The transcription factor STAT3 is required for T helper 2 cell development.
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The involvement of the JAK-STAT signaling pathway in chronic inflammatory skin disease atopic dermatitisSTAT6-dependent and -independent mechanisms in Th2 polarizationThe effects of TLR activation on T-cell development and differentiationTreatment of allergic asthma: modulation of Th2 cells and their responsesAlong the Axis between Type 1 and Type 2 Immunity; Principles Conserved in Evolution from Fish to MammalsInfluence of Aspergillus fumigatus conidia viability on murine pulmonary microRNA and mRNA expression following subchronic inhalation exposureAnalysis of time-resolved gene expression measurements across individualsArid5a regulates naive CD4+ T cell fate through selective stabilization of Stat3 mRNA.Computational analysis of cell-to-cell heterogeneity in single-cell RNA-sequencing data reveals hidden subpopulations of cells.Neutralization of interleukin-17 suppresses allergic rhinitis symptoms by downregulating Th2 and Th17 responses and upregulating the Treg response.Inhibition of Hematopoietic Cell Kinase Activity Suppresses Myeloid Cell-Mediated Colon Cancer ProgressionIL-10 induces a STAT3-dependent autoregulatory loop in TH2 cells that promotes Blimp-1 restriction of cell expansion via antagonism of STAT5 target genesThe adaptor TRAF5 limits the differentiation of inflammatory CD4(+) T cells by antagonizing signaling via the receptor for IL-6.Th17 cells demonstrate stable cytokine production in a proallergic environmentTranscriptional and epigenetic control of T helper cell specification: molecular mechanisms underlying commitment and plasticity.A simple theoretical framework for understanding heterogeneous differentiation of CD4+ T cells.The transcription factor Etv5 controls TH17 cell development and allergic airway inflammation.Transcriptional and epigenetic regulation of T-helper lineage specification.Targeting the JAK-STAT pathway in lymphoma: a focus on pacritinib.Genome-Wide Gene Expression Profiling Revealed a Critical Role for GATA3 in the Maintenance of the Th2 Cell Identity.Mechanisms controlling Th17 cytokine expression and host defenseS100A4-neutralizing antibody suppresses spontaneous tumor progression, pre-metastatic niche formation and alters T-cell polarization balanceLocal and systemic alterations in signal transducers and activators of transcription (STAT) associated with human abdominal aortic aneurysms.STAT6-dependent regulation of Th9 development.STAT1 is Constitutively Activated in the T/C28a2 Immortalized Juvenile Human Chondrocyte Line and Stimulated by IL-6 Plus Soluble IL-6RBatf is important for IL-4 expression in T follicular helper cells.Gene set enrichment analysis identifies LIF as a negative regulator of human Th2 cell differentiationIL-2 regulates expression of C-MAF in human CD4 T cellsGenetic partitioning of interleukin-6 signalling in mice dissociates Stat3 from Smad3-mediated lung fibrosis.JAK and STAT signaling molecules in immunoregulation and immune-mediated disease.Poly (ADP-ribose) polymerase 14 and its enzyme activity regulates T(H)2 differentiation and allergic airway disease.T-cell-intrinsic Tif1α/Trim24 regulates IL-1R expression on TH2 cells and TH2 cell-mediated airway allergy.Distinct transcriptional regulatory modules underlie STAT3's cell type-independent and cell type-specific functions.Helper T-cell identity and evolution of differential transcriptomes and epigenomes.Interleukin-9 is required for allergic airway inflammation mediated by the cytokine TSLP.STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy.Early in vivo signaling profiles in MUC1-specific CD4(+) T cells responding to two different MUC1-targeting vaccines in two different microenvironmentsWheezing and itching: The requirement for STAT proteins in allergic inflammationDecreased STAT5 phosphorylation and GATA-3 expression in NOX2-deficient T cells: role in T helper developmentComprehensive genomic profiling in diabetic nephropathy reveals the predominance of proinflammatory pathways.
P2860
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P2860
The transcription factor STAT3 is required for T helper 2 cell development.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
The transcription factor STAT3 is required for T helper 2 cell development.
@en
The transcription factor STAT3 is required for T helper 2 cell development.
@nl
type
label
The transcription factor STAT3 is required for T helper 2 cell development.
@en
The transcription factor STAT3 is required for T helper 2 cell development.
@nl
prefLabel
The transcription factor STAT3 is required for T helper 2 cell development.
@en
The transcription factor STAT3 is required for T helper 2 cell development.
@nl
P2093
P2860
P1433
P1476
The transcription factor STAT3 is required for T helper 2 cell development.
@en
P2093
Evelyn T Nguyen
Gretta L Stritesky
Jared Travers
Rajarajeswari Muthukrishnan
Ritobrata Goswami
Sarita Sehra
P2860
P356
10.1016/J.IMMUNI.2010.12.013
P407
P577
2011-01-06T00:00:00Z