Nf1+/- mice have increased neointima formation via hyperactivation of a Gleevec sensitive molecular pathway. - Wikidata - wikimedia - TriplyDB

Nf1+/- mice have increased neointima formation via hyperactivation of a Gleevec sensitive molecular pathway.

Nf1+/- mice have increased neointima formation via hyperactivation of a Gleevec sensitive molecular pathway.

http://www.wikidata.org/entity/Q42538041

about

RASopathies: unraveling mechanisms with animal models Alternative splicing of the neurofibromatosis type I pre-mRNA Small G proteins in the cardiovascular system: physiological and pathological aspects A new horizon of moyamoya disease and associated health risks explored through RNF213 Genetic and cellular evidence of vascular inflammation in neurofibromin-deficient mice and humans.Mast cells and the neurofibroma microenvironment The NF1 gene revisited - from bench to bedside.Nf1 limits epicardial derivative expansion by regulating epithelial to mesenchymal transition and proliferation Matrix-dependent perturbation of TGFβ signaling and disease Spectrum and prevalence of vasculopathy in pediatric neurofibromatosis type 1.Partial Blindness to Submicron Topography in NF1 Haploinsufficient Cultured Fibroblasts Indicates a New Function of Neurofibromin in Regulation of Mechanosensoric.Plexiform neurofibroma genesis: questions of Nf1 gene dose and hyperactive mast cells Heterozygous inactivation of the Nf1 gene in myeloid cells enhances neointima formation via a rosuvastatin-sensitive cellular pathway Nf1+/- monocytes/macrophages induce neointima formation via CCR2 activation.Pathogenesis of plexiform neurofibroma: tumor-stromal/hematopoietic interactions in tumor progression.Ras-Mek-Erk signaling regulates Nf1 heterozygous neointima formation.Genetic variants promoting smooth muscle cell proliferation can result in diffuse and diverse vascular diseases: evidence for a hyperplastic vasculomyopathy.Imatinib mesylate for plexiform neurofibromas in patients with neurofibromatosis type 1: a phase 2 trial.Expert opinion on emerging drugs in childhood arterial ischemic stroke.Neurocutaneous Disorders for the Practicing Neurologist: a Focused Review.The G-quadruplex ligand, SYUIQ-FM05, targets proto-oncogene c-kit transcription and induces apoptosis in K562 cells.Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans.Neurofibromatosis 1-associated panhypopituitarism presenting as hypoglycaemic seizures and stroke-like symptoms.Cerebral vasculopathy in a Chinese family with neurofibromatosis type I mutation.Thoracic aortic disease in tuberous sclerosis complex: molecular pathogenesis and potential therapies in Tsc2+/- mice.Moyamoya syndrome in children with neurofibromatosis type 1: Italian-French experience.Restoring functional neurofibromin by protein transduction.Use of imatinib in a patient with cutaneous vasculopathy in the context of von Recklinghausen disease/neurofibromatosis.

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P2860

Nf1+/- mice have increased neointima formation via hyperactivation of a Gleevec sensitive molecular pathway.

http://www.wikidata.org/entity/Q42538041

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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type

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Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

@en

Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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Human Molecular Genetics

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Nf1+/- mice have increased neo ...... c sensitive molecular pathway.

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P2093

D Wade Clapp

http://www.w3.org/2001/XMLSchema#string

David A Ingram

http://www.w3.org/2001/XMLSchema#string

Elisabeth A Lasater

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Fang Li

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Laura E Mead

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Simon J Conway

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Waylan K Bessler

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Whitney E Horn

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P2860

Dysregulation of HSG triggers vascular proliferative disorders

Tumor microenvironment and neurofibromatosis type I: connecting the GAPs

Insights into the pathogenesis of neurofibromatosis 1 vasculopathy

The Ki-67 protein: from the known and the unknown

NF1 tumor suppressor gene function: narrowing the GAP

Proteins regulating Ras and its relatives

The neurofibromatosis type 1 gene encodes a protein related to GAP

Nf1 has an essential role in endothelial cells

Neurofibromin is a novel regulator of RAS-induced signals in primary vascular smooth muscle cells

Somatic inactivation of Nf1 in hematopoietic cells results in a progressive myeloproliferative disorder

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10.1093/HMG/DDN134

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15

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17

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18442999

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2733812

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