Normalization of glutamate decarboxylase gene expression in the entopeduncular nucleus of rats with a unilateral 6-hydroxydopamine lesion correlates with increased GABAergic input following intermittent but not continuous levodopa.
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Effects of Pharmacological Block of GABA(A) Receptors on Pallidal Neurons in Normal and Parkinsonian State.Basal Ganglia circuits underlying the pathophysiology of levodopa-induced dyskinesia.Unilateral 6-hydroxydopamine lesion of dopamine neurons and subchronic L-DOPA administration in the adult rat alters the expression of the vesicular GABA transporter in different subsets of striatal neurons and in the substantia nigra, pars reticulaTime-course of SKF-81297-induced increase in glutamic acid decarboxylase 65 and 67 mRNA levels in striatonigral neurons and decrease in GABA(A) receptor alpha1 subunit mRNA levels in the substantia nigra, pars reticulata, in adult rats with a unilatMetabotropic glutamate mGluR5 receptor blockade opposes abnormal involuntary movements and the increases in glutamic acid decarboxylase mRNA levels induced by l-DOPA in striatal neurons of 6-hydroxydopamine-lesioned rats.Adenosine A2A Receptor Gene Knockout Prevents l-3,4-Dihydroxyphenylalanine-Induced Dyskinesia by Downregulation of Striatal GAD67 in 6-OHDA-Lesioned Parkinson's Mice.Metabolic changes detected by proton magnetic resonance spectroscopy in vivo and in vitro in a murin model of Parkinson's disease, the MPTP-intoxicated mouse.Metabolic changes detected by ex vivo high resolution 1H NMR spectroscopy in the striatum of 6-OHDA-induced Parkinson's rat.
P2860
Q33701415-E409230C-5819-4037-955F-BC2C54781169Q34165820-B68D9C7D-8861-4DAC-A30C-30F9C567FD12Q35846784-1ECC802E-0680-45FC-A487-E7BD19BF1EA8Q36791342-16E90602-5D54-4A52-A0DD-55B3954799E7Q37384351-BD64E61F-6150-471A-86A5-F2A0351ADEE1Q37712460-E62AB283-EF93-411E-8218-A8F0D5E744E0Q46844307-1FDE8EE5-463D-4FC9-9E15-568962B840E8Q48393298-2B9E10D4-8BB6-4C13-BD49-ACCE5B0D23CB
P2860
Normalization of glutamate decarboxylase gene expression in the entopeduncular nucleus of rats with a unilateral 6-hydroxydopamine lesion correlates with increased GABAergic input following intermittent but not continuous levodopa.
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2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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name
Normalization of glutamate dec ...... t but not continuous levodopa.
@en
Normalization of glutamate dec ...... t but not continuous levodopa.
@nl
type
label
Normalization of glutamate dec ...... t but not continuous levodopa.
@en
Normalization of glutamate dec ...... t but not continuous levodopa.
@nl
prefLabel
Normalization of glutamate dec ...... t but not continuous levodopa.
@en
Normalization of glutamate dec ...... t but not continuous levodopa.
@nl
P1433
P1476
Normalization of glutamate dec ...... t but not continuous levodopa.
@en
P2093
K M Nielsen
P356
10.1016/J.NEUROSCIENCE.2003.08.010
P407
P577
2004-01-01T00:00:00Z