Clone-specific secondary aberrations are not detected in neonatal blood spots of children with ETV6-RUNX1-positive leukemia.

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Return of Results from Research Using Newborn Screening Dried Blood Samples

P2860

Q36197045-5799C872-53B7-4453-8163-A7A8277B288C

P2860

Clone-specific secondary aberrations are not detected in neonatal blood spots of children with ETV6-RUNX1-positive leukemia.

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http://www.wikidata.org/entity/Q42583768

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

@zh-hk

2013年論文

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2013年論文

@zh-tw

2013年论文

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2013年论文

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2013年论文

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name

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@en

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@nl

type

Item

label

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@en

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@nl

prefLabel

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@en

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@nl

P1476

Clone-specific secondary aberr ...... ETV6-RUNX1-positive leukemia.

@en

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Claus Meyer

http://www.w3.org/2001/XMLSchema#string

Georg Mann

http://www.w3.org/2001/XMLSchema#string

Maria Morak

http://www.w3.org/2001/XMLSchema#string

Oskar A Haas

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P2860

Chromosome 12p deletions in TEL-AML1 childhood acute lymphoblastic leukemia are associated with retrotransposon elements and occur postnatally

Genome-wide analysis of genetic alterations in acute lymphoblastic leukaemia

Backtracking leukemia to birth: identification of clonotypic gene fusion sequences in neonatal blood spots.

Chromosome translocations and covert leukemic clones are generated during normal fetal development

The origin and nature of tightly clustered BTG1 deletions in precursor B-cell acute lymphoblastic leukemia support a model of multiclonal evolution.

Small sizes and indolent evolutionary dynamics challenge the potential role of P2RY8-CRLF2-harboring clones as main relapse-driving force in childhood ALL.

Developmental timing of mutations revealed by whole-genome sequencing of twins with acute lymphoblastic leukemia.

Origins of chromosome translocations in childhood leukaemia.

Role of the TEL-AML1 fusion gene in the molecular pathogenesis of childhood acute lymphoblastic leukaemia.

Genomic analysis of the clonal origins of relapsed acute lymphoblastic leukemia.

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e108-10

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scholarly article

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10.3324/HAEMATOL.2013.090860

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http://www.w3.org/2001/XMLSchema#string

P478

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Rolf Marschalek

Renate Panzer-Grümayer

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2013-09-01T00:00:00Z

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256400916

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24006408

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3762111

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Clone-specific secondary aberrations are not detected in neonatal blood spots of children with ETV6-RUNX1-positive leukemia.

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P2860

P2860

Clone-specific secondary aberrations are not detected in neonatal blood spots of children with ETV6-RUNX1-positive leukemia.

description

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type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P932