Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception.

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Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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Voltage-gated sodium channels: ...... ution to visceral nociception.

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Voltage-gated sodium channels:

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label

Voltage-gated sodium channels: ...... ution to visceral nociception.

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Voltage-gated sodium channels:

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Voltage-gated sodium channels: ...... ution to visceral nociception.

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Voltage-gated sodium channels:

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P1476

Voltage-gated sodium channels: ...... bution to visceral nociception

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P2093

Andelain Erickson

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Annemie Deiteren

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Ashlee Caldwell

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Sonia Garcia-Caraballo

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P2860

Molecular cloning and functional expression of the human sodium channel beta1B subunit, a novel splicing variant of the beta1 subunit

Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells

Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain

The neural control of micturition

A multicentre open-label safety and efficacy study of tetrodotoxin for cancer pain

Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons

Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.

Tetrodotoxin, an Extremely Potent Marine Neurotoxin: Distribution, Toxicity, Origin and Therapeutical Uses

SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes

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785-807

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scholarly article

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10.1113/JP273461

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English

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596

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P577

2018-02-06T00:00:00Z

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29318638

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P932

5830430

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