Molecular mechanisms of N-formyl-methionyl-leucyl-phenylalanine-induced superoxide generation and degranulation in mouse neutrophils: phospholipase D is dispensable.
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The phospholipase D superfamily as therapeutic targetsMembrane-binding and activation of LKB1 by phosphatidic acid is essential for development and tumour suppression.PLD1 promotes dendritic spine development by inhibiting ADAM10-mediated N-cadherin cleavage.Cellular and physiological roles for phospholipase D1 in cancer.Interactions between Neutrophils and Leishmania braziliensis Amastigotes Facilitate Cell Activation and Parasite ClearancePhysiological and pathophysiological roles for phospholipase D.Phospholipase D regulates the size of skeletal muscle cells through the activation of mTOR signaling.The role of diacylglycerol kinase ζ and phosphatidic acid in the mechanical activation of mammalian target of rapamycin (mTOR) signaling and skeletal muscle hypertrophyNew roles for Smad signaling and phosphatidic acid in the regulation of skeletal muscle mass.The role of mTORC1 in regulating protein synthesis and skeletal muscle mass in response to various mechanical stimuli.The immediate protective response to microbial challenge.Role of phospholipase d in g-protein coupled receptor functionTargeting phospholipase D in cancer, infection and neurodegenerative disorders.Phosphatidic acid-dependent recruitment and function of the Rac activator DOCK1 during dorsal ruffle formation.Inhibition of formyl peptide-stimulated phospholipase D activation by Fal-002-2 via blockade of the Arf6, RhoA and protein kinase C signaling pathways in rat neutrophils.ROS and glutathionylation balance cytoskeletal dynamics in neutrophil extracellular trap formation.Measuring Phospholipase D Enzymatic Activity Through Biochemical and Imaging Methods.Physiological function of phospholipase D2 in anti-tumor immunity: regulation of CD8+ T lymphocyte proliferation.Neutrophil extracellular trap formation requires OPA1-dependent glycolytic ATP productionRNA-seq of muscle from pigs divergent in feed efficiency and product quality identifies differences in immune response, growth, and macronutrient and connective tissue metabolism
P2860
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P2860
Molecular mechanisms of N-formyl-methionyl-leucyl-phenylalanine-induced superoxide generation and degranulation in mouse neutrophils: phospholipase D is dispensable.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@en
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@nl
type
label
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@en
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@nl
prefLabel
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@en
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@nl
P2093
P2860
P356
P1476
Molecular mechanisms of N-form ...... hospholipase D is dispensable.
@en
P2093
Megumi Sakamoto
Takanobu Sato
Tsunaki Hongu
Yasunori Kanaho
Yuji Funakoshi
P2860
P304
P356
10.1128/MCB.00869-12
P407
P577
2012-10-29T00:00:00Z