Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
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Biology of Bone Tissue: Structure, Function, and Factors That Influence Bone CellsArterial calcification in chronic kidney disease: key roles for calcium and phosphatePhosphate/pyrophosphate and MV-related proteins in mineralisation: discoveries from mouse modelsThe role of phosphatases in the initiation of skeletal mineralizationThe phosphate transporter PiT1 (Slc20a1) revealed as a new essential gene for mouse liver developmentDipeptidyl peptidase-4 inhibitor gemigliptin protects against vascular calcification in an experimental chronic kidney disease and vascular smooth muscle cells.Mapping of the minimal inorganic phosphate transporting unit of human PiT2 suggests a structure universal to PiT-related proteins from all kingdoms of lifeThe appearance and modulation of osteocyte marker expression during calcification of vascular smooth muscle cells.Roles of major facilitator superfamily transporters in phosphate response in DrosophilaProstaglandin F2α: a bone remodeling mediator.Klotho deficiency causes vascular calcification in chronic kidney disease.Cellular ATP synthesis mediated by type III sodium-dependent phosphate transporter Pit-1 is critical to chondrogenesisGenetic determinants of phosphate response in Drosophila.Phosphate sensing.Mice with hypomorphic expression of the sodium-phosphate cotransporter PiT1/Slc20a1 have an unexpected normal bone mineralization.BMP-2 promotes phosphate uptake, phenotypic modulation, and calcification of human vascular smooth muscle cells.Adenosine Signaling Mediates Osteogenic Differentiation of Human Embryonic Stem Cells on Mineralized Matrices.Inorganic Phosphate as a Novel Signaling Molecule with Antiproliferative Action in MDA-MB-231 Breast Cancer Cells.Synergistic cytotoxic effects of inorganic phosphate and chemotherapeutic drugs on human osteosarcoma cells.Skeletal Mineralization Deficits and Impaired Biogenesis and Function of Chondrocyte-Derived Matrix Vesicles in Phospho1(-/-) and Phospho1/Pi t1 Double-Knockout Mice.An integrated understanding of the physiological response to elevated extracellular phosphate.The Interplay between the bone and the immune systemThe Na+-Pi cotransporter PiT-2 (SLC20A2) is expressed in the apical membrane of rat renal proximal tubules and regulated by dietary PiPhosphate: an old bone molecule but new cardiovascular risk factor.Calcium phosphate-bearing matrices induce osteogenic differentiation of stem cells through adenosine signaling.Osteocyte regulation of bone mineral: a little give and take.A Molecular View of Pathological Microcalcification in Breast Cancer.Sodium-dependent phosphate cotransporters and phosphate-induced calcification of vascular smooth muscle cells: redundant roles for PiT-1 and PiT-2.Inorganic phosphate enhances sensitivity of human osteosarcoma U2OS cells to doxorubicin via a p53-dependent pathway.Bioinspired Collagen Scaffolds in Cranial Bone Regeneration: From Bedside to Bench.Effects of Artemisia Princeps Supplementation on Bone Metabolism in Ovariectomized Rats.1alpha,25-dihydroxyvitamin D3 acts predominately in mature osteoblasts under conditions of high extracellular phosphate to increase fibroblast growth factor 23 production in vitro.Immobilization of alkaline phosphatase on microporous nanofibrous fibrin scaffolds for bone tissue engineering.Effects of extracellular phosphate on gene expression in murine osteoblasts.ZBTB16 induces osteogenic differentiation marker genes in dental follicle cells independent from RUNX2.Incisor enamel formation is impaired in transgenic rats overexpressing the type III NaPi transporter Slc20a1.bFGF and JAGGED1 regulate alkaline phosphatase expression and mineralization in dental tissue-derived mesenchymal stem cells.Phosphate (Pi)-regulated heterodimerization of the high-affinity sodium-dependent Pi transporters PiT1/Slc20a1 and PiT2/Slc20a2 underlies extracellular Pi sensing independently of Pi uptake.Loss of PiT-2 results in abnormal bone development and decreased bone mineral density and length in mice.Human stanniocalcin-1 or -2 expressed in mice reduces bone size and severely inhibits cranial intramembranous bone growth.
P2860
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P2860
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@en
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@nl
type
label
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@en
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@nl
prefLabel
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@en
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@nl
P2093
P2860
P356
P1476
Osteoblast autonomous Pi regulation via Pit1 plays a role in bone mineralization.
@en
P2093
G Antonio Candeliere
Jane E Aubin
Norihiko Maeda
Yuji Yoshiko
P2860
P304
P356
10.1128/MCB.00104-07
P407
P577
2007-04-16T00:00:00Z