NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
about
Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation.Factors, fiction and endothelium-derived hyperpolarizing factor.Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Combination of Ca2+ -activated K+ channel blockers inhibits acetylcholine-evoked nitric oxide release in rat superior mesenteric artery.Physiologically relevant measurements of nitric oxide in cardiovascular research using electrochemical microsensors.Endothelium-dependent hyperpolarizations: past beliefs and present facts.A role for nitroxyl (HNO) as an endothelium-derived relaxing and hyperpolarizing factor in resistance arteries.Upregulation of intermediate calcium-activated potassium channels counterbalance the impaired endothelium-dependent vasodilation in stroke-prone spontaneously hypertensive rats.Reduction of nitrite to nitric oxide during ischemia protects against myocardial ischemia-reperfusion damage.EDHF: an update.Endothelial calcium-activated potassium channels as therapeutic targets to enhance availability of nitric oxide.Modulation of vascular function by perivascular adipose tissue: the role of endothelium and hydrogen peroxide.Endothelium-derived hyperpolarization factor (EDHF) is up-regulated in a pig model of acute liver failure.Direct demonstration of beta1- and evidence against beta2- and beta3-adrenoceptors, in smooth muscle cells of rat small mesenteric arteries.Developmental changes in myoendothelial gap junction mediated vasodilator activity in the rat saphenous artery.Impaired EDHF-mediated vasodilatation in adult offspring of rats exposed to a fat-rich diet in pregnancy.Temporal characteristics of nitric oxide-, prostaglandin-, and EDHF-mediated components of endothelium-dependent vasodilation in the kidney.The effect of the polymerized bovine haemoglobin solution, Hb-200, on endothelial function in isolated arterial rings from rats.Intermittent hypoxia conditioning prevents endothelial dysfunction and improves nitric oxide storage in spontaneously hypertensive rats.The production of vasoconstriction-induced residual NO modulates perfusion pressure in rat mesenteric vascular bed.Vasorelaxant and Hypotensive Effects of an Ethanolic Extract of Eulophia macrobulbon and Its Main Compound 1-(4'-Hydroxybenzyl)-4,8-Dimethoxyphenanthrene-2,7-Diol.
P2860
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P2860
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
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2003年學術文章
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name
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@en
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@nl
type
label
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@en
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@nl
prefLabel
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@en
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores.
@nl
P2093
P50
P1476
NO contributes to EDHF-like responses in rat small arteries: a role for NO stores
@en
P2093
Awahan Rahman
Raymond MacAllister
Sharmila Chauhan
P304
P356
10.1016/S0008-6363(02)00611-9
P577
2003-01-01T00:00:00Z