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Functional expression of inward rectifier potassium channels in cultured human pulmonary smooth muscle cells: evidence for a major role of Kir2.4 subunitsProstacyclins have no direct inotropic effect on isolated atrial strips from the normal and pressure-overloaded human right heartInhibition of phosphodiesterase 2 augments cGMP and cAMP signaling to ameliorate pulmonary hypertension.The large-conductance Ca2+-activated K+ channel is essential for innate immunity.A comparative study of PGI2 mimetics used clinically on the vasorelaxation of human pulmonary arteries and veins, role of the DP-receptor.The pore-forming subunit of the K(ATP) channel is an important molecular target for LPS-induced vascular hyporeactivity in vitro.Role of KATP channels in sepsis.Vasopressin: mechanisms of action on the vasculature in health and in septic shock.The mechanistic basis of prostacyclin and its stable analogues in pulmonary arterial hypertension: Role of membrane versus nuclear receptors.IP receptor-dependent activation of PPARgamma by stable prostacyclin analogues.Autonomic receptors and cell membrane potential.Do anionic phospholipids serve as cofactors or second messengers for the regulation of activity of cloned ATP-sensitive K+ channels?Different molecular sites of action for the KATP channel inhibitors, PNU-99963 and PNU-37883A.BK large conductance Ca²+-activated K+ channel-deficient mice are not resistant to hypotension and display reduced survival benefit following polymicrobial sepsis.Binding and activity of the prostacyclin receptor (IP) agonists, treprostinil and iloprost, at human prostanoid receptors: treprostinil is a potent DP1 and EP2 agonist.Effects of the adenylyl cyclase inhibitor SQ22536 on iloprost-induced vasorelaxation and cyclic AMP elevation in isolated guinea-pig aorta.Differential effects of vasopressin and norepinephrine on vascular reactivity in a long-term rodent model of sepsis.Terlipressin for norepinephrine-resistant septic shock.Inhibition of vascular adenosine triphosphate-sensitive potassium channels by sympathetic tone during sepsis.The molecular site of action of K(ATP) channel inhibitors determines their ability to inhibit iNOS-mediated relaxation in rat aorta.NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.Variable effects of inhibiting iNOS and closing the vascular ATP-sensitive potassium channel (via its pore-forming and sulfonylurea receptor subunits) in endotoxic shock.Reversal of life-threatening, drug-related potassium-channel syndrome by glibenclamide.Ex vivo relaxations of pulmonary arteries induced by prostacyclin mimetics are highly dependent of the precontractile agents.Evidence that inward rectifier K+ channels mediate relaxation by the PGI2 receptor agonist cicaprost via a cyclic AMP-independent mechanism.The diverse effects of noradrenaline and other stimulants on 86Rb and 42K efflux in rabbit and guinea-pig arterial muscle.Attenuated vascular responsiveness to K+ channel openers in diabetes mellitus: the differential role of reactive oxygen species.Stoking up BKCa channels in hemorrhagic shock: which channel subunit is really fueling the fire?Outward currents in rabbit pulmonary artery cells dissociated with a new technique.Differential actions of the prostacyclin analogues treprostinil and iloprost and the selexipag metabolite, MRE-269 (ACT-333679) in rat small pulmonary arteries and veins.Nuclear translocation of calcineurin Abeta but not calcineurin Aalpha by platelet-derived growth factor in rat aortic smooth muscle.Abnormal activation of K+ channels underlies relaxation to bacterial lipopolysaccharide in rat aorta.Improving Interpretation of Cardiac Phenotypes and Enhancing Discovery With Expanded Knowledge in the Gene OntologyDual regulation of M current in gastric smooth muscle cells: beta-adrenergic-muscarinic antagonism.Role of prostanoid IP and EP receptors in mediating vasorelaxant responses to PGI2 analogues in rat tail artery: Evidence for Gi/o modulation via EP3 receptors.Prostanoid EP₂ Receptors Are Up-Regulated in Human Pulmonary Arterial Hypertension: A Key Anti-Proliferative Target for Treprostinil in Smooth Muscle CellsInotropic Effects of Prostacyclins on the Right Ventricle Are Abolished in Isolated Rat Hearts With Right-Ventricular Hypertrophy and FailureThe BK ChannelEvidence that Ca2+-activated K+ channels play a major role in mediating the vascular effects of iloprost and cicaprostProperties of the ATP-sensitive K+ current activated by levcromakalim in isolated pulmonary arterial myocytes
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Lucie H Clapp
@ast
Lucie H Clapp
@en
Lucie H Clapp
@es
Lucie H Clapp
@nl
Lucie H Clapp
@sl
type
label
Lucie H Clapp
@ast
Lucie H Clapp
@en
Lucie H Clapp
@es
Lucie H Clapp
@nl
Lucie H Clapp
@sl
prefLabel
Lucie H Clapp
@ast
Lucie H Clapp
@en
Lucie H Clapp
@es
Lucie H Clapp
@nl
Lucie H Clapp
@sl
P1053
B-1088-2008
P106
P21
P31
P3829
P496
0000-0001-7802-4481