Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure.
about
Cardiac regulation by phosphoinositide 3-kinases and PTENAkt1 regulates pathological angiogenesis, vascular maturation and permeability in vivoAkt1 in endothelial cell and angiogenesisPhosphoinositide 3-kinase: friend and foe in cardiovascular diseaseOngoing controversies surrounding cardiac remodeling: is it black and white-or rather fifty shades of gray?Aerobic exercise training promotes physiological cardiac remodeling involving a set of microRNAsAkt isoforms in vascular diseaseRegulation of Akt signaling by sirtuins: its implication in cardiac hypertrophy and agingNADPH oxidases in heart failure: poachers or gamekeepers?Molecular basis of physiological heart growth: fundamental concepts and new playersOptimization of cardiac metabolism in heart failureHeart failure with preserved ejection fractionCardiac matrix: a clue for future therapyBone marrow support of the heart in pressure overload is lost with agingMechanistic target of rapamycin (Mtor) is essential for murine embryonic heart development and growthThe cell biology of disease: cellular mechanisms of cardiomyopathy.The role of sirtuins in cardiac diseaseMolecular Imaging of Angiogenesis in Cardiac RegenerationThe critical role of Akt in cardiovascular functionMicroRNA-214 Is Upregulated in Heart Failure Patients and Suppresses XBP1-Mediated Endothelial Cells AngiogenesisCardiac myocyte follistatin-like 1 functions to attenuate hypertrophy following pressure overloadDeletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptationRegulation of phosphodiesterase 3 and inducible cAMP early repressor in the heartVon Willebrand factor and the right ventricle (the MESA-Right Ventricle Study)Redox-sensitive Akt and Src regulate coronary collateral growth in metabolic syndromePhosphorylation of the Twist1-family basic helix-loop-helix transcription factors is involved in pathological cardiac remodelingCardiomyocyte GATA4 functions as a stress-responsive regulator of angiogenesis in the murine heartAnti-remodeling effects of rapamycin in experimental heart failure: dose response and interaction with angiotensin receptor blockadeTongxinluo protects against pressure overload-induced heart failure in mice involving VEGF/Akt/eNOS pathway activationRegulation of cardiac hypertrophic signaling by prolyl isomerase Pin1Connective tissue growth factor overexpression in cardiomyocytes promotes cardiac hypertrophy and protection against pressure overloadCopper-induced regression of cardiomyocyte hypertrophy is associated with enhanced vascular endothelial growth factor receptor-1 signalling pathwayHuman Pericardial Fluid Contains Exosomes Enriched with Cardiovascular-Expressed MicroRNAs and Promotes Therapeutic Angiogenesis.Cancer drug related cardiotoxicity during breast cancer treatment.Insulin Signaling and Heart Failure.Phenotypic screen quantifying differential regulation of cardiac myocyte hypertrophy identifies CITED4 regulation of myocyte elongation.Cancer therapy-induced cardiotoxicity: basic mechanisms and potential cardioprotective therapies.Intermittent pressure overload triggers hypertrophy-independent cardiac dysfunction and vascular rarefaction.Fog2 is critical for cardiac function and maintenance of coronary vasculature in the adult mouse heart.Increased afterload induces pathological cardiac hypertrophy: a new in vitro model
P2860
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P2860
Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Disruption of coordinated card ...... e transition to heart failure.
@en
Disruption of coordinated card ...... e transition to heart failure.
@nl
type
label
Disruption of coordinated card ...... e transition to heart failure.
@en
Disruption of coordinated card ...... e transition to heart failure.
@nl
prefLabel
Disruption of coordinated card ...... e transition to heart failure.
@en
Disruption of coordinated card ...... e transition to heart failure.
@nl
P2093
P2860
P356
P1476
Disruption of coordinated card ...... e transition to heart failure.
@en
P2093
Ichiro Shiojima
Kaori Sato
Masahiro Ito
Ronglih Liao
Stephan Schiekofer
Wilson S Colucci
Yasuhiro Izumiya
P2860
P304
P356
10.1172/JCI24682
P407
P577
2005-08-01T00:00:00Z