Mitochondrial membrane lipidome defines yeast longevity.
about
A novel approach to the discovery of anti-tumor pharmaceuticals: searching for activators of liponecrosisThe many ways to age for a single yeast cellSix plant extracts delay yeast chronological aging through different signaling pathwaysEmpirical Validation of a Hypothesis of the Hormetic Selective Forces Driving the Evolution of Longevity Regulation MechanismsDiscovery of plant extracts that greatly delay yeast chronological aging and have different effects on longevity-defining cellular processesMechanisms Underlying the Essential Role of Mitochondrial Membrane Lipids in Yeast Chronological AgingMechanisms underlying the anti-aging and anti-tumor effects of lithocholic bile acidThe fusogenic lipid phosphatidic acid promotes the biogenesis of mitochondrial outer membrane protein Ugo1Quasi-programmed aging of budding yeast: a trade-off between programmed processes of cell proliferation, differentiation, stress response, survival and death defines yeast lifespan.Mechanisms by which different functional states of mitochondria define yeast longevity.Cell-autonomous mechanisms of chronological aging in the yeast Saccharomyces cerevisiaeSpecific changes in mitochondrial lipidome alter mitochondrial proteome and increase the geroprotective efficiency of lithocholic acid in chronologically aging yeastMechanism of liponecrosis, a distinct mode of programmed cell death.Lithocholic bile acid accumulated in yeast mitochondria orchestrates a development of an anti-aging cellular pattern by causing age-related changes in cellular proteome.A mitochondrially targeted compound delays aging in yeast through a mechanism linking mitochondrial membrane lipid metabolism to mitochondrial redox biology.A laboratory test of evolutionary aging theories.Caloric restriction extends yeast chronological lifespan via a mechanism linking cellular aging to cell cycle regulation, maintenance of a quiescent state, entry into a non-quiescent state and survival in the non-quiescent state.Mitochondria operate as signaling platforms in yeast aging.Living longer through mitochondrial housekeeping.Natural variation of chronological aging in the Saccharomyces cerevisiae species reveals diet-dependent mechanisms of life span control.Yeast Cells Exposed to Exogenous Palmitoleic Acid Either Adapt to Stress and Survive or Commit to Regulated Liponecrosis and Die.Caloric restriction delays yeast chronological aging by remodeling carbohydrate and lipid metabolism, altering peroxisomal and mitochondrial functionalities, and postponing the onsets of apoptotic and liponecrotic modes of regulated cell death.Mechanisms through which lithocholic acid delays yeast chronological aging under caloric restriction conditions
P2860
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P2860
Mitochondrial membrane lipidome defines yeast longevity.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
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2013年论文
@zh
2013年论文
@zh-cn
name
Mitochondrial membrane lipidome defines yeast longevity.
@en
Mitochondrial membrane lipidome defines yeast longevity.
@nl
type
label
Mitochondrial membrane lipidome defines yeast longevity.
@en
Mitochondrial membrane lipidome defines yeast longevity.
@nl
prefLabel
Mitochondrial membrane lipidome defines yeast longevity.
@en
Mitochondrial membrane lipidome defines yeast longevity.
@nl
P2093
P2860
P356
P1433
P1476
Mitochondrial membrane lipidome defines yeast longevity.
@en
P2093
Adam Beach
Anna Leonov
Michelle T Burstein
Mylène Juneau
Olivia Koupaki
Rachel Feldman
Simon D Bourque
Tatiana Iouk
Vincent R Richard
Vladimir I Titorenko
P2860
P304
P356
10.18632/AGING.100578
P577
2013-07-01T00:00:00Z