TET-mediated oxidation of methylcytosine causes TDG or NEIL glycosylase dependent gene reactivation.
about
Modifiers and Readers of DNA Modifications and Their Impact on Genome Structure, Expression, and Stability in DiseaseThe Mechanisms of Generation, Recognition, and Erasure of DNA 5-Methylcytosine and Thymine OxidationsBinding of MBD proteins to DNA blocks Tet1 function thereby modulating transcriptional noise.Methyl-CpG binding domain protein 1 regulates localization and activity of Tet1 in a CXXC3 domain-dependent manner.Prolonged re-expression of the hypermethylated gene EPB41L3 using artificial transcription factors and epigenetic drugsUracil-DNA Glycosylase UNG Promotes Tet-mediated DNA DemethylationBiochemical reconstitution of TET1-TDG-BER-dependent active DNA demethylation reveals a highly coordinated mechanismRe-expression of Selected Epigenetically Silenced Candidate Tumor Suppressor Genes in Cervical Cancer by TET2-directed Demethylation.Tautomerization-dependent recognition and excision of oxidation damage in base-excision DNA repair.DNA damage and Repair Modify DNA methylation and Chromatin Domain of the Targeted Locus: Mechanism of allele methylation polymorphism.Genomic and epigenomic analysis of high-risk prostate cancer reveals changes in hydroxymethylation and TET1.Neil DNA glycosylases promote substrate turnover by Tdg during DNA demethylation.Role of Base Excision "Repair" Enzymes in Erasing Epigenetic Marks from DNADNA demethylation pathways: Additional players and regulators.Site-specific recruitment of epigenetic factors with a modular CRISPR/Cas system.New Insights into 5hmC DNA Modification: Generation, Distribution and Function.The Mbd4 DNA glycosylase protects mice from inflammation-driven colon cancer and tissue injury.Nei-like 1 (NEIL1) excises 5-carboxylcytosine directly and stimulates TDG-mediated 5-formyl and 5-carboxylcytosine excision.Phosphorylation of TET proteins is regulated via O-GlcNAcylation by the O-linked N-acetylglucosamine transferase (OGT).IFN-α-mediated Base Excision Repair Pathway Correlates with Antiviral Response Against Hepatitis B Virus Infection.L1 retrotransposition is activated by Ten-eleven-translocation protein 1 and repressed by methyl-CpG binding proteins.Functional impacts of 5-hydroxymethylcytosine, 5-formylcytosine, and 5-carboxycytosine at a single hemi-modified CpG dinucleotide in a gene promoter.Target specificity of mammalian DNA methylation and demethylation machinery.Responses of bovine early embryos to S-adenosyl methionine supplementation in culture.
P2860
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P2860
TET-mediated oxidation of methylcytosine causes TDG or NEIL glycosylase dependent gene reactivation.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@en
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@nl
type
label
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@en
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@nl
prefLabel
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@en
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@nl
P2093
P2860
P356
P1476
TET-mediated oxidation of meth ...... e dependent gene reactivation.
@en
P2093
Andrea Rottach
Heinrich Leonhardt
Michael Siegl
Udo Müller
P2860
P304
P356
10.1093/NAR/GKU552
P407
P577
2014-06-19T00:00:00Z