about
Mitral valve disease--morphology and mechanisms.Endoglin regulates mural cell adhesion in the circulatory systemRapamycin improves TIE2-mutated venous malformation in murine model and human subjectsMesenchymal stem cells and adipogenesis in hemangioma involution.Vascular endothelial growth factor receptor signaling is required for cardiac valve formation in zebrafish.Intravital molecular imaging of small-diameter tissue-engineered vascular grafts in mice: a feasibility study.Losartan inhibits endothelial-to-mesenchymal transformation in mitral valve endothelial cells by blocking transforming growth factor-β-induced phosphorylation of ERK.Differential functions of genes regulated by VEGF-NFATc1 signaling pathway in the migration of pulmonary valve endothelial cellsHuman pulmonary valve progenitor cells exhibit endothelial/mesenchymal plasticity in response to vascular endothelial growth factor-A and transforming growth factor-beta2.Hemogenic endothelial progenitor cells isolated from human umbilical cord bloodVasculogenesis in infantile hemangioma.Heart valve development: endothelial cell signaling and differentiationEndothelial progenitor cells as a sole source for ex vivo seeding of tissue-engineered heart valves.Functional small-diameter neovessels created using endothelial progenitor cells expanded ex vivo.Endothelial colony forming cells and mesenchymal progenitor cells form blood vessels and increase blood flow in ischemic muscle.Endothelial PGC-1α mediates vascular dysfunction in diabetes.A switch in Notch gene expression parallels stem cell to endothelial transition in infantile hemangioma.Host myeloid cells are necessary for creating bioengineered human vascular networks in vivo.Propranolol targets the contractility of infantile haemangioma-derived pericytes.E-selectin is required for the antiangiogenic activity of endostatinNeuropilin-1 functions as a VEGFR2 co-receptor to guide developmental angiogenesis independent of ligand bindingTargeting NF-κB in infantile hemangioma-derived stem cells reduces VEGF-A expression.Glucose transporter 1-positive endothelial cells in infantile hemangioma exhibit features of facultative stem cells.Monoclonal expansion of endothelial cells in hemangioma: an intrinsic defect with extrinsic consequences?Reciprocal interactions between mitral valve endothelial and interstitial cells reduce endothelial-to-mesenchymal transition and myofibroblastic activation.AKT hyper-phosphorylation associated with PI3K mutations in lymphatic endothelial cells from a patient with lymphatic malformation.Type I collagen, fibrin and PuraMatrix matrices provide permissive environments for human endothelial and mesenchymal progenitor cells to form neovascular networksVEGFR-1 mediates endothelial differentiation and formation of blood vessels in a murine model of infantile hemangioma.Bioengineered human vascular networks transplanted into secondary mice reconnect with the host vasculature and re-establish perfusion.Cyclic strain induces dual-mode endothelial-mesenchymal transformation of the cardiac valve.Aortic valve endothelial cells undergo transforming growth factor-beta-mediated and non-transforming growth factor-beta-mediated transdifferentiation in vitroIncreased Tie2 expression, enhanced response to angiopoietin-1, and dysregulated angiopoietin-2 expression in hemangioma-derived endothelial cells.Endothelial progenitor cells from infantile hemangioma and umbilical cord blood display unique cellular responses to endostatin.Hydrogel surfaces to promote attachment and spreading of endothelial progenitor cells.The GPR 55 agonist, L-α-lysophosphatidylinositol, mediates ovarian carcinoma cell-induced angiogenesis.Valvular interstitial cells suppress calcification of valvular endothelial cells.Clonality and altered behavior of endothelial cells from hemangiomas.E-selectin mediates stem cell adhesion and formation of blood vessels in a murine model of infantile hemangiomaThe endogenous zinc finger transcription factor, ZNF24, modulates the angiogenic potential of human microvascular endothelial cells.Somatic Activating Mutations in GNAQ and GNA11 Are Associated with Congenital Hemangioma
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Joyce Bischoff
@ast
Joyce Bischoff
@en
Joyce Bischoff
@es
Joyce Bischoff
@nl
Joyce Bischoff
@sl
type
label
Joyce Bischoff
@ast
Joyce Bischoff
@en
Joyce Bischoff
@es
Joyce Bischoff
@nl
Joyce Bischoff
@sl
prefLabel
Joyce Bischoff
@ast
Joyce Bischoff
@en
Joyce Bischoff
@es
Joyce Bischoff
@nl
Joyce Bischoff
@sl
P106
P1153
7101715717
P31
P496
0000-0002-6367-1974