about
Disruption of SUMO-specific protease 2 induces mitochondria mediated neurodegenerationThe mouse Fused locus encodes Axin, an inhibitor of the Wnt signaling pathway that regulates embryonic axis formationIdentification of a domain of Axin that binds to the serine/threonine protein phosphatase 2A and a self-binding domainSUMO-specific protease 2 is essential for modulating p53-Mdm2 in development of trophoblast stem cell niches and lineagesThe role of Axin2 in calvarial morphogenesis and craniosynostosisReciprocal regulation of Wnt and Gpr177/mouse Wntless is required for embryonic axis formationSUMO-specific protease 2 in Mdm2-mediated regulation of p53Gpr177 regulates pulmonary vasculature developmentTransgenic control of mitochondrial fission induces mitochondrial uncoupling and relieves diabetic oxidative stress.Gpr177 deficiency impairs mammary development and prohibits Wnt-induced tumorigenesis.Manipulating gene activity in Wnt1-expressing precursors of neural epithelial and neural crest cells.Development of a unique system for spatiotemporal and lineage-specific gene expression in mice.Expression of Gpr177, a Wnt trafficking regulator, in mouse embryogenesisThe balance of WNT and FGF signaling influences mesenchymal stem cell fate during skeletal development.Runx2 is required for early stages of endochondral bone formation but delays final stages of bone repair in Axin2-deficient mice.Axin1 prevents Salmonella invasiveness and inflammatory response in intestinal epithelial cellsEpidermal Wnt controls hair follicle induction by orchestrating dynamic signaling crosstalk between the epidermis and dermis.β-catenin/cyclin D1 mediated development of suture mesenchyme in calvarial morphogenesis.Gpr177/mouse Wntless is essential for Wnt-mediated craniofacial and brain development.The inductive role of Wnt-β-Catenin signaling in the formation of oral apparatus.Derivation of mouse trophoblast stem cells from blastocysts.BMP-FGF signaling axis mediates Wnt-induced epidermal stratification in developing mammalian skin.Impaired neural development caused by inducible expression of Axin in transgenic mice.Cartilage-specific β-catenin signaling regulates chondrocyte maturation, generation of ossification centers, and perichondrial bone formation during skeletal developmentImpaired mammary gland and lymphoid development caused by inducible expression of Axin in transgenic mice.Stem cells of the suture mesenchyme in craniofacial bone development, repair and regeneration.Extraembryonic but not embryonic SUMO-specific protease 2 is required for heart development.Runx2 protein represses Axin2 expression in osteoblasts and is required for craniosynostosis in Axin2-deficient miceEctodermal Wnt controls nasal pit morphogenesis through modulation of the BMP/FGF/JNK signaling axis.Gpr177, a novel locus for bone mineral density and osteoporosis, regulates osteogenesis and chondrogenesis in skeletal developmentIntra-epithelial requirement of canonical Wnt signaling for tooth morphogenesis.Co-opted JNK/SAPK signaling in Wnt/beta-catenin-induced tumorigenesis.Sustained Neurog3 expression in hormone-expressing islet cells is required for endocrine maturation and functionAxin2 controls bone remodeling through the beta-catenin-BMP signaling pathway in adult mice.Domains of axin and disheveled required for interaction and function in wnt signaling.CDC42 inhibition suppresses progression of incipient intestinal tumors.BMP-2 induces ATF4 phosphorylation in chondrocytes through a COX-2/PGE2 dependent signaling pathway.Rap1b Is an Effector of Axin2 Regulating Crosstalk of Signaling Pathways During Skeletal Development.Motoneuron Wnts regulate neuromuscular junction developmentThe requirement of SUMO2/3 for SENP2 mediated extraembryonic and embryonic development
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Wei Hsu
@ast
Wei Hsu
@en
Wei Hsu
@es
Wei Hsu
@nl
Wei Hsu
@sl
type
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Wei Hsu
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Wei Hsu
@en
Wei Hsu
@es
Wei Hsu
@nl
Wei Hsu
@sl
prefLabel
Wei Hsu
@ast
Wei Hsu
@en
Wei Hsu
@es
Wei Hsu
@nl
Wei Hsu
@sl
P106
P1153
14015875100
P31
P496
0000-0001-6738-6030