.Loss of secreted frizzled-related protein-1 leads to deterioration of cardiac function in mice and plays a role in human cardiomyopathy.A functional analysis of Wnt inducible signalling pathway protein -1 (WISP-1/CCN4).Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice.Blockade of CCN4 attenuates CCl4-induced liver fibrosis.Anti-fibrotic effect of CCN3 accompanied by altered gene expression profile of the CCN family
P2860
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P2860
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@en
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@nl
type
label
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@en
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@nl
prefLabel
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@en
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@nl
P2093
P2860
P1476
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor.
@en
P2093
Chandrasekar B
Colston JT
Freeman GL
Gonzales K
de la Rosa SD
P2860
P304
P356
10.1152/AJPHEART.00428.2007
P577
2007-07-06T00:00:00Z