Natural killer (NK) cell response to virus infections in mice with severe combined immunodeficiency. The stimulation of NK cells and the NK cell-dependent control of virus infections occur independently of T and B cell function.
about
Temporal association of cellular immune responses with the initial control of viremia in primary human immunodeficiency virus type 1 syndromeImmunobiology of congenital cytomegalovirus infection of the central nervous system—the murine cytomegalovirus modelGenetic Causes of Human NK Cell Deficiency and Their Effect on NK Cell SubsetsDefective interleukin (IL)-18-mediated natural killer and T helper cell type 1 responses in IL-1 receptor-associated kinase (IRAK)-deficient miceEffects of virally expressed interleukin-10 on vaccinia virus infection in mice.Cytomegalovirus-encoded beta chemokine promotes monocyte-associated viremia in the host.Acute, lethal, natural killer cell-resistant myeloproliferative disease induced by polyomavirus in severe combined immunodeficient mice.Systemic priming-boosting immunization with a trivalent plasmid DNA and inactivated murine cytomegalovirus (MCMV) vaccine provides long-term protection against viral replication following systemic or mucosal MCMV challengeCytomegalovirus evasion of natural killer cell responses.The pathogenicity of cytomegalovirus.A mutation of Ikbkg causes immune deficiency without impairing degradation of IkappaB alpha.Mucosal and parenteral vaccination against acute and latent murine cytomegalovirus (MCMV) infection by using an attenuated MCMV mutant.Salivary gland NK cells are phenotypically and functionally uniqueGamma interferon-dependent clearance of cytomegalovirus infection in salivary glandsCD8+ T lymphocytes control murine cytomegalovirus replication in the central nervous system of newborn animals.Murine cytomegalovirus m02 gene family protects against natural killer cell-mediated immune surveillance.IKBKG (nuclear factor-kappa B essential modulator) mutation can be associated with opportunistic infection without impairing Toll-like receptor function.Genetic control of innate immune responses against cytomegalovirus: MCMV meets its match.Rapid tumor formation of human T-cell leukemia virus type 1-infected cell lines in novel NOD-SCID/gammac(null) mice: suppression by an inhibitor against NF-kappaBGain of virulence caused by loss of a gene in murine cytomegalovirusIdentification of Mouse Cytomegalovirus Resistance Loci by ENU Mutagenesis.Interferon gamma-producing gammadelta T cell-dependent antibody isotype switching in the absence of germinal center formation during virus infection.Characterization of hu-PBL-SCID mice with high human immunoglobulin serum levels and graft-versus-host disease.Potential role of natural killer cells in controlling tumorigenesis by human T-cell leukemia viruses.Murine cytomegalovirus with a deletion of genes spanning HindIII-J and -I displays altered cell and tissue tropismDistinct organ-dependent mechanisms for the control of murine cytomegalovirus infection by natural killer cells.Role of ITAM-containing adapter proteins and their receptors in the immune system and bone.Recognition of virus-infected cells by natural killer cell clones is controlled by polymorphic target cell elements.Selective long-term elimination of natural killer cells in vivo by an anti-interleukin 2 receptor beta chain monoclonal antibody in miceMurine cytomegalovirus is regulated by a discrete subset of natural killer cells reactive with monoclonal antibody to Ly49H.Natural killer cell tolerance in mice with mosaic expression of major histocompatibility complex class I transgeneInterferon gamma regulates acute and latent murine cytomegalovirus infection and chronic disease of the great vessels.Evolving role of 2B4/CD244 in T and NK cell responses during virus infection.NK cells controlling virus-specific T cells: Rheostats for acute vs. persistent infections.ATF3 regulates MCMV infection in mice by modulating IFN-gamma expression in natural killer cells.Natural killer cells act as rheostats modulating antiviral T cells.Mechanisms for virus-induced liver disease: tumor necrosis factor-mediated pathology independent of natural killer and T cells during murine cytomegalovirus infection.Antiviral immune responses of lymphocytic choriomeningitis virus-infected mice lacking CD8+ T lymphocytes because of disruption of the beta 2-microglobulin gene.In vivo role of lymphocyte subpopulations in the control of virus excretion and mucosal antibody responses of cattle infected with rotavirus.Lymphocytes protect against and are not required for reovirus-induced myocarditis.
