Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
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Bcl-xL phosphorylation at Ser49 by polo kinase 3 during cell cycle progression and checkpointsHexokinase II: cancer's double-edged sword acting as both facilitator and gatekeeper of malignancy when bound to mitochondriaThe role of VDAC in cell death: friend or foe?Regulated necrotic cell death: the passive aggressive side of Bax and BakAdenine nucleotide translocator transports haem precursors into mitochondriaA yeast BH3-only protein mediates the mitochondrial pathway of apoptosis.Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cellsAdenine nucleotide translocator isoforms 1 and 2 are differently distributed in the mitochondrial inner membrane and have distinct affinities to cyclophilin DMcl-1 promotes lung cancer cell migration by directly interacting with VDAC to increase mitochondrial Ca2+ uptake and reactive oxygen species generationActivation of Bak in ultrasound-induced, JNK- and p38-independent apoptosis and its inhibition by Bcl-2Mitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic TargetArabidopsis voltage-dependent anion channel 1 (AtVDAC1) is required for female development and maintenance of mitochondrial functions related to energy-transactionMitochondria, aging and longevity--a new perspective.Molecular mechanisms for apoptosis induced by signaling through the B cell antigen receptor.Programmed death in yeast as adaptation?Down-regulation of adenine nucleotide translocase 3 and its role in camptothecin-induced apoptosis in human hepatoma QGY7703 cells.Peptides in apoptosis research.The mitochondrial death pathway: a promising therapeutic target in diseasesKnockout of Vdac1 activates hypoxia-inducible factor through reactive oxygen species generation and induces tumor growth by promoting metabolic reprogramming and inflammation.Mediation of the antiapoptotic activity of Bcl-xL protein upon interaction with VDAC1 proteinMinute kinetics of proapoptotic proteins: BAX and Smac/DIABLO in living tumor cells revealed by homeostatic confocal microscopy.Basic Fibroblast Growth Factor Inhibits Apoptosis and Promotes Proliferation of Adipose-Derived Mesenchymal Stromal Cells Isolated from Patients with Type 2 Diabetes by Reducing Cellular Oxidative Stress.Ethanol influences on Bax associations with mitochondrial membrane proteins in neonatal rat cerebellum.VDAC1: from structure to cancer therapy.Functions of BCL-X L at the Interface between Cell Death and MetabolismClinical implication of voltage-dependent anion channel 1 in uterine cervical cancer and its action on cervical cancer cells.NMR structural investigation of the mitochondrial outer membrane protein VDAC and its interaction with antiapoptotic Bcl-xL.An interaction between Bcl-xL and the voltage-dependent anion channel (VDAC) promotes mitochondrial Ca2+ uptake.VDAC regulation: role of cytosolic proteins and mitochondrial lipids.The mitochondrial permeability transition pore and ischemia-reperfusion injurySuppression of mitochondrial function by oxidatively truncated phospholipids is reversible, aided by bid, and suppressed by Bcl-XLMetabolic approaches to overcoming chemoresistance in ovarian cancer.Voltage-dependant anion channels: novel insights into isoform function through genetic models.The Mitochondrial Permeability Transition Pore and ATP Synthase.Yeast as a tool for studying proteins of the Bcl-2 familyDirected evolution of mammalian anti-apoptosis proteins by somatic hypermutationProteomic approaches in understanding action mechanisms of metal-based anticancer drugs.Deletion of the BH1 domain of Bcl-2 accelerates apoptosis by acting in a dominant negative fashion.Mitochondrial voltage-dependent anion channel is involved in dopamine-induced apoptosis.Bcl-xL blocks transforming growth factor-beta 1-induced apoptosis by inhibiting cytochrome c release and not by directly antagonizing Apaf-1-dependent caspase activation in prostate epithelial cells.
P2860
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P2860
Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@en
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@nl
type
label
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@en
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@nl
prefLabel
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@en
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@nl
P2093
P2860
P356
P1433
P1476
Bax and Bcl-xL independently r ...... enine nucleotide translocator.
@en
P2093
Shinohara Y
Tsujimoto Y
P2860
P2888
P304
P356
10.1038/SJ.ONC.1203788
P407
P577
2000-09-01T00:00:00Z
P6179
1050286271