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Pathophysiology of L-dopa-induced dyskinesias.Effects of age on axon terminals forming axosomatic and axodendritic inhibitory synapses in prefrontal cortex.Decreased GAD67 mRNA levels in cerebellar Purkinje cells in autism: pathophysiological implications.Decreased glutamic acid decarboxylase mRNA expression in prefrontal cortex in Parkinson's disease.Unilateral 6-hydroxydopamine lesion of dopamine neurons and subchronic L-DOPA administration in the adult rat alters the expression of the vesicular GABA transporter in different subsets of striatal neurons and in the substantia nigra, pars reticulaTime-course of SKF-81297-induced increase in glutamic acid decarboxylase 65 and 67 mRNA levels in striatonigral neurons and decrease in GABA(A) receptor alpha1 subunit mRNA levels in the substantia nigra, pars reticulata, in adult rats with a unilatDecreased parvalbumin mRNA expression in dorsolateral prefrontal cortex in Parkinson's disease.Decreased GAD65 mRNA levels in select subpopulations of neurons in the cerebellar dentate nuclei in autism: an in situ hybridization study.Metabotropic glutamate mGluR5 receptor blockade opposes abnormal involuntary movements and the increases in glutamic acid decarboxylase mRNA levels induced by l-DOPA in striatal neurons of 6-hydroxydopamine-lesioned rats.Decreased parvalbumin mRNA levels in cerebellar Purkinje cells in autism.Basal ganglia and autism - a translational perspective.Ultrastructural analysis of the serotonin hyperinnervation in adult rat neostriatum following neonatal dopamine denervation with 6-hydroxydopamine.Tyrosine kinase B and C receptors in the neostriatum and nucleus accumbens are co-localized in enkephalin-positive and enkephalin-negative neuronal profiles and their expression is influenced by cocaine.Dual effects of intermittent or continuous L-DOPA administration on gene expression in the globus pallidus and subthalamic nucleus of adult rats with a unilateral 6-OHDA lesion.Normalization of glutamate decarboxylase gene expression in the entopeduncular nucleus of rats with a unilateral 6-hydroxydopamine lesion correlates with increased GABAergic input following intermittent but not continuous levodopa.High-dose methamphetamine acutely activates the striatonigral pathway to increase striatal glutamate and mediate long-term dopamine toxicity.Two isoforms of glutamate decarboxylase: why?Comparative effects of acute or chronic administration of levodopa to 6-hydroxydopamine-lesioned rats on the expression of glutamic acid decarboxylase in the neostriatum and GABAA receptors subunits in the substantia nigra, pars reticulata.Increased GAD67 mRNA expression in cerebellar interneurons in autism: implications for Purkinje cell dysfunction.c-fos gene expression is induced in a subpopulation of striatal neurons following a single administration of a dopamine D1-receptor agonist in adult rats lesioned with 6-OHDA as neonates.Haloperidol treatment after high-dose methamphetamine administration is excitotoxic to GABA cells in the substantia nigra pars reticulata.Differential regulation of glutamate decarboxylase and preproenkephalin mRNA levels in the rat striatum.Dopamine and serotonin interactions in the modulation of the expression of the immediate-early transcription factor, nerve growth factor-inducible B, in the striatum.Gene expression of the GAD67 and GAD65 isoforms of glutamate decarboxylase is differentially altered in subpopulations of striatal neurons in adult rats lesioned with 6-OHDA as neonates.Serotonin innervation in adult rat neostriatum. I. Quantified regional distribution.Subchronic administration of L-DOPA to adult rats with a unilateral 6-hydroxydopamine lesion of dopamine neurons results in a sensitization of enhanced GABA release in the substantia nigra, pars reticulata.Effects of nigrostriatal lesions on the levels of messenger RNAs encoding two isoforms of glutamate decarboxylase in the globus pallidus and entopeduncular nucleus of the rat.Messenger RNAs encoding glutamate-decarboxylases are differentially affected by nigrostriatal lesions in subpopulations of striatal neurons.Effects of quinolinic acid on messenger RNAs encoding somatostatin and glutamic acid decarboxylases in the striatum of adult rats.Preproenkephalin mRNA expression in the caudate-putamen of MPTP monkeys after chronic treatment with the D2 agonist U91356A in continuous or intermittent mode of administration: comparison with L-DOPA therapy.Glutamate decarboxylase (GAD65) gene expression is increased by dopamine receptor agonists in a subpopulation of rat striatal neurons.Glutamate decarboxylase (GAD67 and GAD65) gene expression is increased in a subpopulation of neurons in the putamen of Parkinsonian monkeys.Elevation of dopamine D2 but not D1 receptors in adult rat neostriatum after neonatal 6-hydroxydopamine denervation.Radioautographic study of 3H-GABA uptake in the oculomotor nucleus of the cat.Loss of glutamic acid decarboxylase (Gad67) in striatal neurons expressing the Drdr1a dopamine receptor prevents L-DOPA-induced dyskinesia in 6-hydroxydopamine-lesioned mice.Monoamine innervation of the oculomotor nucleus in the rat. A radioautographic study.Serotonin innervation in adult rat neostriatum. II. Ultrastructural features: a radioautographic and immunocytochemical studyGABA innervation in adult rat oculomotor nucleus: a radioautographic and immunocytochemical studyMK-801 decreases striatal and cortical GAD65 mRNA levelsEffect of 6-OHDA lesions on striatal mRNA levels encoding for glutamate receptor subunits
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description
hulumtues
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researcher
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հետազոտող
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-Jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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Jean-jacques Soghomonian
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P106
P21
P31
P496
0000-0001-5950-026X