Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain.
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Stretch-activated ion channels: what are they?RNA-Seq Analysis of Human Trigeminal and Dorsal Root Ganglia with a Focus on ChemoreceptorsMyogenic temporomandibular disorders: Clinical systemic comorbidities in a female population sample.Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation.Sodium channel Na v 1.7 immunoreactivity in painful human dental pulp and burning mouth syndrome.Multiple roles for NaV1.9 in the activation of visceral afferents by noxious inflammatory, mechanical, and human disease-derived stimuli.Comprehensive RNA-Seq expression analysis of sensory ganglia with a focus on ion channels and GPCRs in Trigeminal gangliaAnimal toxins can alter the function of Nav1.8 and Nav1.9.Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism.Mechanism of sodium channel NaV1.9 potentiation by G-protein signalingThe Nav1.9 channel regulates colonic motility in mice.Neurological perspectives on voltage-gated sodium channels.Transduction and encoding sensory information by skin mechanoreceptors.The voltage-gated sodium channel NaV 1.9 in visceral pain.Reduction of epithelial secretion in male rat distal colonic mucosa by bile acid receptor TGR5 agonist, INT-777: role of submucosal neurons.Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy.The role of Nav1.9 channel in the development of neuropathic orofacial pain associated with trigeminal neuralgia.Activation of neurokinin 3 receptor increases Na(v)1.9 current in enteric neurons.Voltage-gated sodium channels in nociceptive versus non-nociceptive nodose vagal sensory neurons innervating guinea pig lungs.GTP up-regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9.Electrophysiological and Pharmacological Analyses of Nav1.9 Voltage-Gated Sodium Channel by Establishing a Heterologous Expression System.Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception.Sodium channels and pain: from toxins to therapies.Enteric glial activity regulates secretomotor function in the mouse colon but does not acutely affect gut permeability.Segmental differences in the non-neuronal cholinergic system in rat caecum.Membrane cholesterol depletion as a trigger of Nav1.9 channel-mediated inflammatory pain.Sema3A chemorepellant regulates the timing and patterning of dental nerves during development of incisor tooth germ.The Role of Nav1.9 Channel in the Development of Neuropathic Orofacial Pain Associated with Trigeminal Neuralgia.Novel Sodium Channel Inhibitor From Leeches.Novel Sodium Channel Inhibitor From Leeches.
P2860
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P2860
Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Expression and localization of ...... x function and orofacial pain.
@en
Expression and localization of ...... x function and orofacial pain.
@nl
type
label
Expression and localization of ...... x function and orofacial pain.
@en
Expression and localization of ...... x function and orofacial pain.
@nl
prefLabel
Expression and localization of ...... x function and orofacial pain.
@en
Expression and localization of ...... x function and orofacial pain.
@nl
P2093
P1476
Expression and localization of ...... x function and orofacial pain.
@en
P2093
Amy M Ritter
Bertrand Coste
François Maingret
Françoise Padilla
Henry Magloire
Marcel Crest
Marie-Lise Couble
Nadine Clerc
Patrick Delmas
P304
P356
10.1016/J.MCN.2007.02.008
P577
2007-02-15T00:00:00Z