P2860
Q24648628-B25F3DA8-1FE4-4F72-AA42-38029E911A3CQ27026924-F338FFD2-5F25-4EAE-856A-A58DD9D7DF10Q28067993-9D77447E-F61A-441D-AB16-EFC90D9265A0Q30322144-8A117CF8-8799-404E-A777-A0B7DB870BC1Q30452141-77D779F3-8D0B-47FC-A580-6FA97F7450A2Q30478992-E5C0B393-AB1A-407D-A825-F5553A0ED29CQ33493551-DFF793AF-1D50-422D-88A1-CB033BE70D94Q33551402-D294234B-6235-4BEC-BD18-0A2A2ABD502CQ33683395-15580459-0DE3-4679-B40A-A7BD53AE01FAQ33697113-CF6BB005-6FA8-4077-ADBE-262D77A29C8CQ33733874-358A5A86-A6A4-40AD-BDED-3793D15EFF13Q33782029-5D08141B-5610-41A9-82CF-1B46125E0605Q33797827-A1034CC3-8E70-4548-B35F-85AE91AEC110Q33930607-15CAA4C2-A0FF-4BAF-BEA7-523BC586C802Q34166586-85467926-79A1-4A33-A57A-5E109CCA5DA0Q34357626-12178494-0DC0-4AB9-BA55-F87500945737Q34630390-74F08CC0-0B2C-4BAD-9529-98F031A7E215Q34761230-B97828AF-E442-4EF0-BD47-0D095280A398Q34975395-EFBE7FE6-1510-4131-AF3C-204B5E94490FQ35123311-D4F13CDF-F6D9-42FB-959F-EE07C1354FB1Q35259980-96F15EF7-3DE4-4261-87AA-CE10D3B3A196Q35797635-6B8B4861-68F4-4280-BF25-60A956B7AEC6Q35832063-FA2AECAF-3B70-4F95-9750-D008EEAA8AE6Q35833430-F66A0D4C-5D96-4C89-8914-DFB5E80BFA94Q35855843-A7B3DDDC-94F8-4332-8DCC-3F786FE102EBQ35874845-D3082572-DCCB-4222-A82E-9FA54FA1DD28Q36324920-AD958445-5CCE-4B2C-8F5F-50CAE7AEF476Q36362289-C5CBE7AD-1021-4886-BEA8-C78D325373EFQ36362323-348B1BB5-CF56-4C4D-AA29-028514D1B96DQ36369341-9A109E4E-AAF4-4D9F-BE28-6421D9453EC7Q36380455-795EBD58-8C13-4FF8-9E36-CC2440E5169AQ36401507-CDEF57F6-1CB1-4EDA-9144-4BBACCD05FAEQ36457304-2345D438-9420-4BAF-B6A8-DCFE14BEFD6AQ36464528-35E0EBA5-9AFE-4706-8BE1-CC67FE2657DAQ36497388-D9EB5B84-4E7C-4D7A-A9CB-71F7E350827BQ36517134-765DF8AF-DAAB-4D2D-83C1-330EF5EA9722Q36548764-B5CD2606-D51E-4DAD-8BE1-8286A952DA5AQ36639479-EB6920E8-B257-418F-A634-CB4C61BAD90AQ36651476-D5DD94C2-6271-424A-BE52-07CADF724607Q36653708-2FB7DBB8-ACDC-4965-9F1C-4A1763282A47
P2860
Natural killer (NK) cell response to virus infections in mice with severe combined immunodeficiency. The stimulation of NK cells and the NK cell-dependent control of virus infections occur independently of T and B cell function.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Natural killer (NK) cell respo ...... ntly of T and B cell function.
@en
type
label
Natural killer (NK) cell respo ...... ntly of T and B cell function.
@en
prefLabel
Natural killer (NK) cell respo ...... ntly of T and B cell function.
@en
P2093
P2860
P356
P1476
Natural killer (NK) cell respo ...... ntly of T and B cell function.
@en
P2093
C L O'Donnell
J O Brubaker
M Vargas-Cortes
P2860
P304
P356
10.1084/JEM.173.5.1053
P407
P577
1991-05-01T00:00:00Z